ANS Oro Facial Aspects Flashcards
what does ANS do
control body systems except skeletal muscle
what does the ANS regulate
involuntary function
- smooth muscle
- cardiac
- gland sec
what awesome untrained aspects of ANS
urination
defaction
what does parasymp do
coordinates the bodys haemostatic functions
what does symp do
coord body response to stress
assc with fight r flight
what are simp and paarsymp not
not reciprocal
what does parasymp do
increased sec
vasodilation
increase watery saliva
what does simp do
alpha 1 and beta 2 adregenic receps
increased sec
organic components increased
vasoconstriciton
what is the axon like for parasymp
long pre - ACh
short post - ACh
what are the asscnerve with parsym
facial - summand and sibling glands
glossoPh - parotid and lingua
what are the gang affected by parasymp
remaks
otic
submandibular
pterygopalatine
what is receptor for parasymp
muscarinic
what si the result of parasymp
increase watery vol of saliva
smallinc organic comps
contract myopia cell sin acini
what is the antagonist of parasymp
atropine
what issymp axon like
short pre - ACh
long post - NA (ACh at sweat glands)
when does FReys syndomr occur
after parotid surgery
what is greys syndorme
damage to auriulotemoral nerve
paarsymp to salivary gland
simp to facial sweat gland
what causes freys
regeneration of damged nerves mixed and misdirected
salivary nerve inv to sweat glands
what is the result of freys
activation of sweat glands to gustatory stimulus
what are the neural controls of pro facial blood vessels
autonomic - simp vasoaons/dilator
afferent - axon reflex - triple response
what is triple response
blunt trauma effects 3 steps - red reaction or flush - flare - wheal
what siit eh red reaction or flush
cap dilation
histamine release from damaged tissue
LA no effect
what is flare
arteriolar dilation
increased blood flow
LA abolish flare
what is wheal
increase cap perm
histamina releade
what parts of triple response mediated by axon reflex
flare and wheal
what are the hormonal controls of pro facial vessels
systemic - adrenaline
local inflam - bradykinin
what are the metabolic controls of pro facial vessels
metabolites
carbon dioxide
vasodiltion
what is the normal flow to pulp
40-50ml/min/100g tissue
what is the control of pulpal blood vessels
efefrent - symp vasocon or vaso di
afferent - pain, inc blood flow, inc cap perm inc sensitivity
what are the local factors affecting pulpal blood vessels
bradykinin
what is the description of pulpal blood vessels
constat volume problem
what is the constant volume problem
local vasodi inc vol in area inc pressure close vessels ischaemia death pulp
what is ischaemia
reduced blood flow
reduced nutrient s and metabolites
what is pulp death due to
stasis of blood in vessels
what is the acton of alpha 1 adrenoreceptor
smooth muscle contraction
vasoonstrition
what si the agonist and antagonist
agonist - adrenaline
antag - phentolamine
what is the action of beta 1 adrenoreceptors
heart muscle contraction
what is agonist and antag of beta 1
agonist - adrenaline
antag - propranolol, atenolol
what is the action of beta 2 adrenoreceptoos
smooth muscle relax
what is the agonist and antag of beta 2
agonist - adrenaline and salbutamol
antag - propanolol
what are some beta blockers
propanolol and atenolol
