Cardiology Flashcards
depolarization cycle
- SA node
- AV node
- atrial contraction
- bundle of His
- L and R bundle branches
- purkinje fibers
ventricular filling includes
atrial systole and diastole
2 steps of ventricular systole
isovolumetric and ejection
2 steps of ventricular relaxation
isovolumetric and relaxation
which is longer, systole or diastole
diastole
what are the atrio valves called
AV
which is the bicuspid valve
left AV
2 semilunar valves
aortia and pulmonary
diastole has which valve closed
aortic
lowest pressure
diastolic pressure
highest pressure
systolic pressure
what happens in systole
arotic valves open and ejection occurs
what causes the dicrotic notch in systole
backflow of blood in aorta
MAP
mean pressure in aorta over cardiac cycle
MAP is closer to DP or SP
DP
lubb
AV valves
dubb
SL valves
F =
delta P/ R
lower MAP than normal
hypotension
MAP > normal
hypertension
stress on heart and blood vessels
short term regulation includes
CO and total peripheral resistance
neural control
what kind of regulation includes the heart and blood vessels
short term regulation
what kind of regulation includes the kidneys
long term regulation
LTR includes
blood volume regulation
primarily hormonal control
negative feedback loops for MAP include
detector = baroreceptors integration = CV centers in CNS controllers = ANS effectors = heart and blood vessels
pressure receptors
baroreceptors
where are arterial baroreceptors found
aortic arch and carotid sinuses
what are arterial baroreceptors also called
sinoaortic receptors
external measure of electrical activity of the heart
electrocardiogram
distance and amplitude of spread depends on 2 factors
size of potentials
synchronicity of potentials from other cells
P wave
atrial depolarization
QRS wave
ventricular depolarization
T wave
ventricular repolarization
PQ segment
AV node delay
QT segment
ventricular systole
TQ interval
ventricular diastole
time in between heartbeats
R-R interval
sinus rhythm
pace generated by SA node
slowed/ diminished conduction through AV node
increases PQ segment duration/ delay between artrial and ventricular contraction
first degree heart block
slowed, sometimes stopped conduction through AV
loss of 1-1 relationship between P wave and QRS
second degree heart block
loss of conduction through VA node
P wave is independewnt of QRS
third degree heart block
extra contraction results in
extra systole
premature atrial contraction (PAC) followed by extra ventricular contraction
loss of coordination of electrical activity of the heart
ventricular fibrillation