Psychology 5b - Brain and Behaviour Flashcards

1
Q

List the stages of memory

A
  • Input from our senses into the memory system (registration)
  • Processing and combining of received information (encoding)
  • Holding that input in the memory system (storage)
  • Recovering stored information from the memory system (retrieval)
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2
Q

Describe different durations of memory

A
  • Sensory
  • Working or short term memory (7 plus minus 2)
  • Long-term memory
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3
Q

Describe a model of memory

A
  • Information in to sensory
  • Sensory to long term memory (eg. trauma - treatment involves shifting this pattern)
  • Sensory to working memory (attention)
  • Rehearsed information will stay in the working memory, otherwise it is lose
  • Storage of some information into long term memory
  • Retrieval from long term to working memory
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4
Q

List types of long-term memory

A
  • Long term memory may be declarative or non-declarative
  • Declarative (episodic or semantic)
  • Non-declarative (procedural, priming, conditioning, non-associative loss)
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5
Q

What is priming?

A
  • Priming is the implicit memory effect in which exposure to a stimulus influences response to a later stimulus.
  • Used in associative learning
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6
Q

Where is declarative memory performed?

A

Medial temporal lobe and diencephalon

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7
Q

Which part of the brain is used in episodic memory?

A

Medial temporal lobes, hippocampus, entorhinal cortex, mammilary bodies and parahippocampal cortex

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8
Q

Describe the case of HM

A
  • Hippocampus surgically removed to treat epilepsy
  • Resulted in anterograde amnesia (with some retrograde)
  • Episodic memories were lost, but he had procedural memory
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9
Q

What is the hippocampus important for?

A
  • Sematic - knowledge
  • Procedural - how to do things
  • Working - short term
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10
Q

What is often intact in memory disorders?

A

Implicit memory (learning)

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11
Q

Describe the case of Clive Wearing

A
  • Muscian, composer, scholar
  • Herpes simplex encephalitis
  • Temporal lobe damage
  • Severe amnesia (could still play the piano and recognise his wife)
  • In a permanent state of having just woken up
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12
Q

Compare effect of left sided to a right sided lesion

A
  • Lesions left verbal informaton processing

- Lesion right non-verbal information processing, including face processing (prosopagnosia)

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13
Q

Describe serial position effect

A
  • Primacy effect (memory of the beginning things of a list - lost in Alzheimers)
  • Recency effect (memory of the end things in a list)
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14
Q

What is the probability of recalling a word related to?

A
  • Order in the list
  • Personal salience of words
  • Number of words
  • Chunking or other encoding strategy (frontally mediated deficits result in difficulty with this)
  • Delay time
  • Distraction
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15
Q

List clinical implications of recall

A
  • Give important information at the beginning and end of the consultation
  • Emphasise and repeat important information
  • Make salient to the person
  • Chunk information into meaningful categories
  • Avoid overloading with information
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16
Q

What is language?

A
  • System of symbols and sounds governed by rules
  • No humans let discovered without language
  • May be due to innate (critical periods of language acquisition) or exposure
  • Both organic and environmental factors are important
17
Q

Describe structure of language

A
  • Phoneme: smallest unit of speech sound in a language that can signal a difference in meaning (44 in english)
  • Morphenes: smallest units of meaning in a language (typically consist of syllables combined into words)
  • Words form phrases, which form sentences which forms conversational discorse
18
Q

What is syntax?

A
  • Rules and principles which govern the way in which morphemes and words can be combined to communicate meaning in a particular language
  • Universal grammar - under normal conditions humans will develop language with particular properties (eg. distinguising nouns from verbs)
  • Following exposure to non-grammatic language there is formation of a structured language in children
19
Q

Describe language development

A
  • 1-3 months (crying and cooing, can distinguish speech from non-speech sounds)
  • 4-6 months (babbling sounds begin)
  • 7-11 months (babbling sounds narrow to form phonemes, tongue is moved with vocalisations and imitations)
  • 12 months (first recognisable words spoken as one word utterances)
  • 12-18 months (use of single words to express whole phrases or requests)
  • 18-24 months (50-100 words spoken, li usually consists of two words)
  • 2-4 years (expansion of several hundred words every 6 months, basic language syntax and longer sentences)
  • 4-5 years (basic grammatical rules are known)
20
Q

What happens if a child is not exposed to language?

A
  • Genie - deprived of social interaction from birth until discovered aged 13
  • Completely without language, and after 7 years of rehabilitation she still lacked linguistic competence
21
Q

When is the critical period in language acquisition?

A
  • Between ages of 5 and puberty language acquisition becomes harder (based on feral children, children with brain injuries, second language acquisition)
  • Hardest at age 12
  • Exposure to other people is therefore required
22
Q

Describe brain structures underlying language

A
  • Hemispheric specialisation
  • 95% right handed people left hemisphere dominance, 18.8% left handed right hemisphere dominance
  • 19.8% left handed have bilateral language distribution
23
Q

How can memory be enhanced?

A
  • Chunking
  • Repetition
  • Acronyms
24
Q

What is expressive aphasia?

A
  • Requires lots of effort to express yourself
  • Non-fluent speech
  • Impaired repetition
  • Poor ability to produce syntactically correct sentences
  • Intact comprehension
  • Affecting Broca’s region
25
Q

What is receptive aphasia?

A
  • Affecting Wernicke’s area
  • Problems in comprehending speech
  • Fluent meaningless speech
  • Paraphasias (errors in producing specific words)
  • Semantic paraphasias (substituting words similar in meaning) and phonemic paraphasias (substituting words with similar sounds)
  • Neologisms (non words such as galump)
  • Poor repetition and impairment in writing
26
Q

Describe the language circuit when speaking a heard word (repetition)

A
  • Informaiton about sound analysed by primary auditory cortex and transmitted to Wernicke’s area
  • Wernicke’s area analyses the sound information to determine the word that was said
  • Transmitted through arcuate fasciculus to Brocas area
  • Broca’s area forms a motor plan to repeat the word and sends that information to the motor cortex
  • Motor cortex implements the plan, manipulating the larynx and related structures to say the word
27
Q

List forms of aphasia

A
  • Global aphasia (difficulty with all aspects)
  • Mixed transcortical aphasia (impaired fluency and comprehension with intact repetition)
  • Broca’s aphasia (comprehension but no fluency or repeats)
  • Transcortical motor aphasia (intact repetition impaired fluency)
  • Wernickes aphasia (fluency but no comprehension or repeats)
  • Transcortical sensory aphasia (intact repetition, impaired comprehension)
  • Conduction aphasia (lesion in the connection - fluent and comprehends with no repeats)
  • Anomic aphasia (deficit in naming specifically, with fluency, repetition and comprehension)
28
Q

List conditions associated with aphasia

A
  • Stroke
  • Traumatic brain injury
  • Cerebral tumour
  • Progressive neurodegenerative conditions
29
Q

What is dysexecutive syndrome?

A
  • Disruption of executive function, closely related to frontal lobe damage
  • Executive functioning skills are mental processes that enable us to plan, focus attention, remember instructions and juggle multiple tasks successfully
  • Encompasses cognitive, emotional and behavioural symptoms
  • Dysexecutive syndrome can result from head trauma, tumours, degenerative diseases, cerebrovascular disease and several psychiatric conditions
  • Eg. Phineas Gage
30
Q

List behavioral and emotional aspects of dysexecutive syndrome

A
  • Hypoactivity
  • Lack of drive
  • Apathy
  • Poor initiation of tasks
  • Emotional bluntness
  • Theory of mind difficulties and reduced empathy

Could instead have:

  • Hyperactivity
  • Impulsive
  • Disinhibitive
  • Perseverative
  • Emotional dysregulation
  • Socially inappropriate
  • Rude, crass, prone to swearing
31
Q

Describe cognitive aspects of dysexecutive syndrome

A
  • Attentional and working memory difficulties
  • Poor planning and organisation
  • Difficulty coping with novel situations
  • Difficulty multitasking
  • Difficulty moving from task to task
  • Difficulty with complex/ abstract thinking
32
Q

Which areas of the brain are different aspects of dysexecutive syndrome associated with?

A

Different areas of the frontal lobe

  • Orbito-frontal (impulsivity and disinihibition)
  • Medial (loss of spontaneity, initiation - akinetic mutism)
  • Lateral (inability to formulate and carry out plans)
  • Connection to cortical and subcortical (basal ganglia, cerebellum, thalamus) structures can result in similar behaviour changes (eg. Parkinsons)
33
Q

Describe conductive aphasia

A
  • Lesions of the arcuate fasciculus
  • Disrupts the transfer from Wernicke’s area to Broca’s area, so the patient has difficulty repeating
  • However, retains comprehension and fluency as the Wernickes and Broca areas are intact