COPD Flashcards
What is COPD
- progressive disase that makes it hard to breathe
- not cured but can be prevented and treated
what can COPD cause
- Wheezing
- SOB
- chest tightness
- coughing the produces large amounts of mucus
What can cause COPD
- cigarette smoking (leading cause)
- long term exposure to other irritants (polution, chemical fumes or dusts etc)
- rare genetic condition called alpha-1 antitrypsin deficiency
risk factors for COPD
- smoking/second hand smoke
- lung irritants
- family history
- history of respiratory infections as a child
What combinations of diseases overlap with COPD/are COPD disorders
- Chronic bronchitis
- emphysema
- asthma
Pathogenesis of COPD
- air movement in the lungs comes in forcefully and with increased pressure
- air movement out of the lungs is not as forceful and therefore bronchial walls collapse causing air trappings
Obstruction can occur with
- inflammation of air airways
- bronchial tone/spasm
- weakening or support structures
- alveolar destruction
- lack of surfactant
- retained secretions
Normal FEV vs COPD FEV
- should be able to breathe out 75% of air within 1 sec
- takes those with COPD longer
Lung volumes and capacities with COPD
- increased RV
- increased TLC
Radiographic signs of COPD
- wider AP diametes
- AP ratio is 1:1 (normal 2:1)
- enlarged heart (due to right side overworking)
Common characteristics of COPD
- Dyspnea,
- chronic cough
- hypoxia with or without hypercapnia
- alveoli that are narrow and less flexible
- radiographic and functional changes to thorax
- spirometry changes
- pulmonary HTN which can lead to right heart failure
- can be septic or non-septic (both also prone to infection)
- weakness, muscule atrophy, osteoporosis related to decreased mobility becuase of disease
- cognitive impairment, anxiety, depression
Non-septic COPD
- Emphysema
- Chronic bronchitis
- possibly chronic asthma
Asthma
- meets general criteria but it is reversible
- hyperreactive airway that causes inflammatory response
- bronchioles can remodle
- exercise induced: can occur hours after due to loss of water and heat in the lower respirtoary system
- can hear wheezes
- higher rate in females
- usually mothers side/smoked during pregancy
Treatments for asthma
- ask if they use inhlater before an dafter sport
- short term inhalers and long term inhalers
- often times a mixture of smooth muscule dilators and anti-inflammatory medication
- can also use nebulizer
Emphysemia
- damaged alveoli
- loss of surface are for gas exchnage
- breakdown areas = bullae
Alpha-1 antitrypin deficiency: genetically acquired emphysema
- AAT is a protein that is made in the liver
- inhibites elastace which breaks down lung tissue
- deficiency leads to breakdown of tissue in lungs and liver dysfunction
- breakdown can be accelerated in those who smoke
- can also lead to liver cirrhosis
Chronic bronchitis
describe
- comes back very year or every so often
- long term inflammation of epithelium,
- enlarged submucusal gland/mucus accumulation
- hyperinflation of alveoli
- damaged cilia
Septic obstructive pulmonary diseases
- Cystic fibrosis
- bronchiectasis
Cyctis fibrosis
- genetic defect in lungs,
- excessive production of thickened dehydrated hyperviscous mucus
Organs affected by cystic fibrosis
- Sinuses (infection)
- lungs: thick sticky mucus buildup, bacterial infection and widened airways
- skin: sweat glands produce salty sweat (can be diagnositic )
- pacrease: blocked pancreatic ducts
- intestines: cannot fully absorb nutrients
- reporductive organs: problems with fertility or delyaed puberty
Bronchiectasis
- flabby airways with mucus hypersecretion
- weak wall causes collapse with exhale
GOLD classification system
- Group a: low symptoms/risk, mMRC 0-1; >50% FEV, 0-1 outpatient or 0 hopsitalized excaerbation
- Group B: high symptoms/low risk, mMRC 2-4, >50% FEV, 0-1 outpatient or 0 hopsitalized exacerbations
- Group C: low symptoms, High risk, mMRC 0-1, <50% FEV, >2 outpatinet or >1 hospitalized exacerbations
- Group d: High symptoms, High risk, mMRC 2-4, <50% FEV, >2 outpatient or >1 hospitalized exacerbations
dont memorize but look over
What to watch for with COPD both septic and non-septic
- increase cough or sputum production above normal
- increased body temp
- increased respiratory rate above patient norm
- changes in X-ray, auscultation, ABGs, PFTs
- Decreased appetite, weight activity levels
Treatment for COPD
As severity increases (least severe to most)
- self management education and smoking cessation
- bronchodilators
- inhaled corticosteriods
- pulmonary rehabilitation
- oxygen
- surgery
Medications for COPD
- bronchodilators: short or long acting
- Corticosteriods
- mucolytic agents
- antibiotics
- oxygen
Physical therapy for COPD
- breathing exercises
- chest mobility, posture, shoulder ROM
- postural drainage
- bronchial hygiene techniques when neeeded (precussion/vibration/cough. huff)
- aerobic/strength conditioning
- relaxation training
surgical options for COPD
- bullectomy: can remove a portion
- lung volume reduction (segment or lobe remove)
- lobectomy
- transplant