69. Misc Ortho Dz Flashcards

1
Q

define osteochonrodysplasia

A

type of constitutive bone/cartilage diseaseoften heritable=abN endochondral and/or intramembranous ossificationdwarfism (disproportionate or proportionate)

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2
Q

T/F most osteochondrodysplasias are disproportionate dwarfism

A

TRUE osteochondrodysplasias and nutritional deficiencies are disproportionate dwarfismUSUALLY dwarfism w disproportional SHORT limbs

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3
Q

Hemimelia

A

APPENDICULAR DYSOSTOSEScongenital complete or partial absence of one of more BONESusually unilateralnot always heritable (except Chihuahuas, DSH, Siamese)usually non painfulterminal (transverse, longitudinal)intercalary (transverse, longitudinal)

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4
Q

Hemimelia classification

A

terminal (transverse, longitudinal)-absence of all or some of the bones of a limb distal to a certain point . intercalary (transverse, longitudinal)-absence of all or some of the middle bones of a limb with the proximal and distal bones still present (middle one is missing)transverse: complete absence across the limbs widthlongitudinal: absence along the medial or lateral side of a limb

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5
Q

radial hemimelia

A

usually pre-axial (medial) intercalary longitudinalusually unilateral, marked varus, unable to bear weight, flexure deformitysurgery: declaw (palliative), arthrodesis + reconstruct radial defect (graft, distraction osteogenesis), amputate

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6
Q

dimelia

A

APPENDICULAR DYSOSTOSEScongenital duplication of the whole or part of a limbdog (not seen in cat)figure out which is nonfunctional and amputate it

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7
Q

ectrodactyly

A

APPENDICULAR DYSOSTOSEScongenital digital cleft formation extending between metacarpal bonesmost btwn 1 and 2 metacarpal bonesunilateral usually of thoracic limbs

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8
Q

polydactyly

A

APPENDICULAR DYSOSTOSESpresence of one or more extra digitspreaxial (medial); post axial (lateral)normal autosomal recessive trait in St Bernards, Collies—medial pelvic limb

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9
Q

syndactyly

A

APPENDICULAR DYSOSTOSESlack of ddx btwn two or more digits simple vs complexcomplete vs incomplete

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10
Q

HOD signalment and presentation

A

2-6 months of ageLarge -giant breed dogs (Weimaraners)Males are 2.3 times more likelyMetaphyseal swelling (distal RU and tibia most common)Often bilateral+/- pyrexia, pain, warmth

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11
Q

List pediatric bone diseases

A

–Hypertrophic osteodystrophy HOD–Panosteitis–Craniomandibular osteopathy–Retained ulnar cartilagenous core–Legg calve perches disease–Femoral capital physeal dysplasia–Multiple cartilaginous exostoses

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12
Q

Possible etiologies for HOD

A

Vitamin D insufficiency (scurvy)Heritable (Weimaraners 0.68 inherited immunodeficiency w low Ig)Infection (blood culture)Canine distemper virus

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13
Q

Pathognomonic radio graphic sign of HOD

A

Lucent line in the metaphysis parallel to a narrow zone of increased radiodensity immediately immediately adjacent to the physiscan also see see metaphyseal “flare” or widening of the bone

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14
Q

Panosteitis signalment and disease progression

A

Self limiting Inflammatory disease of the bone marrow of long bonesShifting leg lameness w long bone pain5-12 months - up to 5 YO (older than HOD)Large-giant breed Males 4:1eosinophilic inflammation!

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15
Q

Radiographic diagnosis of panosteitis

A

Early (rare)- decreased radiodensity evident in medullary cavity near nutrient foramen (changes in intraosseous pressure to increase with vascular congestion–maybe from protein accumulation)Later- increase in medullary opacity w granular pattern or w loss of the normal trabecular pattern+/- Periosteal bone formation as it progresses the medullary canal becomes more diffuse and homogenous

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16
Q

Treatment of panosteitis

A

RestAnalgesicsSelf limiting Benzopyron (not available) proteolytic substance to decrease intraosseous pressure+/- recurrence

17
Q

Craniomandibular osteopathy CMO

A

<6-12 months of ageWest highland white terriers, cairn, Scottish terriersSuggests heritable causeUnilateral or bilateral symmetric irregular osseous proliferation of mandibles and/or temporal bonesPain, dysphagia or inability to eat/drinkSalivation Self limiting at 11-13 mo of agemale = female

18
Q

Retained ulnar cartilagenous core

A

Viable hypertrophic chondrocytes that project from the distal ulnar growth plate into the distal metaphysis with failure to convert to metaphyseal bone. Can lead to ALD from radial bowing, elbow incongruity, carpal valgusradiolucent core (triangle) of cartilage distal to metaphysis +/- surrounded by a zone of sclerosisMay need surgical correction if severe

19
Q

Legg calve perthes

A

Avascular necrosis of femoral headNo inflammatory local ischemiaSmall breed dogs– toys & terriers1. necrosis2. fragmentation (apple core)3. collapse/thickening 4. revascularize/reossification

20
Q

how many bilateral presence of Legg Calve Parthes

A

12-17%

21
Q

treatment for legg calve perthes

A

only 25% resolve lameness with conservative mgmt–FHO–THRpx 84-100% with surgery

22
Q

signalment for slipped capital femoral epiphysis

A

through the growth platenontraumatic ( NOT SH 1 fracture)CATS–male neutered young overweight (may be related to early neuter and hypotestosteronism)often BILATERAL

23
Q

multiple cartilaginous exostoses

A

young dogsold cats (FeLV)may or may not have malignant transformationmay occur from secondary migration of chondrocytes from the physis into metaphysissingle (osteochondroma) or multiple well circumscribed calcified lesionsaffects vertebra, ribs. long bones

24
Q

most common reason for surgical excision of cartilaginous exostosis

A

vertebral exostosis causing spinal cord compression

25
Q

causes of metabolic bone disease

A

–primary HPT–nutritional or renal secondary HPT–vit D toxicoses–hypo vit D (ricketts)–hypo/hyper vit A–hypovit E**most often affect Ca metabolism/homeostasis—osteopenia and brittle bones seen

26
Q

osteogenesis imperfecta

A

metabolic bone diseasedisruption in collagen I

27
Q

hypertrophic osteopathy

A

HO—older dogsassociated with neoplasia (abdominal, thoracic) can also be nonneoplastic (spirocerca lupin, heart worm dz)periosteal reaction of DISTAL (diaphyseal) extremities due to peripheral blood flow changes stimulating an overgrowth of vascular connective tissue and periosteal proliferation radiographs appear aggressive or nonaggressive. look for palisade formation of bone

28
Q

disseminated idiopathic skeletal hyperostosis

A

spinal and extraspinal manifestation of heavy bone formationlimited spinal ROM1. continuous flowing calcification > 3 vertebra2. relative preservation of disc space3. periarticular osteophytosis around zygapophyseal joint4. pseudoarthrosis btwn spinous processes5. periarticular osteophytosis and calcification of soft tissuesneed 4 out of 5 to make dx

29
Q

T/Fbone cysts are lined by epithelium

A

FALSEbone cysts are lined by fibrous connective tissueyounger larger breed dogs (male 2:1), Dobiesradiographs reveal expansile, locally aggressive lucent lesion with minimal to no periosteal reaction, most are eccentrically located, most involve metaphysis and diaphysis

30
Q

acute caudal myopathy

A

limber tailpointers and labradorspainful at base of tail and tail is flaccidusually after extreme exertionself limiting but can recur

31
Q

swimmer syndrome

A

flat-pup, turtle-pupneonates affectedsternal with splayed legspectus excavatumneed physical rehab in first 3 weeks, prognosis can be good

32
Q

puppy carpal laxity syndrom

A

carpal hyperextension >180 degrees (dropped hock)OR carpal hypo extension < 180 degrees uni or bilateral; self limited +/- splints/braces65% recover in two weeks

33
Q

normal flexion in carpus vs hypoflexion

A

flexion 20-30 degreeshypoflexion (older dogs) >30 degrees

34
Q

iliopsoas muscle injury

A

attaches to lesser trochancter of femurdiscomfort on extension and internal rotation of hipneed rehab and rest +/- NSAIDssevere cases—tenomyectomy