PA 10. motor brain Flashcards

1
Q

key motor function of primary motor cortex

A

execution

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2
Q

key motor function of `premotor cortex

A

preparation of actions

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3
Q

key motor function of prefrontal cortex

A

higher level of planning

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4
Q

key motor function of parietal cortex

A

sensory-motor links

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5
Q

Hemiplegia

A

paralysis of one side

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6
Q

Hemiparesis

A

weakness of one side

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7
Q

M1- connections

A

Input from supplementary
motor area, premotor
area and primary
somatosensory area

Output to spinal cord –
control of muscles

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8
Q

Lateral Premotor Cortex responsible for

A

externally generated actions (taping to music)

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9
Q

Supplementary Motor Area – (medial PC) responsible for

A

internally generated actions e.g. well learnt
sequences

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10
Q

SMA in sequence learning

A

responsible only for most complex sequences

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11
Q

Prefrontal - Higher level responsible for

A
  • Attention to action – when difficult or
    learning
  • Longer term goals and intentions
  • Not specific to action, e.g. generating
    random numbers
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12
Q

Prefrontal Lesions
Can produce:

A
  • Perseveration – repeat same action when
    no longer relevant
  • Utilisation behaviour – act on irrelevant
    (or inappropriate) object in environment
  • Disinhibition e.g. antisaccade task
  • Frontal apraxia – not able to follow steps
    in routine tasks (e.g. making tea)
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13
Q

Antisaccades

A
  • Required to look in
    opposite direction to
    the target
  • Must inhibit (prepotent) tendency to
    look at target
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14
Q

Normal and Shallice model

A

Contention scheduling – selects appropriate
schema
Supervisory attentional System (SAS) –
required for novel/less automatic actions

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15
Q

Explaining action errors

PRESERVATION

Utilisation beahviour

A
  • Perseveration – unable to change
    schemas when no longer appropriate
  • Utilisation behaviour – schemas activated
    by environment without SAS suppressing
    them
    (cf. Cognitive failures questionnaire)
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16
Q

Damage to the parietal
cortex can lead to apraxia:

A

Inability to perform skilled
purposeful movement

17
Q

Ideomotor Apraxia

A

idea and execution disconnected – retain
knowledge of action
* Can recognise action performed by another
* Fail in pantomiming action (e.g. body part as
tool)
* Can perform sequence but not components

18
Q

Cerebellar patients
Can lead to

A
  • Action tremor
  • Dysmetria – over and undershooting of
    movements
  • Deficits in:
    – coordinating across joints
    – Motor learning
    – Timing
19
Q

Parkinson’s disease symptom

A
  • 1 in 1000, 1 in 100 over 65
    1. Bradykinesia – slow movement
    2. Tremor (resting)
    3. Rigidity
    Shuffling
    Death of dopaminergic cells in substantia nigra
    pars compacta
  • Internal/external – more problems with
    internally generated movements
  • Complex movements – bimanual,
    sequences
  • Cognitive effects – attention shifting,
    everyday cognitive failures