Cardiomyopathies and valve disease Flashcards

1
Q

Cardiomyopathies

A
  • disease where contraction and relaxation of the myocardial fibers is impaired
  • genetic, acquired or mixed, could also be idiopathic
  • overuse of alcohol.drugs, infection, muscular dystrophy
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2
Q

3 primary classifications of cardiomyopathies and how they are determined

A
  • Determined by EKG and heart muscle biopsy
  • dilated cardiomyopathy
  • hypertrophic cardiomyopathy
  • restrictive cardiomyopathy
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3
Q

Dilated cardiomyopathy
clinical features

A
  • increase cardiac mass
  • dilation of all 4 chambers
  • minimal or no wall thickening
  • systolic dysfunction
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4
Q

How does a dilated cardiomyopathy occur

A
  • mitochondrial problem within the muscle fiber
  • thins out the muscle
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5
Q

How does the dysfunction impact physiology during a dilated cardiomyopathy

A
  • valves don’t meet = valve dysfunction and pressure changes
  • ESV/ESP change
  • atrium then dilates and the pressures move backward
  • can sustain CO with elevated HR
  • later on will need a + ionotrop to assist the workload of the heart
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6
Q

Hypertrophic cardiomyopathy clinical features

A
  • hypertrophy of the cardiac muscle
  • increase wall thickness (myocytes lay doesn’t in an asymmetrical fashion)
  • no dilation
  • normal or increased systolic function
  • diastolic dysfunction
  • may have outflow obstruction due to septum becoming enlarged
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7
Q

How does the dysfunction of hypertrophic cardiomyopathy impact function?

A
  • more O2 is required for more muscle but outflow gets obstructed
  • blood will back up into atria
  • may come with arrhythmia
  • require (-) inotropic to decrease work of heart
  • can remove overgrowth of the heart
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8
Q

Restrictive cardiomyopathy clinical features

A
  • increase stiffness of ventricular walls
  • has features of both diastolic and systolic dysfunction
  • heart cannot contract as well but also doesn’t relax as well
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9
Q

What has been associated with restrictive cardiomyopathy

A
  • amyloidosis: excess protein lay down
  • post radiation
  • diabetes
  • fibrosis
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10
Q

Heart valve disease symptoms

A
  • my include chest discomfort
  • SOB
  • dizziness
  • edema
  • palpitations
  • rapid weight gain from increased in fluid
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11
Q

causes of heart valve disease

A
  • birth defects
  • infections (rheumatic fever and endocarditis)
  • damage caused from other conditions
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12
Q

treatment for heart valve disease

A
  • valvuloplasty
  • medications for symptoms
  • valve repair/replacement surgery
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13
Q

how does heart valve disease affect the heart

A
  • when valves become stenosed, prolapsed or insufficient overall impact is more work for the PRECEDING chamber
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14
Q

What is heard with a prolasped valved

A
  • murmur
  • due to not closing fully and letting some back flow of blood
  • there is more space in the chamber below due to the valve ballooning out
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15
Q

Infective endocarditis

what is it and symptoms

A
  • bacterial or fungal infection on valve an prevents it from closing
  • presents at first with flu like symptoms then SOB and fatigueT
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16
Q

Treatment for valve disease

Surgical and non surgical

A
  • often anticoagulants to prevent clotting in valves from stagnant flow
  • surgical intervention - repair (valvuloplasty) or replacement
17
Q

valvuloplasty

A
  • blow up ballon to open the valve that has stenosis
18
Q

Valve repair (annuloplasty

A
  • ring insertion to help maintain leaflet contact
  • replace the ring around the valve to help it function better
19
Q
  • Valve replacement
A
  • mechanical vs biological
  • mechanical = different types but made form metal
  • biological: comes from cows and pigs
20
Q

Biological prosthetic valve

A
  • lasts 8-10 years
  • no anticoagulation meds
  • no audible click
21
Q

Mechanical prosthetic valve

A
  • lasts > 20 years
  • lifelong anticoagulants
  • click
22
Q

Valve replacement surgery types

not material used

A
  • TAVR: transfemoral
  • TAVR: transapical
  • TAVR: transaortic
  • open heart (AVR)
  • minimal incision valve surgery
23
Q

Pericardial effusion

A
  • caused by pericarditis
  • inflammation of the heart’s pericardium due to injury or disease leading to accumulation of fluid
  • affects contraction and relaxations as there is not room to move
24
Q

cardiac tamponade: factors leading to it

A
  • rate of accumulation
  • amount of fluid in pericardium
  • compliance of pericardium
25
Q

pathophysiology of cardiac tamponade

A
  • increased volume in pericardial space = compression of atria, vena cave and pulmonary veins
  • reduced RV filling in diastole = decreased stroke volume/CO
  • RV collapse occurs leading to cardiac arrest
26
Q
A