Ophthalmology Flashcards

1
Q

Peripheral vision loss (sudden shadow), blurred/distorted vision, flashes/floaters

A

Retinal detachment

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2
Q

Sudden painless loss of vision. FUndoscopy shows flame + blot haemorrhages, optic disc oedema, macular oedema

A

Retinal vein occlusion

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3
Q

Sudden, painless loss vision. Cherry red spot + pale retina (hazy white fog). Relative afferent pupillary defect

A

Central retinal artery occlusion

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4
Q

Generalized reduction in visual acuity + starbursts around lights + halos
DM can cause

A

Cataracts

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5
Q

Central loss + crooked/wavy appearance of straight lines.

A

Macular degeneration

-Dry = drusen (most common)
-Wet - choroidal neovasculrisation. (fatser visual loss, v rare)

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6
Q

Glaucoma, acte vs chronic for associations

A

acute = as with hypermetropia - long sited

Open - myopia ass -short sited

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7
Q

Peripheral loss of vision + halos around light

Severe pain
Cupping optic disc
Semi dilated pupil

Dx and mx

A

Glaucoma

Dx - ophthalmologist - slit lamp, tonometry, gonioscopy.
Aim - reduce aqueous secretion + induce pupillary constriction
SUrgery is definitive but after IOP reduced

1.Prostaglandin analogue
2.BB, Cabronic anhydrase inhibitor, sympathomimetic eyedrop

ACute:
Combo eye drops - direct parasympathomimetic (pilocarpine etc), BB, alpha-2 agonist
+
iV Acetylzolamide

Definitive is laser peripheral iridtomy

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8
Q

Small fixed oval pupil, ciliary flush, blurred vision + photophobia
Acutely painful, decreased visual acuity

A

uveitis

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9
Q

Unilat decrease in visual acuity, poor discrimination colours (red desaturation), pain worse on eye movement, relative aff pupillary defect, central scotoma.

A

optic neuritis

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10
Q

Severe pain, exacerbated by movements, tender, may be underlying autoimmune
With phenylephrine, redness doesn’t blanch

A

Scleritis

Same day referral to ophthalmology (oral NSIDAs-> top steroids)

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11
Q

difference with episcleritis vs scleritis

A

In episcleritis - vessels blanch and move with cotton bud and doesn’t usually cause pain

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12
Q

Dendritic corneal ulcer

A

Herpes keratitis

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13
Q

Corneal ulcer- focal circular/oval shaped
(Pseudomonas aeruginosa in contact lens weares)

A

Bacterial keratitis

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14
Q

Hypertensive retinopathy stages

A

I - AV narrowing + tortuosity, increased light reflex + silver wiring
II- Arteriovenous nipping
III - Cotton wool exudates, flame + blot haemorrhages (macular star)
IV - Papilloedema

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15
Q

Tx for non proliferative and proliferative diabetic retinopathy

A

Non-p = observe and if severe then pan retinal laser photocoagulation

Proliferative = VEGF + pan retinal laser photocoagulation

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16
Q

Allergic conjunctivitis tx

A

1.Topical/systemic antihistamines
2.Topicla mast cell stabilisers

17
Q

Red eye, pain + visual loss after eye surgery

A

Endothalmitis

18
Q

Pianless, transient monocular blindness (black curtain coming down)

CVD FRS

A

Amaurosis fugal

19
Q

3rd nerve palsy vS horners syndrome

A

3rd nerve palsy = Ptosis, dilated non reactive pupil
DIvergent strabismus (squint)

Horners = Ptosis and constricted (meiosis) pupil
Enophthalmos +/- anhidrosis

20
Q

WHta is a relative aff pupillary defect

A

Affected and normal eye appears to dilate when light is shone on affected eye
Causes - retinal detachment, optic neuritis.

21
Q

Periorbital vs orbital cellulitis and mx

A

Periorbital = Hot, redness, swelling

Orbital = pain on eye movement, change in vision + proptosis

CT with contrast
Orbital - admit to hospital for iV Abx

22
Q

dx, mx

A

herpes zoster opthalm..

VSV in ophthalmic division trigemnial nerve. (Shingles)
Vesicular rash around eye, hutchingsons sign (rash on top or side nose - nasociliary involvement and strong RF for ocular involvement)

Oral antiretroviral tx 7-10days, topical steroids if secondary inflammation of eye
Ocular Involvement - urgent ophthalmology review

most recover

23
Q

Bilateral grittiness and discomfort, particularly around eyelid margins. Eyes may be sticky in morning,eyelid margins can be red. Styes/chalazions common.

dx, mx

A

blepharitis

1.Hot compress twice a day
2.Lid hygiene (remove debris with cotton bud)
Artificial tears

24
Q

PG analogues in glaucoma - moa, s.e

A

Prostaglandin analogues
lantroprost
increases uveoscleral outflow
OD
A/E include brown pigmentation of iris, increased eyelash length

25
Q

BB in glaucome - MOA

A

timolol - reduces aqueous production - avoid in asthmatic and pts with heart block

26
Q

Sympathomimetics in Glaucoma

A

brimonidine, a2 adrenoreceptor agonist - reduced aqueous production and increases outflow
Avoid if taking MAOI or TCA
A/E - hyperaemia

27
Q

Carbonic hydrase inhibitors in glaucoma

A

(Dorzolamide) - reduce aqueous production
Systemic absorption may cause sulphonamide like reactions

28
Q

Miotics in glaucoma - moa, a/e

A

pilocarpine (muscarinic receptor agonist) - increases uveoscleral outflow
a/e - constricted pupil, headache, blurred vision

29
Q

Anterior uveitis mx

A

Steroid and cycleplegic eye drops

30
Q

Argyll-RObertson pupil

A

Seen in neurosyphilis
Pupil accommodates but doesnt reaction
Small irregular pupils. No response to light but will accommodate
Causes - DM, syphilis

31
Q

Tonically dilated pupil, slowly reactive to light with more definitive accommodation response.
Caused by damage to parasympathetic innervation of eye due to viral or bacterial infection
Commonly in females, accompanied by absent knee or ankle jerks

A

adie pupil

32
Q

Relative aff pupillary defect seen during swinging light exam of pupil response
Pupils construct less and therefore appear to dilate when a light is swung from unaffected to affected eye
Most commonly caused by damage to optic nerve or severe retinal disease

A

Marcus gunn pupil

33
Q

Unilateral dilated pupil which is unresponsive to light. Result of compression of occulomotor nerve of same side, by intracranial mass (tumour, haematoma)

A

Hutchinson’s