Theories Flashcards
Theories of Addiction
Physical-Dependence Theory
Positive-Incentive Theory
Disease Model
Incentive-Sensitization Theory
Physical-Dependence Theory
drug addicts take drug to alleviate withdrawal symptoms
Problems with Physical-Dependence Theory
- detoxified addicts return to drug-taking
- some highly addicting drugs (e.g. cocaine) have minimal withdrawal symptoms
- doesn’t explain how addiction process begins
Positive Incentive Theory
the craving for the positive incentive properties of the drug is the primary factor in addiction
Incentive
pleasure producing
Problems with Positive Incentive Theory
- not all drugs are pleasure
- doesn’t explain how addiction begins
- doesn’t address individual differences
- the anticipated pleasure of drug taking is the basis of addiction not so much the pleasurable effects of drug
- drugs act as reinforcers
reinforcers examples
smiles, money, drugs and alcohol, food
Disease Model
- addiction is a disease
- research has identified genetic predispositions to alcohol dependence
Strengths of the Disease Model
explains why not everyone develops a drug problem
-the usual rules that govern behavior will not apply in the disease state
Problems with the Disease Model
- have not identified the disease
- how does a disease make us take drugs?
- lack of control
example, lack of control
drug addicts will say they can’t do anything about drug intake because “I have a disease I can’t do anything about it”
can theories interact?
not one alone explains drug addiction, they all interact to produce addiction
Theory 1 + Theory 2 + Theory 3
Incentive-Sensitization Theory
attempts to explain drug craving
- stimuli that activate the mesolimbic dopamine system have incentive salience
- with repeated use a drug will acquire greater incentive value (sensitization)
incentive salience
- the stimulus is attended to by the organism
- the stimulus motivates behavior towards it
- the organism “wants” the stimulus (anything that makes them feel good)
neuronal evidence
chronic use of several drugs of abuse increase glutamatergic input onto dopamine neurons in the VTA
–also occurs with the stimuli associated with the drug abuse