Ears & Eyes Anatomy/Physio Flashcards
Visible portion of ear
Pinna
Air filled sspace with 3 bones called the ossicle (malleus, incus, stapes)
Middle Ear
Low Frequency
heard at apex near helicotrema (wide and flexible)
High Frequency
base of cochlea (thin and rigid)
Tonotopy
Each frequency leads to vibration at specific location on the basilar membrane
Conductive Hearing loss
Bone > Air
Weber test will localize to affected ear
Sensorineural Hearing Loss
Air > Bone
Weber localizes to unaffected near
Noise induced hearing loss
damage to stereocilia cells in organ of Corti; loss of high frequency hearing 1st
Complete destruction of the facial nucleus or its branchial efferent fibers (facial nerve proper)
Facial nerve palsy
Lyme disease, herpes simplex/zoster, sarcoidosis, tumors and diabetes
Facial Nerve Palsy
Tx for Facial nerve palsy
Corticosteroids
Muscles that close jaw
Masseter, temporalis, medial pterygoid
Muscle that lowers the jaw
Lateral Pterygoid
IL paralysis of upper and lower face
LMN Lesion
Lesion ofmotor cortex or connection between Cx and facial nucleus
CL paralysis of lower face, forehead spared due to BL
UMN Lesion
Eye too short for refractive power of cornea and lens, light focused behind retina
Hyperopia, Farsighted
Eye too long for refractive power of cornea and lens, light focused in front of retina
Myopia, Nearsighted
Abnormal curvature of cornea resulting in different refractive power at different axes
Astigmatism
Decrease in focusing ability during accommodation due to sclerosis and decreased elasticity
Presbyopia
hypopyon
Sterile pus
Inflammation of anterior uvea and iris, often associated with Sarcoid, Rheumatoid arthritis, juvenile idiopathic arthritis, TB, HLA-B27)
Uveitis
Retinal edema and necrosis leading to scar, often viral (CMV, HSV, HZV)
Retinitis
Acute, painless monocular vision loss, retina cloudy with attenuated vassels and “cherry-red” spot at fovea
Central Retinal Artery Occlusion
Non-proliferative diabetic retinopathy
leakage of capillaries, lipids and fluid seep into retina, hemorrhages and macular edema
Tx of non-proliferative diabetic retinopathy
Blood sugar control, Macular laser
Proliferative Diabetic Retinopathy
Chronic hypoxia results in new blood vessel formation with resultant traction on retina
Tx of Proliferative Diabetic Retinopathy
Peripheral retinal photocoagulation, anti-VEGF injections
Collects aqueous humor from trabecular meshwork
Canal of Schlemm
Collects Aqueous humor that flows through Anterior Chamber
Trabecular Meshwork
Ciliary muscle
M3
Produces aqueous humor
Ciliary epithelium (Beta)
Painless increase in IOP and progressive peripheral visual field loss
Open Angle Glaucoma
Enlargement or forward movement of lens against central iris (pupil margin) leads to obstruction of normal aqueous flow through pupil
Primary Closed/Narrowed Angle Glaucoma
Hypoxia from retinal disease like diabetes and vein occlusion that induces vasoproliferation in iris that contracts angle
Seconadry Closed/Narrowed Angle Glaucoma
Chronic Closed Angle Glaucoma
Often asymptomatic with damage to optic nerve and peripheral vision
Acute Closed Angle Glaucoma
True ophthalmic emergency, very painful, sudden vision loss, halos around lights, rock-hard eye, frontal headache
Opacification of lens
Cataract
Optic Disc Swelling due to increased ICP
Papilledema
Enlarged blind spot and elevated optic disc with blurred margins on fundoscopic exam
Papilledema
Eye looks down and out; ptosis, pupillary dilation, loss of accommodation
CN III damage
Problems going down stairs, compensatory head tilt to CL side going downstairs
CN IV damage
eyes move upward wuth CL gaze and head tilt to side of lesion
CN IV damage
Medially directed eye that cannot abduct
CN VI damage
Have patient look medial and up
Inferior Oblique
Have Patient look medial and down
Superior Oblique
Have patient look lateral and up
Superior Rectus
Have patient look lateral and down
Inferior Rectus
Look lateral
Lateral rectus
Look medial
Medial rectus
Miosis
Constriction, PS, Edinger-Westphal nucleus to ciliary ganglion via CN III
Mydriasis
Dilation, Sympathetic, Hypothalamus to ciliospinal center of Budge
Short ciliary nerves
Parasympathetic, causes Miosis (constriction)
Long Ciliary Nerve
Sympathetic, causes Mydriasis (dilation)
Marcus Gunn Pupil
Afferent pupillary defect, optic nerve or severe retinal injury.
decreased BL pupillary constriction with light in affected eye, light in unaffected eye will cause BL pupillary constriction
Swinging Flashlight test
Marcus Gunn Pupil
Interior CN III damage
Diabetes, vascular issues; motor output is affected
ptosis, down and out gaze
Peripheral CN III damage
compression of PCom aneurysm or uncal herniation
diminished or absent pupillary light reflex; “blown pupil” can have down and out gaze
Flashes and Floaters, eventual monocular loss of vision like curtain drawn down
Retinal detachment
Meyer loop
inferior retina; loops around inferior horn of lateral ventricle
Dorsal Optic Radiation
superior retina; takes shortest path via internal capsule
Pituitary Lesion, Chiasm
Bitemporal Hemianopia
Right temporal Lesion, MCA
Left upper quadrantic anopia
Right parietal lesion, MCA
Left lower quadrantic anopia
PCA infarct
Left hemianopia with macular sparing
Macular degeneration
Central Scotoma
Lesion of Medial Longitudinal Fasciculus
Internuclear Ophthalmoplegia, Abducting eye gets nystagmus (CNVI overfires to stimulate CNIII) convergence is normal
MLF in looking left
Left nucleus of CN VI fires contracting Lat rectus, Stimulates CL (right) nucleus of CN III via right MLF to contract the left medial rectus
Naming INO
refers to which eye is paralyzed, Right INO is a right MLF Lesion (right eye can’t adduct and left eye, abducting eye has nystagmus)
Do 10 push ups
or 20