NMS 2 Exam 3 Flashcards
Type 1 Spondylolisthesis
-Dysplastic
-Congenital defect in neural arch or sacrum
Type 2 Spondylolisthesis
-Isthmic
-Lytic: Stress fracture in pars
-Acute: Fracture in Pars
Type 3 Spondylolisthesis
-Degenerative
-Degenerative arthrosis of regional lumbar anatomy
Type 4 Spondylolisthesis
-Traumatic
-Fracture of neural arch other than pars
Type 5 Spondylolisthesis
-Pathologic
-Occurs as a result of bone pathology
Lumbar Myelopathy
-Compression of the spinal cord causing neurologic dysfunction (gait disturbance, pathologic reflexes, muscle weakness, and/or numbness)
Causes of Lumbar Myelopathy
Degenerative changes, central disc herniations, autoimmune disorders, hernias, cysts, hematomas and spinal tumors
Symptoms of Lumbar Myelopathy
-Leg/Lower back pain
-Tingling, numbness or weakness
-Increased reflexes in the extremities
-Difficulty walking
-Loss of urinary or bowel control
-Issues with balance and coordination
Lumbar Radiculopathy
-IVF compresses the nerve root as it exits the vertebrae
-Can cause compressive or inflammatory lesions
-Compressive: Space occupying lesion
-Inflammatory: Reaction leading to sensitivity
Examples of Lumbar Radiculopathy
Disc herniations, degenerative changes and expansile lesions
Risk factors of Lumbar radiculopathy
Degenerative, repetitive lifting/twisting, chronic overloading, prior trauma, poor posture, muscle imbalance, congenital defects, family history, lack of exercise, poor core strength
Lumbar Spinal Stenosis
Narrowing of the lumbar vertebral spinal canal with possible subsequent neural compression (neurogenic claudication)
Neurogenic claudication
Cramping/weakness in legs
Stenosis: Clinical Presentation
-Elderly undergoing extensive degenerative changes
-Discomfort is generally local as dull/achy pain
-Pain worse with extension and extercise
-Pain better with rest and flexion
-Most common at L5/S1
Central Canal Disc Lesion
-Compressed the spinal cord in the cervical and thoracic regions, compresses the cauda equina in the lumbar region
-Myelopathy and cauda equina syndrome
-Central, paracentral, lateral
IVF disc lesion
Compresses the nerve root as it exits the vertebrae
-Radiculopathy
-Medial/Lateral
Types of Disc Lesions
-Contained (intact annular fibers): Protrusion, Subannular extrusion
-Non-Contained (disruption of annular fibers): Transannular, Sequestered
Progression of Disc Lesions
-90% of patients have resolution of symptoms within 3 months of onset
-Nearly complete recovery, although flareups can occur
-75% of herniations resolve spontaneously within 6 months due to resporption of herniated material
At what point should we be looking at surgery for a disc lesion if conservative care isn’t working
Approximately 8-12 weeks
Lumbar Facet Syndrome
LBP d/t inflammation or degeneration of the facet joint or joint capsule often aggravated by hyperlordosis or extension movements
-Peak incidence in 40-50 years old
Lumbar Facet Syndrome: Presentation
-Dull, achy LBP w/ some sharp, acute episodes
-Insidious (degenerative)/Sudden (trauma)
-Pain worse with prolonged standings/lumbar ext
-Pain better lying supine, sitting or lumbar flexion
Causes of stenosis
-Primary causes: Anything that alters size/shape of the canal, usually during the developmental years
-Secondary causes: Degenerative changes or any condition that invades or compromises the space
Grade 1 Sprain/Strain
-Mild swelling/point tenderness over ligament
-No bruising
>10% fiber damage
-Mild stretch, no instability
(-) stretch tests, mild pain at extreme ROM
-Recovery: 2-14 days
Grade 2: Sprain/Strain
-Mild to moderate swelling
-Large spectrum injury that can include partial tearing
-11-90% fiber damage
-Mild to moderate instability may be demonstrated
-Recovery: 14 days to 2 months
Grade 3: Sprain/Strain
-Severe deep bruising/swelling
-Complete tearing of multiple ligaments, muscles, and joint capsules
-Marked instability
-Almost complete loss of ROM
-Recovery: 1-3 months
Lumbar Sprain/Strain: Presentation
-LBP, stiffness, or muscle spasms
-Prior history of back injury
-Limited ROM in low back d/t pain
-Pain better with rest, heat and non-weight bearing
-Pain is aggravated by activity and lifting
-Know specifics of techniques and MOI
Coccygodynia
-Ongoing pain syndrome around the coccyx
-Pain around the sacrococcygeal articulation
-Acute or chronic
-May have history of a sit down fall
-Pain with sitting or sit to stand
-Pain relieved by walking
-Pain with palpation/ROM
Segmental instability
Reversible, hypomobile segments that respond well to manipulation
Segmental Instability
Reversible, hypomobile segments respond well to manipulation
Vascular claudication
-Narrowing or blockage of an artery
-X-Rays may show arteiosclerosis
Use of bracing
Only use if severe, to help stabilize lumbar spine
(Not sure on time)
DDD/DJD
-Progressive loss of articular fibrocartilage and reactive changes to the joint margins and subchondral bone
-DJD is the most common form of joint disease and leading cause of disability with elderly
Primary DJD
-Idiopathic
-Abnormal biomechanics or posture
-Genetic disposition
-Metabolic causes
Secondary DJD
-Usually following joint trauma
-Equal gender distribution
-Childhood anatomic abnormalities
-Joint trauma or fracture
-Obesity
-Repetitive action/joint motion
Phases of Degeneration (Kirkaldy-Willis Degenerative cascade)
-Phase 1 (Dysfunctional Phase)
-Phase 2 (Unstable Phase)
-Phase 3 (Stabilization Phase)
Phase 1: Dysfunctional Phase
-Circumferential tears in outer annulus
-Nucleus pulposis loses water and proteoglycan content
Phase 2: Unstable Phase
-Loss of mechanical integrity, internal disc disruption, resorption and loss of disc height
-Leading to segmental instability
Phase 3: Stabilization Phase
Further disc resorption, disc space narrowing, endplate destruction, osteophyte formation
-Possible discogenic pain results
What is the most common spondy
L5
What is the most common disc
L4
Who is spondy more common in
Females, 40+ yoa
Ankylosing Spondylitis is more prevalent in
Males (4:1) in their 20s
Ankylosing Spondylitis
Chronic, inflammatory disorder of the axial skeleton resulting in formation of new bone at the t of ligaments and tendons