STREPTOCOCCUS Flashcards
STREOPTOCOCCUS CAUSES WHAT THREE MAIN THINGS?
- SEPTICEMIA
- STRANGLES
- MASTITIS
STREPTOCOCCUS CAUSES THREE TYPES OF HEMOLYSIS. WHAT ARE THEY?
BETA HEMOLYTIC- COMPLETE HEMOLYSIS
ALPHA HEMOLYTIC- PARTIAL HEMOLYSIS
GAMMA HEMOLYTIC- NO HEMOLYSIS
STREPTOCOCCUS GENERAL FACTS
GRAM POSITIVE COCCI CHAINS CATALASE NEGATIVE FACULTATIVE ANAEROBE COMMENSAL IN MUCOUS MEMBRANES ZOONOTIC PRIMARY OPPORTUNISTIC PATHOGEN LOW ENVT RESISTANCE
STREPTOCOCCUS VIRULENCE FACTORS THAT FUNCTION IN INVASION
STREPTOKINASE
HYALURONIDASE
STREPTOCOCCUS VIRULENCE FACTOR THAT FUNCTIONS IN ADHESION
PROTEIN F
STREPTOCOCCUS VIRULENCE FACTORS THAT FUNCTION IN EVASION
M PROTEIN
HYALURONAN
HEMOLYSIN/STREPTOLYSIN
STREPTOCOCCUS TOXINS
STREPTOCOCCUS EXOTOXINS
LIPOTEICHOIC ACID
STREPTOCOCCUS EQUI SUBSP EQUI CAUSES
STRANGLES AKA EQUINE DISTEMPER
WHAT IS STRANGLES?
UPPER RESPIRATORY INFECTION WITH ABSCESSATION OF SUBMANDIBULAR AND RETROPHARYNGEAL LYMPH NODES
SWELLING OF THE LYMPH NODES LEADS TO AIRWAY OBSTRUCTION
WHAT AGES OF HORSE DOES STRANGLES INFECT ?
ALL AGES ARE SUSCEPTIBLE BUT YEARLINGS AND WEANED FOALS ESPECIALLY, <4MOS
TRANSMISSION OF STRANGLES
DIRECT- EXUDATES FROM UPPER RESP, DISCHARGING ABSCESSES FROM CARRIER OR INFECTED ANIMAL
INDIRECT- FROM CONTAMINATED BUCKETS/WATER TROUGHS, FEED, STABLES, GRASS, FLIES
STRANGLES COMPLICATION
- GUTTURAL POUCH EMPYEMA
- BASTARD STRANGLES
- PURPURA HEMORRHAGICA
DIAGNOSIS OF STRANGLES
- CLINICAL SYMPTOMS
- CULTURE FROM ABCESS, NASAL SWAB, LAVAGE FLUID
- ELISA
- PCR FOR M PROTEIN
POTENTIAL CARRIERS OF STRANGLES
NEWLY ACQUIRED HORSES
NON AFFECTED IN CONTACT HORSES
AFFECTED IN CONTACT HORSES
WHEN CAN A STRANGLES SUSPECTED HORSE BE RELEASED FROM QUARANTINE?
AFTER THREE NEGATIVE CULTURES
STRANGLES IS A REPORTABLE DISEASE T/F
TRUE
STREPTOCOCCUS SUIS IN PIGS (4 CONDITIONS)
- MENINGITIS
- ARTHRITIS
- SEPTICEMIA
- BRONCHOPNEUMONIA
STEPTOCOCCUS SUIS IS ZOONOTIC
TRUE MOTHER TRUCKER
TREATMENT OF STREP SUIS
PENICILLIN/AMPICILLIN
DIAGNOSTICS FOR STREP SUIS SHOULD INCLUDE
SEROTYPING
PREDISPOSING FACTORS FOR STREP SUIS
CHANGE IN VENTILATION CHANGE IN TEMP OVERCROWDING STRESS DECREASED IMMUNE SYSTEM
PROPHYLACTIC TREATMENT OF STREP SUIS
TREAT SOWS 1 WEEK PRIOR TO FARROWING
TREAT PIGLETS WITHIN 2 WEEKS OF LIFE
STREPTOCOCCUS AGALACTIAE
CHRONIC CONTAGIOUS MASTITIS
COLONIZES MILK DUCTS
STREP DYSGALACTIAE
ACUTE ENVIRONMENTAL MASTITIS
COLONIZES BUCCAL CAVITY AND GENITALIA
STREP UBERIS
ENVIRONMENTAL SUBLCLINICAL MASTITIS
COLONIZES SKIN, TONSILS, VAGINAL MUCOSA
STREP EQUI SUBSP ZOOEPIDEMICUS
ZOONOTIC
PYOGENIC- UPPER RESP INF, PNEUMONIA, MASTITIS, FOAL SEPTICEMIA
COLONIZE MUCOUS MEMBRANES
STREPTOCOCCUS SUIS IS COMMENSAL IN WHAT
TONSILS
STREPTOCOCCUS CANIS CAUSES ______ IN _______. IT IS COMMENSAL IN __________
TOXIC SHOCK SYNDROME AND NECROTIZING FASCIITIS
PUPPIES AND KITTENS
SKIN AND MUCOUS MEMBRANES
STREPTOCOCCUS PYOGENES
MILD SKIN DISEASE (IMPETIGO) TO LIFE THREATENING SYSTEMIC INFECTION
FLESH EATING BACTERIUM
ENTEROCOCCUS KEY CHARACTERISTICS
GRAM + DIPLOCOCCI CATALASE - COMMENSAL OF THE GI OPPORTUNISTIC PATHOGEN NATURAL AND ACQUIRED RESISTANCE
ENTEROCOCCUS FAECIALIS/ FAECIUM
- WOUND INFECTIONS
- MASTITIS
- UTI AND EAR INFECTION IN DOGS
- NOSOCOMIAL INFECTION