Trauma 2/28/17 Flashcards
zones approach (old approach) to neck trauma how new approach updates
III - toward head - worried about arteries - but tough to explore - get arteriogram
II - middle - arteries, trach, espophagus - go to surgery, easy to explore
I - toward thorax - arteries, trach, esophagus - scary to operate as things can retract / fall into thorax - get arteriogram, esophogram, bronchoscope
the above if pt stable
if unstable, to surgery regardless of zone
new approach - OR if unstable, CT angiogram if stable but symptomatic (evals arteries, esoph, trach), observe if asymptomatic or CT angio negative, to OR if CT angio positive, repeat CT angio if asymptomatic becomes symptomatic
…. all regardless of zone.
so basically CT angiogram changed things and is the first step in eval if stable symptomatic… to OR if unstable still… use zone method if CT angio unavailable
define penetrating neck injury
platysma has been disrupted
hard signs vs soft signs of instability w neck trauma
hard
- airway gurgle, stridor, loss of airway
- vascular expanding hematoma, pulsatile (arterial) bleeding, stroke, frank shock
soft
-signs of air digestive or vascular injury that are not that bad - dysphonia dysphasia, subq emphysema / crepitus, non-pulsatile bleeding non-expanding hematoma
dorsal columns medial lemniscus system
where in spinal cord transverse cut
where decussate
function
posteromedial
decussates up in… brain or brainstem… not spinal cord
proprioception
(fin feels the way)
vibratory sense
(shivers down the back)
ALS
aka aka aka
where in spinal cord transverse cut
where dessucate
function
anterolateral system
ventrolateral system
spinothalamic tract
anterolateral (and MEDIAL - DECUSSATES anterior to spinal canal)
pain
temperature
(vent the pain and temperature)
(spinoThermometer tract .. put thermometer in spine it will be painful)
corticospinal tract
where in spinal cord transverse cut
function
lateral
(decussates way up in pyramids i think… not in spinal cord)
motor
DCML
STT
CST
where in transverse cut of spinal cord
where decussate
what function
DCML posteromedial, up in brain/stem whatever not in spine, proprioception (fin) vibratory sense (chills)
STT anterolateral/ventrolateral but decussates at the level - pain and temp (put spinothermometer into spine will get pain and temp)
CST lateral slightly dorsal - decussates up in pyramids - motor
brown sequard syndrome
hemisection of spinal cord
(almost always knife trauma to neck)
contralateral pain/temp loss below level, bilateral at level (STT/ALS/VLS)
ipsilateral motor loss… flaccid LMN at level, spastic UMN below level… (CST)
ipsilateral vib and prop loss below level (DCML)
cord compression
sx
dx
tx
fnd
ed (erectile dysfunction)
u/b incontinence
dexamethasone (to reduce inflammatory edema, which is the cause of the compression… not the trauma itself)
MRI AFTER dex because want to treat asap, MRI takes a while uncomfortable maybe dangerous
anterior cord syndrome
dcml spared (prop and vib)
LMN at level, UMN below level (CST)
loss of pain temp below (STT ALT VLT)
central cord syndromes
syringomyelia (swelling of central canal)
hyperextension in elderly
Loss of pain/temp, LMN AT THE LEVEL ONLY…eg usually CAPE-LIKE distribution down back and arms from cervicle spine lesion
(other syndromes affect below the level too)
posterior cord syndrome
lose dcmls
(sensation below the level)
motor (cst) pain and temp (stt als vls) preserved
evaluate airway patency in emergency situation
speaking full sentences
no accessory muscle use
bilateral breath sounds
-patent
can see expanding hematoma
cutaneous emphysema
-urgent airway
GCS less than 8, intubate
gurgling, gasping, stridor
-emergent airway - INTUBATE
what is the significance of inspiratory stridor
entrance to entire airway is collapsing
evaluate breathing in emergency situation
full breaths, satting well on pulse-ox, probably ok
deeper look:
pCO2 on ABG measures ventilation (MV = TVxRR)
pO2 on ABG (or pulse ox is almost as good really) measures oxygenation (controlled with PEEP, FiO2)
*don’t be fooled by ETCO2 endotracheal capnography (40 is normal) just tells you that ET tube is in the right place, assists in ET tube placement
eval for circulation in emergency situation
pale cold diaphoretic
SBP v90 MAP v65 not absolutely scary but should get antenna going
U output v.5cc/kg/hr
manage airway in trauma
jaw thrust finger sweep O2 nc, face tent, non-rebreather bag valve mask intubate cric (done in ed) trach (done in or in more controlled circumstances for longer placement)
MAP =
MAP = CO x SVR MAP = HRxSV x SVR MAP = HR x PreloadxContractility x SVR
General concepts that can contribute to shock
HR too fast or slow
Preload down… via volume (hemorrhage) or obstruction (tension pneumo, pericardial tamponade, PE)
contractility down (MI, CHF, contusion)
SVR down (sepsis anaphylaxis, anesthesia, spinal trauma)
tf
hemorrhage picked up by Hb being low
careful
Hb is a concentration, don’t pick it up until fluid shift eg after giving fluids that anemia becomes evident on lab
abdominal bleed in trauma bay, how to dx, manage
FAST US to dx
apply pressure
take to OR - put in IVs, type and cross, IVF, Blood as necessary on the way over
pericardial tamponade in trauma bay, how to dx, manage
FAST - ECHO
pericardiocentesis
Warm vs cold hypotension ddx
Warm svr problem
-sepsis anaphylaxis anesthesia spinal trauma
Cold a cardiac output (hr sv contractility) problem, svr contracts in reponse
-hemorrhage tension pneumo tamponade chf mi contusion
Normal tidal volume
500cc
High tidal volume, ventilator, hypotension, think…
Pneumothorax
In general, keep tidal volumes lower (500cc is normal) and respiratory higher if necessary to prevent pneumothorax
Pulsus paradoxus
Deeper drop in sbp w inspiration than normal (more than 10mmhg, so radial pulses may disappear during inspiration)
From pressure around hear preventing expansion for more venous return, r heart expands into left heart, increasing afterload and dec stroke volume so sbp lower
Eg in tamponade pericarditis copd osa
(Normally sbp drops a little from pooling in pulm vasculture)
Define massive PE
Causes hypotension
S1Q3T3
deep s wave in lead 1
Deep q wave lead 3
Inverted t wave lead 3
Sign of acute r heart strain
Eg PE (only 10% pe’s will have s1q3t3)
Similar to pattern in left posterior fascicular block
Lung and heart sounds Pneumothorax Pericardial tamponade PE MI
Pneumo reduced lung normal heart
Tamponade normal lung distant heart
PE normal both
MI normal both if R sided
Pulm edema normal heart if L sided
In emergency, order of attempts at venous access
Peripheral venous line x3 attempts
(or at least Say you tried 3 times per some veteran medics)
IO intraosseous line (good for 24 hours if need right now, can get fluid up and get more definitive peripheral line later)
….
Central line (jugular, subclavian, femoral)
Is jugular venous access considered central or peripheral?
Internal jugular is central
External jugular is peripheral
When is ng tube with enteral hydration typically called for
For ICU pt getting meds and fluids but not drinking, develops hypernatremia - enteral ng tube hydration for gradual restoration of fluid balance
Sites for central venous access
Internal jugular
Subclavian
Femoral
When is a PICC line typically placed
In non-emergent situation for Pt who needs long term abx, chemo, or becomes difficult to stick (sickle cell pt in 30’s or 40’s)
What is a saphenous vein cutdown and when will it be used
Peripheral venous access via dissecting out saphenous ant to medial malleolus and ligating it, using proximal end to insert a catheter
Not used in too many emergency situations
TF
In tachycardic hypotensive pt w story consistent w hemorrhage, wait for low hb before transfusing
F
Hb will be normal in acute blood loss because it is a concentration. Give fluids, give blood
“Don’t transfuse above 7 or 8 because worse outcomes” does not apply to acutely hemorrhaging pt