Week 4.2 Interventions for weakness 1 Flashcards
TF: there is strong evidence and support for efficacy of strength training in people with neurological disorders
true
strength training can do what to the cortex
increase cortical excitability
why are there fewer motor neurons areas activated after training
because of inhaled efficacy of synapses
what does strengthening do to motor unit recruitment and neural drive
increases in neural drive, and changes in recruitment
TF: strengthening does not improve motor unit recruitment
false, it does
do we want longer or shorter treatments
longer
how can we customize a strengthening intervention
by making it fit the characteristics of the patient, like age, co-morbidities, functional level, balance ability, stage of disease.
why is it important to have ongoing evaluation and progression
we need continual challenge to the neuromuscular system to adapt and facilitate optimal recovery
what is 5D. what will improve function
non progressive disorders of the CNS, specifically related to site and severity of the BI. correlates with functional status, and remediation will improve function
what is 5E
progressive disorder of the CNS, with a progressive decline with strength. So MS, PD, ALS
what is 5G
acute or chronic polyneuropathies like GBS, so be careful with recovery phase not to over fatigue
what is 5H
non progressive disorders of the SC. weakness or total paralysis, like SCI. so the recovery depends on extent of damage, what is spared and neuroplasticity of the SC
is the weakness on the ipsilateral or contralateral side of the lesion
contralateral side, although I guess there is weakness on the ipsilateral side too sometimes because of tracts that are bilateral or do not decussate
after a stroke, is flexion or extension affected more
equally
what happens to force generation and production after a stroke
generation is reduced, and production is slower
what causes decreases in force production after a stroke
central activation deficits, transsynaptic degeneration and secondary changes in the muscles.
what is the cause of weakness after an acquired brain injury
central neural activation deficits, impaired ability to active MU
Is it common for people who have acquired BI to have disuse atrophy
yes
what happens to axons and nerve conduction in MS
axon degeneration and reduced or blocked nerve conduction
what happens to force production in MS
reduced and slow
