Abomasal Disorders Flashcards

1
Q

What is the most common displacement of the abomasum

A

LEFT DISPLACED ABOMSAUM (lda)

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2
Q

What breed is predisposed to LDA

A

Dairy cattle, especially deep chested Holsteins

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3
Q

How is age a factor in LDA

A

Less common in primiparous/heifers
Older cattle higher risk

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4
Q

How is stage of lactation a factor in LDA

A

80% of LDAs within 4 weeks postpartum

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5
Q

What are other risk factors besides age in LDA

A

Infectious disease
mastitis, metritis, enteritis, lameness
Metabolic disease
hypocalcaemia, negative energy balance

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6
Q

How is Transition cow nutrition a factor in LDA

A

Reduced dry matter intake pre-calving
High concentrate intake pre-calving

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7
Q

LDA diagnosis- History

A

Reduced feed intake
Drop in milk production
Decline in rumination time (if monitored)
Recent calving
Concurrent postpartum disease

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8
Q

LDA diagnosis- Clinical Exam

A

Characteristic “ping” on auscultation and ballottement
Dull

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9
Q

What is pH level of fluid from abdominocentesis

A

<3.5

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10
Q

Three catergories to correct LDA

A

Conservative management
Percutaneous fixation
Surgical fixation

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11
Q

What does conservative manage of LDA entail

A

Cast & roll
Little evidence to support the use of medical management alone High recurrence rate

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12
Q

What does Percutaneous fixation of LDA entail

A

Cast into dorsal recumbency
Clip and surgical scrub from sternum to umbilicus
Instill 10-20ml local anesthetic at surgical sites
With cow in dorsal recumbency identify “ping”
Insert trocar at caudal site, push toggle through then clamp suture end to hold
Move ~10cm up to cranial site and repeat process
Tie both suture together loosely
Leave one hand’s width between suture and body wall
Risk of necrosis if too tight
Roll cow clockwise into sternal recumbency

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13
Q

Advantages of Percutaenous fixation

A

Quick and easy
Minimal specialist equipment needed
Inexpensive

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14
Q

Disadvantages of percutaenous fixation

A

Safety risk with casting and rolling
Require 2+ assistants
Risk of ventral fistula formation
Blind technique so risk of incorrect fixation

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15
Q

What does surgical correction of LDA entail

A

right flank incision
Locate then deflate abomasum
Reposition abomasum

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16
Q

Differentiate advantages between right and left flank approach to correct LDA

A

Right
Standing animal
Do not require assistants
Good access for abdominal exploration
Ensures correct fixation of the omentum or abomasum

Left
Best technique in pregnant animals (space to work)
Standing animal
Good access to the abomasum so can treat adhesions or ulcers
Ensures direct fixation of the abomasum

17
Q

Differentiate disadvantages between right and left flank approach to correct LDA

A

Right-
Technically more difficult
More invasive surgery with risk of wound infection or peritonitis
Risk of recurrence if pexy fails
Wrong position
Omentum too friable
Risk of pyloric stenosis (pyloropexy)

Left
Requires assistant
Risk of damage to the milk vein
Risk of ventral fistula formation

18
Q

What approach is contraindicated in pregnant cow

A

Paramedian approach

19
Q

Does Right displaced abomasum and volvulus or LDA have more clinical signs

A

RAV more severe clinical signs
Signs of colic
Elevated heart rate and respiratory rate
Signs of shock and endotoxaemia
Palpable distended viscus behind last rib

20
Q

What surgical approach to use for RDA

A

Only right flank approach appropriate (not left flank or paramedian)

21
Q

Dietary causes of impaction

A

Fibrous diet low in protein and energy
Heavy ingestion of sand
Poor water availability

22
Q

Non-dietary causes of impaction

A

Abomasal hypomotility in postpartum dairy cattle
Any cause of reduced abomasal outflow e.g Vagal nerve damage

23
Q

Most common reason for loss of wall intergrity

A

Abomasal ulceration

24
Q

Outline the 4 types of ulceration

A

Type 1- Non-perforating ulcer without bleeding
No or minimal clinical signs

Type 2- Non-perforating ulcer bleeding into the abomasal lumen
Melena and signs of anaemia

Type 3- Melena and signs of anaemia
No melena, often minimal signs if walled off

Type 4- Perforating ulcer with generalised peritonitis
No melena, non-specific sick cow depending on severity

25
Q

Abomasal ulceration diagnosis

A

C/S
Faecal occult blood test
Haematology and biochemistry
Low PCV and total protein (Type II)
Inflammatory response (Type III or IV)
Ultrasound of the ventral abdomen +/- abdominocentesis
Exploratory laparotomy

26
Q

Abomasal ulcer treatment

A

Offer high fibre diet
Supportive therapy
Blood transfusion (Type II)
Oral antacids (magnesium oxide, aluminium hydroxide)
Cimetidine, ranitidine, omeprazole are not licensed in food producing animals in UK
Surgery (Type III or IV)

27
Q

What is an abomasal fistula

A

Infrequently develop following abomasopexy
Intraluminal suture placement can allow leakage of abomasal content and weakening of the incision

28
Q
A