Things I Forget Over and Over Again ;) Flashcards
Pre diabetic Range
5.7-6.4
When to consider insulin for diabetes
def @ 10, consider @ 9
Atrial Septal Defect murmur
systolic ejection murmur at pulmonary area
fixed split S2
plus/minus diastolic rumble at tricuspid valve
PDA physiology
connection between pulmonary artery and aorta to shunt blood away from lungs
PDA murmur
continuous machinery murmur at left second ICS
wide pulse pressures, bounding peripheral pulses
Disseminated Zoster
3 or more dermatomes
Indications for Severe C diff Treatment
WBC 15+
Albumin less than 3
Cr 1.5x the prior level
Treatment of systolic CHF
loop
ACEI or ARB
BB
Aldosterone if EF<35%
Pathophysiology of CHF
decreased CO – > Activate RAS + SNS –> systemic vasoconstriction
Systolic HF- main cause
impaired contractility
–ischemic heart disease
Diastolic HF- main cause
impaired ventricular filling during diastole
- HTN
- aortic stenosis
CKD defined as
abnormal markers of kidney damage for 3 months
or GFR less than 60 for more than 3 months
Neonatal vs Infantile Acne
first month of life vs age 3-4 months
Base Deficit
takes out respiratory component so it represents the metabolic acidosis
normal -2 to 2
Albumin in acid base and correction
weak acid
more albumin = more acidic
Add 2.5 to AG for every 1 albumin below 4
Strong Ion Difference
Na - Cl
>38 metabolic alkalosis
<38 NAGMA
Acid/Base pCO2 determination
> 40 resp acidosis
<40 resp alkalosis
Delta Gap Interpretation
more than 2 is metabolic alkalosis
less than 1 is NAGMA or urinary anion loss (DKA, CKD)
Change in Metabolic acidosis
change in c02 = 1.2 x bicarb delta
Change in Metabolic alkalosis
change in co2 = .7 x bicarb delta
Change in Resp Acidosis
acute is .1 x change in co2; chronic is .4 x change in co2
Change in Resp Alkalosis
acute is .2 x change in co2; chronic is .4 x change in co2
Causes of AGMA
GOLD MARK
Glycols, Oxoproline (Tylenol), L-lactate, D-lactate
Methanol, Aspirin, Renal Failure, Ketoacidosis
Causes of NAGMA
HARDUPS
Hyperalimentation, acteozolamide & Addison’s, RTA
Diarrhea, Urteopelvic things, pancreatic fistulas
Saline
Metabolic Alkalosis with LOW urine chloride
responsive Vomiting NG suction Over diuresis post hypercapnia
Metabolic Alkalosis with HIGH urine chloride
non-responsive
HTN: Cushing, Conn, RAS, alkali
no HTN: Low Mag, K, Laxatives, Bartler, Gitelman
Causes of RTAs
type 1 - decreased secretion of H+
type 2 - decreased reabsorption of Bicarb
type 4 - aldosterone def/resistance
Polyuria =
more than 3 L