ANS Oro Facial Aspects Flashcards

1
Q

what does ANS do

A

control body systems except skeletal muscle

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2
Q

what does the ANS regulate

A

involuntary function

  • smooth muscle
  • cardiac
  • gland sec
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3
Q

what awesome untrained aspects of ANS

A

urination

defaction

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4
Q

what does parasymp do

A

coordinates the bodys haemostatic functions

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5
Q

what does symp do

A

coord body response to stress

assc with fight r flight

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6
Q

what are simp and paarsymp not

A

not reciprocal

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7
Q

what does parasymp do

A

increased sec
vasodilation
increase watery saliva

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8
Q

what does simp do

A

alpha 1 and beta 2 adregenic receps
increased sec
organic components increased
vasoconstriciton

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9
Q

what is the axon like for parasymp

A

long pre - ACh

short post - ACh

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10
Q

what are the asscnerve with parsym

A

facial - summand and sibling glands

glossoPh - parotid and lingua

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11
Q

what are the gang affected by parasymp

A

remaks
otic
submandibular
pterygopalatine

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12
Q

what is receptor for parasymp

A

muscarinic

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13
Q

what si the result of parasymp

A

increase watery vol of saliva
smallinc organic comps
contract myopia cell sin acini

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14
Q

what is the antagonist of parasymp

A

atropine

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15
Q

what issymp axon like

A

short pre - ACh

long post - NA (ACh at sweat glands)

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16
Q

when does FReys syndomr occur

A

after parotid surgery

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17
Q

what is greys syndorme

A

damage to auriulotemoral nerve
paarsymp to salivary gland
simp to facial sweat gland

18
Q

what causes freys

A

regeneration of damged nerves mixed and misdirected

salivary nerve inv to sweat glands

19
Q

what is the result of freys

A

activation of sweat glands to gustatory stimulus

20
Q

what are the neural controls of pro facial blood vessels

A

autonomic - simp vasoaons/dilator

afferent - axon reflex - triple response

21
Q

what is triple response

A
blunt trauma effects 
3 steps 
- red reaction or flush
- flare
- wheal
22
Q

what siit eh red reaction or flush

A

cap dilation
histamine release from damaged tissue
LA no effect

23
Q

what is flare

A

arteriolar dilation
increased blood flow
LA abolish flare

24
Q

what is wheal

A

increase cap perm

histamina releade

25
Q

what parts of triple response mediated by axon reflex

A

flare and wheal

26
Q

what are the hormonal controls of pro facial vessels

A

systemic - adrenaline

local inflam - bradykinin

27
Q

what are the metabolic controls of pro facial vessels

A

metabolites
carbon dioxide
vasodiltion

28
Q

what is the normal flow to pulp

A

40-50ml/min/100g tissue

29
Q

what is the control of pulpal blood vessels

A

efefrent - symp vasocon or vaso di

afferent - pain, inc blood flow, inc cap perm inc sensitivity

30
Q

what are the local factors affecting pulpal blood vessels

A

bradykinin

31
Q

what is the description of pulpal blood vessels

A

constat volume problem

32
Q

what is the constant volume problem

A
local vasodi
inc vol in area
inc pressure
close vessels
ischaemia
death pulp
33
Q

what is ischaemia

A

reduced blood flow

reduced nutrient s and metabolites

34
Q

what is pulp death due to

A

stasis of blood in vessels

35
Q

what is the acton of alpha 1 adrenoreceptor

A

smooth muscle contraction

vasoonstrition

36
Q

what si the agonist and antagonist

A

agonist - adrenaline

antag - phentolamine

37
Q

what is the action of beta 1 adrenoreceptors

A

heart muscle contraction

38
Q

what is agonist and antag of beta 1

A

agonist - adrenaline

antag - propranolol, atenolol

39
Q

what is the action of beta 2 adrenoreceptoos

A

smooth muscle relax

40
Q

what is the agonist and antag of beta 2

A

agonist - adrenaline and salbutamol

antag - propanolol

41
Q

what are some beta blockers

A

propanolol and atenolol