unit 9 Control of Body Movements Flashcards

1
Q

Types of proprioceptors and their actions: Golgi Tendon Organs (GTOs)

A

located at junctions of tendons and muscle fibres

consist of nerve endings attached to bone that monitor muscle tension (esp during isometric contractions_

when increased tensions= activate GTO’S = less active fibre= relaxation

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2
Q

Types of proprioceptors and their actions: muscle spindles

A

detects muscle stretch and it’s length

made of several skeletal muscle cells called intrafusal fibres

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3
Q

intrafusal fibres

A

centre region= not contractible (lack myosin and actin) wrapped in dendrites of 1st order

ends of fibres= have actin and myosin and are innervated by gamma motor neurons and can contract

generates most force and are larger/main fibre

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4
Q

alpha-gamma co-activation

A
  1. alpha motor neuron fires, gamma motor neuron fires
  2. muscle and intrafusal fibres both contract
  3. stretch in centres of intrafusal fibres unchanged. firing rate of sensory neuron remains constant
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5
Q

properties of skeletal muscle: myotatic unit:

A

collection of neural pathways controlling. single joint and antagonist and agonist

includes the sensory, alpha lower neuron, and the interneurons connected to them

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6
Q

properties of skeletal muscle: reciprocal inhibition

A

inhibition of contraction in antagonist at the same time agonist contract

directly w/ the alpha lower motor neuron that innervates the agonist (excitatory synapse)

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7
Q

excitatory vs inhibitory interneuron

A
  1. carries signal to alpha motor neuron that goes into agonist
  2. prevents signalling alpha motor neurons going to the antagoinist to allow agonist to contract
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8
Q

what is something reciprocal inhibition not have

A

efferent. (stimulus and receptor= same, response is opp)

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9
Q

levels/phases of motor control: level 1

A

planning movement you are unconsciously about to do

involves the premotor cortex and association areas of the brain

receives input from cerebellum and basal nuclei

signals to primary motor cortex which contains upper motor neurons (synapse w/ lower MN and those synapse w/ muscle)

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10
Q

levels/phases of motor control: level 2

A

initiating movement (primary motor cortex)

contains cell bodies of upper MN that are mapped to control skeletal cells in specific regions of the body

stimulation of the upper MN by the planning areas creatse signals that travel via the corticospinal tract into brainstem or spinal

they synapse w/ lower MN that innervate skeletal muscle

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11
Q

levels/phases of motor control: level 3 brainstem nuclei

A

some contain cranial nerves (location where synapses are)

others contains cell bodies of upper MN responsible for coordination of large muscle groups used to maintain posture and locomotion

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12
Q

levels/phases of motor control: level 4 spinal cord

A

contains cell bodies of alpha motor neurons

contains networks of neurons that form central pattern generators (CPGs)

network of oscillating inhibitory/excitatory neurons that create units responsible for rhythmic contraction and relaxation of flexor and extensor muscles at a joint

these networks allow repetitive movements.

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13
Q

for level 4 spinal cord how do we start/ stop movement

A

signals from the cortex needed to stop/start signal

CPG’s (central pattern generators) maintain it once it’s going

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14
Q

levels/phases of motor control: 5- cerebellum

A

Plans and inhibits movement

Stores planned movements and compares it to actual using sensory input from sources like proprioceptors and correct if needed

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15
Q

levels/phases of motor control: 6 basal nuclei

A

Aid in planning movement

Surpass unwanted motion (esp when at rest and bot moving)

Damage to this= movement disorders like parkinsons and hun

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16
Q

levels/phases of motor control: 7 corticospinal (direct) pathway

A

Signals from the cortex to the skeletal muscle

17
Q

What is cerebellum affected by

A

Drugs/alcohol- uncoordinated movement

18
Q

How does upper mn signal to lower mn

A

Uses neurotransmitter glutamate

19
Q

Destruction of upper Mn

A

Spinal reflex arc still present since lower alpha MN and sensory neuron synapse still intact

20
Q

Destruction of upper Mn: spastic paralysis

A

Muscle tone increases, no atrophy of muscles- no signals from brain

21
Q

Destruction of upper Mn: exaggerated reflex

A

Like knee jerk reflex

Caused by lack of inhibitory signals from brain which makes reflexes stronger than usual

22
Q

Destruction of lower alpha MN

A

Spinal reflex arc= absent

23
Q

Destruction of lower alpha MN: flaccid paralysis

A

Signals from brain/sensory neurons cannot reach muscle

Decrease muscle tone and atrophy of muscle

24
Q

Destruction of lower alpha MN: complete loss of reflexes in affected area

A

Can’t stimulate muscle when it receives signals from sensory neurons