Test 3 - Ch. 14 Flashcards

1
Q

Where are cell bodies of UMN located?

A

cerebral cortex and BS
cerebral cortex - director
BS - laborer

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2
Q

What structure sends the message of plan to move to cortex and BS?

A

BG

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3
Q

What are the two places the UMN communicate to?

A

Axons to BS and SC

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4
Q

What are the three tracts of the medial motor tract?

A

reticulospinal
vestibulospinal
*lateral vestibulospinal
corticospinal

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5
Q

reticulospinal tract

A

Pick stuff up - more in arms
involuntary gross movements
origin of medial BS UMN pathway

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6
Q

Vestibulospinal tract- medial

A

upright against gravity - more in lower body
upper trunk, head and neck
lower degree of voluntary control

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7
Q

vestibulospinal tract- lateral

A

UNILATERAL PROJECTION
axial and lower extremity extensors
rest of Mm up against gravity
lower degree of control

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8
Q

Medial corticospinal tract

A

Voluntary control of neck, shoulder and trunk Mm
project to medial motor pools bilaterally
makes movement functional

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9
Q

What are the lateral tracts?

A

rubrospinal

lateral corticospinal

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10
Q

rubrospinal tract

A

distal extensors
red nucleus-spine
project distally
minor control

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11
Q

lateral corticospinal

A
directly activates distal movements
*** most important 
Fractionated movement 
direct control of LMN
guiding control to BS
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12
Q

Describe non-specific motor tracts

A

descend from BS - lower threshold for all other motor neurons so upper can start lower, increase AP
turned on by activity of limbic

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13
Q

Job of corticobrainstem

A

UMN of cranial Nn

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14
Q

Job of cortical motor

A

med and lat corticospinal homebase

pre-central gyrus of frontal lobe

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15
Q

Job of pre-motor area

A

anticipatory postural control

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16
Q

Job of supplementary motor area

A

plan bimanual and activities that have sequence

17
Q

3 signs of UMN lesions

A
  1. completely cut from LMN - SCI
  2. intact but tonically overactive - PD and TBI
  3. Cortical UMN damaged, BS okay - stroke
18
Q

paralysis

A

no movement below injury, UMN still exists but no connection

19
Q

paresis

A

some UMN cut = weakness

cortical UMN damaged but still connected to BS

20
Q

What happens if a lateral corticospinal tract is damaged?

A

fractionated movement is lost

21
Q

What happens to spinal reflexes below a cut to an UMN?

A

get stronger

22
Q

babinski reflex

A

toes extend instead of flex

UMN cut

23
Q

What is m stretch hyperflexia?

A

exaggerated reflex response to stretch

occurs when descending connection is missing or reduced

24
Q

“velocity-dependent hypertonia”

A

increased reaction to quick stretch

norm stim = abnormally large response

25
Q

Clonus

A

Rhythmic, repeating (usually involuntary) contraction of single muscle group
“velocity-dependent” that repeats itself

26
Q

Clasp-knife

A

initial resistance then relax

27
Q

What is myoplasticity?

A

Adaptive change w/in muscle itself
atrophy of disuse
stiffness
contracture

28
Q

Temporary hypotonia

A

LMN go to sleep w/out UMN input
short time
“spinal shock”
wakes up = hyperactivity because no comm

29
Q

velocity dependent hypertonia

1

A
"velocity-dependent"
stretch reflex overactive
muscle stretch hyperflexia
"spasticity" = muscle stretch hyperflexia
PD and BG damage
30
Q

velocity independent hypertonia

2

A
UMN present but abnormally active
PD and TBI
too many signals always
"rigidity"
decorticate
decerebrate
31
Q

activity dependent hypertonia

3

A

BS active but cortex impaired
only occurs when trying to move
CVA