Poultry Pathology Flashcards

1
Q

Deficiency in vit D, Ca, Ph

A
  • Affects all GPs -> evenly thickened GP -> accumulation of non-mineralised cartilage
  • Tibial dyschondroplasia = proximal tibia, uneven GP, plug of non-mineralised cartilage
  • HypoCa in laying hens -> reduction in lay / whole bird paralysis
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2
Q

Water deprivation

A
  • Gout
  • Due to back up of urates (kidney issues/water deprivation) -> go into viscera first then joints
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3
Q

Vitamin B2/riboflavin deficiency

A
  • Clubbed down + curled toe paralysis
  • CS - poor growth, reluctance to stand or walk, sitting on hocks, toes curl inwards
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4
Q

Egg production

A
  • Lights on 6 am -> egg laid
  • Chickens eat, then drink
  • After few hours, ovulates again
  • Shell forms last in evening + overnight - require stillness + quiet during this time
  • 9 h after lighters out -> lays another egg
    (Only left ovary is developed)
    https://www.thepoultrysite.com/publications/egg-quality-handbook/2/formation-of-the-egg
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5
Q

Trauma to growth plate infection

A
  • Reovirus
  • Mycoplasma synoviae
  • Osteomyelitis
    Cause swelling around the hock joint
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6
Q

Rickets

A
  • Metabolic disorder in young growing birds
  • Thickening and poor mineralisation of the
    growth plates and the bone
  • Dietary Ca, P and vitamin D3 deficiency; disturbed Ca/P ratio, or impaired absorption of one of these elements
  • High dietary phosphorous levels have a similar
    effect as low Ca levels
  • Hypocalcaemic rickets = accumulation of proliferating chondrocytes
  • Hypophosphataemic rickets = accumulation of
    hypertrophic chondrocytes
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7
Q

Tibial dyschondroplasia

A
  • Macroscopically, hypocalcaemic rickets are
    hardly distinguished from tibial dyschondroplasia because the latter condition may be a consequence of the former in its etiological aspect
  • TD can follow rickets as a result of the lowered hypertrophy of chondrocytes when birds try to adapt to the low calcium diet
  • Histopathology: Abnormal masses
    of prehypertrophic cartilage occupy
    the entire metaphysis and disrupting
    the blood supply. No distinct border is
    present between the proliferating and
    hypertrophic cartilage.
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8
Q

Biotin deficiency

A
  • Leads to dermatitis
  • Skin is not a healthy -> prone to ulcerations + pododermatitis
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9
Q

Common viral infectious causes of splenomegaly

A
  • When spleen > ½ length of proventriculus = splenomegaly
  • Viral infections causing tumours on spleen – Marek’s disease virus + Avian leucosis virus
  • Diffuse = spleen grossly attached
  • Nodular = diff parts attached = overall enlargement
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10
Q

Bacterial organisms that cause splenomegaly

A
  • E. coli
  • Salmonella
  • Pseudomonas
  • Staphylococcus
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11
Q

Feathering abnormalities caused by mating damage

A

Mating damage
- Neck
- Back of female bird

Mutilation/over-preening
- Self-mutilation - other feathered part of the body – depends where parasites are
- Pull out their feathers when pulling out eggs, parasites, over-preening, pull out feathers
- Mutilation – bullying, depending on type of environment

Ensure have 1 : 10 male to female ratio

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12
Q

Types of vacc / vacc protocol

A

Live – short duration, when want to boost immunity, admin several times
- IBC, Mycoplasma, Newcastle disease
- Given SC

Inactivated – longer duration + higher immune response, only need to admin once
- Primed w/ live vacc – stimulates memory cells e.g. for IBV
- Attenuated vacc – primed w/ live vacc
- IBD

  • If all ages affected - vacc early
  • Site/region-specific
  • Stabilising agent in water prior to vacc - to neuralise chlorine effects (used to kill microorgaisms)
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13
Q

Pathogens free-range birds are commonly exposed to (Resp)

A
  • Bacteria = mycoplasmosis
  • Virus = IBV
  • Parasite = Syngamus trachea
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14
Q

Difference between non-starters, starve-outs + hypoglycaemia

A
  • Non-starters + starve outs used interchangeably
  • Caused by poor management of feeders, drinkers
    o Collapse of
    o Correct amount of feed given
  • Biodegradable paper on floor for easy access to feed
  • Chicks could be hypoglycaemic when they don’t feed
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15
Q

Challenges during life of broiler - stress/impact on production

A

Stress/impact on production during:
- Brooding
o Temperature
o Feed
o Water available – nipple drinkers are at the correct level, not too high
- Transfer crumb to pellet
o Crumbs have smaller particle size than pellets
o Don’t give too large pellets, will start to starve-out
o Crumbs 1 – 7 d
o When transferring – mix crumbs + pellets e.g. have on diff sides of building
o Water available – helps dissolve feed in crop
- Vacc
o Shouldn’t starve birds for > 1 h, especially broilers, esp during in summer
o Be gentle
- Feathering up – protects them, keeps warm
o Need to give right quantity of feed + to maintain body temp
o Vitamins – give to build-up feathers
- Coccidiosis challenge
o Layers – vacc
o Broilers – incorporate coccidiostats in feed
o Make sure litter is dry – no host for coccidia to sporulate
o Don’t reuse litter – should start on new ground
- Various feed changes throughout life
o Correct particle size
o Correct feeders
o Change gradually
- Thinning
o Dim lights, dec lighting, close curtains
o Raise feeders + drinkers
o Withdraw feed prior

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