Week 4.2 Interventions for weakness 1 Flashcards

1
Q

TF: there is strong evidence and support for efficacy of strength training in people with neurological disorders

A

true

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2
Q

strength training can do what to the cortex

A

increase cortical excitability

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3
Q

why are there fewer motor neurons areas activated after training

A

because of inhaled efficacy of synapses

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4
Q

what does strengthening do to motor unit recruitment and neural drive

A

increases in neural drive, and changes in recruitment

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5
Q

TF: strengthening does not improve motor unit recruitment

A

false, it does

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6
Q

do we want longer or shorter treatments

A

longer

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7
Q

how can we customize a strengthening intervention

A

by making it fit the characteristics of the patient, like age, co-morbidities, functional level, balance ability, stage of disease.

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8
Q

why is it important to have ongoing evaluation and progression

A

we need continual challenge to the neuromuscular system to adapt and facilitate optimal recovery

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9
Q

what is 5D. what will improve function

A

non progressive disorders of the CNS, specifically related to site and severity of the BI. correlates with functional status, and remediation will improve function

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10
Q

what is 5E

A

progressive disorder of the CNS, with a progressive decline with strength. So MS, PD, ALS

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11
Q

what is 5G

A

acute or chronic polyneuropathies like GBS, so be careful with recovery phase not to over fatigue

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12
Q

what is 5H

A

non progressive disorders of the SC. weakness or total paralysis, like SCI. so the recovery depends on extent of damage, what is spared and neuroplasticity of the SC

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13
Q

is the weakness on the ipsilateral or contralateral side of the lesion

A

contralateral side, although I guess there is weakness on the ipsilateral side too sometimes because of tracts that are bilateral or do not decussate

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14
Q

after a stroke, is flexion or extension affected more

A

equally

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15
Q

what happens to force generation and production after a stroke

A

generation is reduced, and production is slower

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16
Q

what causes decreases in force production after a stroke

A

central activation deficits, transsynaptic degeneration and secondary changes in the muscles.

17
Q

what is the cause of weakness after an acquired brain injury

A

central neural activation deficits, impaired ability to active MU

18
Q

Is it common for people who have acquired BI to have disuse atrophy

A

yes

19
Q

what happens to axons and nerve conduction in MS

A

axon degeneration and reduced or blocked nerve conduction

20
Q

what happens to force production in MS

A

reduced and slow