Week 3.10 Ataxia Flashcards

1
Q

what does an ataxic gait look like

A

like you are drunk!

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2
Q

damage to the ____ causes ataxia

A

cerebellum

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3
Q

the cerebellum is responsible for

A

coordinating movement

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4
Q

what does the cerebellum compare

A

the intended output and actual movement

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5
Q

what does the cerebellum do in preparation of movement

A

it makes predictive and anticipatory modifications

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6
Q

the cerebellum has a huge role in motor…

A

learning

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7
Q

TF: the cerebellum learns, memorizes and stores motor programs

A

true

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8
Q

what is so important about the role of adaptation in the cerebellum

A

the cerebellum must adapt to the changing environment, and the speed of the changing environment.

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9
Q

TF the cerebellum controls balance and equilibrium

A

true

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10
Q

TF: the cerebellum does not control motor tone

A

false, it does

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11
Q

the cerebellum makes sure movement has what 4 things

A

direction
extent
force and timing

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12
Q

how does the cerebellum affect speech and eye movements

A

responsible for all of those !

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13
Q

what are some signs and symptoms of cerebellum infarct, and what side of the lesion

A

nausea, vomit, ataxia, nystagmus, headache all ipsilateral to the lesion

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14
Q

what happens in a lateral lesion and a vermin/flocculonodule lesion

A

lateral: limb ataxia

vermis and flocculonodule: more midline, so the drunk gait.

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15
Q

name some things that result from cerebellar dysfunction

A
disorganized movements 
impaired balance and postural control  hypotonicity 
dysmetria 
decomposition 
ataxia 
dysdiadochokinesia 
tremor 
asthenia 
dysarthria 
occular dysmetria
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16
Q

what are some acquired mechanisms of cerebellar damage

A

stroke, tumor, structural changes, toxicity (alcohol drugs), immune mediated (MS, Gluten), trauma, infection (cerebellitis) or endocrine (hypothyroidism)

17
Q

what are some degenerative non-hereditary causes of cerebellar damage

A

MSA

idiopathic late onset cerebellar ataxia.

18
Q

what are some hereditary causes of cerebellar damage

A
autosomal dominant disorders (like episodic of spinocerebellar ataxia's)
autosomal recessive (Friedrich, early onset cerebellar)
X-linked disorders (mitochondrial, fragile X associated tremors)
19
Q

define/describe ataxia

A

without order, incoordination, slurred speech, stumbling, falling, incoordination, trouble eating and swallowing, eye movement abnormalities, tremors, cardiac issues

20
Q

what are 3 traditional function and activity outcome measures

A

FIM
Balance/postural control measures
gait speed

21
Q

what are 2 ataxia specific outcome measures

A

International cooperative ataxia rating scale (ICARS)

Scale for the assessment and rating of ataxia (SARA)

22
Q

TF: there is a lot of evidence for treatment of ataxia

A

false

23
Q

what do we treat in patients with ataxia

A

the symptoms

24
Q

what kinds of things can we treat in patients

A

postural stability
gait
balance
accuracy of limb movements

25
Q

do we do an intensive long or short term motor training

A

long term

26
Q

what should the HEP focus on

A

static and dynamic balance activities like sitting standing and walking

27
Q

TF: there is a lot of evidence for BW supported treadmill training

A

false, not much evidence

28
Q

can we use biofeedback in patients with ataxia

A

yes, like a mirror, EMG, or do it in a small space so they can bump into things

29
Q

do we want to increase or decreases DOF, and how

A

decrease, by adding an AD, resistance, kneeling, 1/2 kneeling

30
Q

activity should focus on what 3 things

A

stability
co-contraction
midrange control

31
Q

can we use resisted movements

A

yes

32
Q

what are some compensatory strategies

A
slow it down ,
reduce the number of moving segments 
widen BOS 
minimal distractions 
weighting axial and limb 
AD
orthotics (decrease DOF)