Class 13 - Memory Flashcards

1
Q

Where did HM have missing parts of his brain?

A

Bilateral temporal lobes

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2
Q

What type of amnesia did HM have?

A

Anterograde amnesia (long term memory after the injury)

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3
Q

What type of learning was left intact in HM?

A

Intact procedural learning

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4
Q

What are the two types of long term memory?

A

Declarative (explicit, you can declare it) and nondeclarative (implicit)

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5
Q

What are the two types of declarative (explicit) memory?

A

Episodic (events) and semantic (facts) memory

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6
Q

Procedural memory, perceptual priming, classical conditioning, nonassociative learning (habituation/sensitization) are what type of memory?

A

Nondeclarative memory

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7
Q

What is semantic memory?

A

Facts and knowledge

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8
Q

What is consolidation theory?

A

Hippocampus consolidates all memories then stores them elsewhere. Damage makes you lose old memories

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9
Q

What theory accounts for episodic memory remaining dependant on the hippocampus and semantic memory becoming independent gradually?

A

Multiple-trace theory

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10
Q

What is reconsolidation theory?

A

When you recall a memory, it becomes labile and then you restore it as a new memory

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11
Q

What is dual-process theory?

A

The hippocampus is crucial only for episodic memory.

Semantic memories are independent of the hippocampus

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12
Q

What connects the posterior neocortex to the hippocampus?

A

Perforant pathway

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13
Q

What connects the hippocampus to the thalamus, frontal cortex, basal ganglia, hypothalamus

A

Fimbria - fornix pathway

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14
Q

What does damage to the fimbria - fornix pathway and temporal stem do?

A

Antero and retrograde amnesia

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15
Q

What conclusions do studies of hippocampal damage suggest?

A

Anterograde deficits are more severe, episodic memories are more affected than semantic memories, patients cannot “time travel” to the past or future

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16
Q

What does the rhinal cortex include? Where does it project to?

A

Entorhinal and perirhinal cortex. It projects to the hippocampus

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17
Q

Why is the perirhinal cortex so crucial at memory?

A

Connected to visual, sensory, auditory, executive, emotion and memory/learning areas of the brain

18
Q

What side of the temporal cortex, when removed leads to deficits in face recognition, spatial position and maze learning

A

Right temporal cortex

19
Q

What side of the temporal cortex, when removed, leads to deficits in word list recall, recall of consonant trigrams and non-spatial associations?

A

Left temporal cortex

20
Q

What is priming?

A

Stimulus sensitizes brain to later presentation of same stimulus.

21
Q

What is a test for priming?

A

The Gollin Incomplete Figures Test

22
Q

What is the study test modality shift?

A

Less likely to see an implicit memory effect when the priming subject is changed

23
Q

What is involved in habit and procedural learning?

A

Basal ganglia

24
Q

What cortex (motor or sensory) is part of implicit knowledge acquisition?

A

Motor cortex

25
Q

What plays a role in classical conditioning?

A

Cerebellum

26
Q

Which memory is present at birth (implicit or explicit)?

A

Implicit memory

27
Q

What could be causing “infatile amnesia”?

A

Lack of language development to label things

28
Q

What impairments does early hippocampal memory usually cause (episodic or semantic or both)?

A

Episodic memory impairments. Semantic and implicit memory is intact

29
Q

How does working and short term memory differ?

A

Working memory makes you do something with the information rather than to just repeat the information in short term memory

30
Q

Are injuries to parietal and occipital cortices causing prosopagnosia and color amnesia actually amnesia?

A

Not really

31
Q

What is hemispheric encoding retrieval asymmetry ( HERA)

A

Left prefrontal cortex encodes semantic memory, right prefrontal cortex retrieves episodic memory

32
Q

What cortex is in charge of retrieval and prospective memory?

A

Frontal cortex

33
Q

What is the partial or total loss of explicit memory?

A

amnesia

34
Q

What is transient global amnesia?

A

Sudden onset and loss of old and new memories

35
Q

How can you make a transient, reversible amnesia (it is also a depression treatment)

A

Electroconvulsive therapy

36
Q

What is infantile amnesia?

A

Loss of memory for the early years of life

37
Q

How do you make specific amnesias?

A

Have restricted brain damage

38
Q

What is anterograde amnesia? Retrograde amnesia?

A

Cant remember new memories, cant remember old memories respectively

39
Q

Where does herpes simplex encephalitis damage?

A

Medial temporal lobe (anterograde amnesia) and insula (retrograde amnesia)

40
Q

What is Korsakoff’s syndrome characterized by?

A

Anterograde amnesia, retrograde amnesia, lies, lack of insight, apathy

41
Q

What vitamin deficiency is Korsakoff’s caused by?

A

Thiamine (vitamin B 1) deficiency

42
Q

Where is the damage in Korsakoff’s syndrome?

A

Medial thalamus, mammillary bodies and general atrophy