Shock Flashcards

1
Q

3 components of perfusion triangle

A

pump (heart)
container (blood vessels)
content (blood)

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2
Q

6 types of shock

A

hypovolaemic
cardiogenic
obstructive

distributive:
septic
anaphylactic
neurogenic

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3
Q

4 stages of shock

A
  1. Stage 1 - body is able to cope, no serious effects
    1. Stage 2 - compensatory stage
    2. Stage 3 - decompensated
      Stage 4 - Irreversible (exsanguination)
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4
Q

causes of hypovolaemic shock

A
  • Burns
    • Sweating
    • Diarrhoea
    • Vomiting
    • Haemorrhage
    • post-partum haemorrhaging
  • Haemoptysis (coughing up blood)
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5
Q

clinical presentation of hypovolaemic shock

A
  • Increased systemic vascular resistance
    • Lowered cardiac output
    • Tachycardia
  • Cyanosis
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6
Q

treatment for hypovolaemic shock

A
  • Check for blood on the floor and four more (abdo, pelvic, L femor, R femor)
    • Control the Haemorrhage
    • IV fluids (resus volume)
    • Hypothermia prevention
  • Blood transfusion
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7
Q

points to consider with blood loss

A
  • Elderly struggle
    • Children compensate longer, but deteriorate rapidly
    • Fit people handle it well
  • Meds affect response to blood loss, beta blockers may blunt the normal tachycardia
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8
Q

what is hypovolaemic shock

A

low volume of blood/fluid in circulation

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9
Q

what is obstructive shock

A

mechanical interference, problem with venous return, a physical obstruction to the flow of blood

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10
Q

causes of obstructive shock

A

Tension pneumothorax (trauma to parietal pleura, air from atmosphere enters pleural cavity, interpleural pressure increases, prevents inspiration + pushes mediastinum/trachea to contralateral side, mediastinum shift compresses the heart)

Aortic dissection (a tear within the aorta causing the inner + middle layers of the aorta to split)

Pericardial tamponade (fluid build up on pericardium, constricts filling of chambers)

Pulmonary embolism (restricted blood flow, stroke volume decreases, cardiac output decreases, hypotension, hypoxemic hypoxia (low level of 02 in blood), ischaemic, organ failure)

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11
Q

treatment of obstructive shock

A

Treat the cause

- Tension pneumothorax - needle decompression (equalise pressures of pleural cavity)
- Pericardial Tamponade - pericardiocentesis (remove build up of fluid in the pericardium)
- Pulmonary Embolism - heparin (anti-coagulants), thrombolytic drugs (streptokase), embolectomy
- Aortic Dissection - Surgery

Treat the symptoms - oxygen therapy, fluids, vasopressors (adrenaline)

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12
Q

clinical presentation of obstructive shock

A
  • Hypotension (constriction of myocardium)

- Muffled/distant heart sounds

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13
Q

what is distributive shock

A

a group of 3 shocks that denotes vessel malfunction, resulting in a decrease in systemic vascular resistance

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14
Q

what is anaphylactic shock

A

severe allergic reaction, cytokine storm

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15
Q

what is septic shock

A

result of severe bacterial infection (anaphylaxis), releases toxins that causes vessels to become leaky + semi permeable

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16
Q

what is neurogenic shock

A

a loss of sympathetic vasomotor tone, due to inhibitions with neurotransmitters

17
Q

describe the generalised process (result) of all types of shock

(9 stages)

A
systemic vasodilation
hypotension
low BP
inadequate tissue perfusion
generalised/localised hypoxia
ischaemia
necrosis
organ failure
death
18
Q

causes of anaphylactic shock

A

Extreme sensitivity to a substance, binds to mast cells, degranulation of mast cells, mass release of chemical mediators (primarily histamines)

19
Q

causes of septic shock

A

result of severe bacterial infection (anaphylaxis), releases toxins that causes increased vascular permeability

20
Q

treatment of anaphylactic shock

A
  • 1:1000 adrenaline, causes bronchodilation and blood vessel constriction
  • Anti-histamines
21
Q

clinical presentation of septic shock

A

Focus on patient history taking, active antibiotic course? Infection management not working?, is the patient vulnerable

- Mottled skin
- Fever (remember cold sepsis)
- Portsmouth sign (when SBP falls below HR) - heart is in overdrive but cardiac output is insufficient - Confusion (especially in elderly, lack of blood supply to the brain)
22
Q

treatment of septic shock

A
  • Blood Testing
    • Broad spectrum antibiotic administered within golden hour (benzylpenicillin)
    • Blood gases testing/cultures
  • Treat the symptoms (fluids, oxygen therapy etc)
23
Q

causes of neurogenic shock

A
  • Acute spinal cord injury (common)

- Regional analgesia

24
Q

clinical presentation of neurogenic shock

A

Autonomic blockade, causes unopposed vagal tone, bradycardia

Essentially nerves are unable to communicate with the body, specifically the cardiovascular system, inhibits vasoconstriction + cardiac contractility, causes vasodilation, low BP, low SVR, inadequate tissue perfusion, ischaemia, necrosis, organ failure

25
Q

treatment of neurogenic shock

A

Vasopressors (adrenaline)
Corticosteroid therapy
Atropine (bradycardia indication)
Fluids

26
Q

what effect does a mass histamine release have on the body

3 main points

A
  • increased vascular permeability, leads to oedema, specifically laryngeal oedema, airway obstruction
    • vasodilation (systemic hypotension), low BP, inadequate tissue perfusion, ischaemia, necrosis, organ failure, death
      constriction of smooth muscle (including bronchi) = respiratory distress