Autonomic Flashcards

1
Q

Do the both myelinated and unmyelinated have schwan cells and what is the difference

A

Yes they both have but in the inmyelinated it is not active

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2
Q

Why is the schwan cells are so important

A

Because they form the neurolemma that give the neurons the ability to regenerate

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3
Q

Which part of the body was a ganglion in the embryonic life

A

The adrenal madula

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4
Q

They sympathitic do vasoconstriction in all vessels except

A

The somatic vessels

Coronary arteries

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5
Q

How does the sympathetic effect the reticular formation

A

It lowers the threshold so we get more alerted

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6
Q

What is horner’s syndrome

A

It is any lesion ior tumer or trama to the cervical ganglion or T1,T2

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7
Q

What does horner syndrome do

A

It cause lose unilateral sympathetic supply to the neck and head

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8
Q

What does the scaral centers control in the ANS

A

Erection
Defeation
Peeing

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9
Q

What does the madulla oblingata control in the ANS

A

Cardio vascular

Respiration

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10
Q

What does the mid brain control in the ANS

A

The eye

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11
Q

What does the hypothalamus control in the ANS

A

Thermostat
Glucostat
Appetitestat

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12
Q

What does the lympbic system control in the ANS

A

The emotional reflexes

The behavioral reflexes

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13
Q

What are the components of the ACh

A

Achytl CoA

Choline

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14
Q

What is the name of the enzyme that is involved in the ACh synthesis

A

Choline acetyl transferase

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15
Q

How is the ACh is removed

A

By distraction

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16
Q

Why we distract ACh instead of removing it

A

Because the accumulation of the ACh is so dangerous

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17
Q

What does the accumulation of the ACh do

A

It doea parasympatheticover

By lowering HR and constriction of branches

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18
Q

What is the name if the enzyme that is involved in ACh distraction

A

It is called ACh estrase

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19
Q

How many types of the ACh estrase do we have ans where are they

A

We have two rypes true and pseudo

True in the nerves ending

Pseudo in the plasma

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20
Q

What is the most famous anti ACh estrase drug

A

Physostigmine

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21
Q

What is the name of the efferents that are leaving the CNS

A

Called centeral

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22
Q

What are the types of the central efferents

A

Somatic

Perganglionic autonomic

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23
Q

What are the name of the efferents that are the leaving the ganglions

A

Peripheral efferents

24
Q

What are the types of the peripheral efferents

A

Pos ganglion sympathatic

Pos ganglion parasympathetic

25
What is the chemical transmitter that is used in the ganglions
ACh
26
What is the chemicla transmitter of the parasympathetic
ACh
27
What is the chemical transmitter of the sympathetic
Nor epi except the sweet glands and the blood vessels of the skeletal muscles
28
What is the use name of the central cholinergic receptors
Nicotinic receptors
29
Why is the nicotinic receptors called like this
Because they are sensitive to nicotine
30
What is the used name of the peripheral cholinergic receptors
Muscarinic
31
How many types of cholinergic receptors we have
Nicotinic | Muscarinic
32
What is the drug that stimuli nicotinic receptors
Nicotine small dose
33
What drug stimulates mascarinic receptors directly
Pilocarpine
34
What are the drugs that stimuli the chloinergic receptors indirectly
ACh estrases like prostigmine
35
What is the treatment for the war gases and patients with insecticide that has anti AChestrase
We give him Atropin to stop the para
36
What is the blocker of the nicotinic receptors
Nicotinic high dose
37
What is the blocker of the N-M junction
Curare
38
What is the blocker of the muscarinic receptors
Atropine
39
What is the difference between the adrenaline and noradrenaline
Adrenaline is a hormone | Noradrenaline is neurotransmitters
40
Which is stronger epi ir norepi and how many time
Epi is stronger by 10 times
41
What is the formation chain of the nor adrenaline
Phenylalanine tyrosine Dopa dopamine Nor adrenaline Adrenaline
42
In how many ways we get rid of noradrenaline from the synapse
1- 80% re uptake by the neuron 2-blood diffusion 3-MAO monoamino oxidase 4-COMT catechol-o-methyltransferase
43
Where is the MAO exists
Liver kidney CNS
44
Where is the COMT enzyme exist
Liver kidney
45
Where is the beta -2 receptors exists and what do they don
``` They are inhibitors Branchiol nuscles Blood vessels expect skeleton muscles vessels Stomach wall Bladder wall ```
46
Where is the beta -1 receptors exists and what do they don
Heart Hormones Metabolic Adrenergic nerves
47
Where is the alpha -1 receptors exists and what do they do
Pupillary dilator Spleen Sphincters
48
Where is the alpha -2 receptors exists and what do they do
Intestines walls | Adrenergic nerve
49
Which receptors are effected by the epi
Alpha ثم alpha ثم alpha ثم beta
50
How does the adrenaline effects the alpha and the beta receptors
It effects them both equally
51
What is the blocker of the alpha
Phentalamine
52
What blockes the beta rec
Propronolol
53
What are the adrenergic nerve endings stimuli drugs
Amphetamine | Ephedrine
54
What are the adrenergic nerve ending inhibitors drugs
False transmiten methyl dope | Guanithidine
55
Are the epi and nor epi hormones or neurotransmitters with explanation
They are hormones and neurotransmitters but the epi work as neurotransmitters in the CNS only but the nor epi work in the CNS and in the PNS in the sympathetic
56
What is the effect if the nor epi on the systolic and diastolic pressure
It increases both | Diastolic by VC and systolic by Heart rate
57
What is the effect of the epi on the systolic and diastolic pressure
It increases systolic by heart rate | But do not effect the diastolic because it do VD