M - 6 - Poisoning and Toxic Ingestion - Adult & Peds

Question 1

Ingestion of a stimulant requires management under what protocol?

Answer 1

Behavioral emergencies (M-10)

Question 2

______ must be administered in a dedicated IV line, mixed with no other drugs.

A. Glucagon
B. Hydroxocobalamin
C. Ondansetron
D. Atropine

Answer 2

B. Hydroxocobalamin

Question 3

Which medications require orders from a physician for admininstration in the case of suspected poisoning?

Answer 3

• Atropine
• Calcium chloride
• Zofran/glucagon
• Hydroxocobalamin

Question 4

For symptomatic organophosphate poisonings, what must be given?

A, Atropine
B. Calcium chloride
C. Zofran/glucagon
D. Hydroxocobalamin

Answer 4

A, Atropine

Question 5

For hypotension associated with beta-blocker/calcium-channel blocker OD:

A, Atropine
B. Calcium chloride
C. Zofran/glucagon
D. Hydroxocobalamin

Answer 5

C. Zofran/glucagon

Question 6

For magnesium toxicity/calcium-channel blocker overdose:

A, Atropine
B. Calcium chloride
C. Zofran/glucagon
D. Hydroxocobalamin

Answer 6

B. Calcium chloride

Question 7

For suspected cyanide poisoning, what must be given?

A, Atropine
B. Calcium chloride
C. Zofran/glucagon
D. Hydroxocobalamin

Answer 7

D. Hydroxocobalamin

Question 8

In the case of suspected dystonic reactions from anti-psychotics, what may be given to adult, non-geriatric patients?

Answer 8

50 mg diphenhydramine IV/IM

Question 9

Known TCA ingestion with wide-complex rhythms are given ______.

Answer 9

Sodium bicarbonate

Question 10

TCA overdoses with wide-complex rhythms in patients between 50-74kg are given ______.

Answer 10

50 mEq sodium Bicarbonate IV

Question 11

TCA overdoses with wide-complex rhythms in patients over 75 kg are given ______.

Answer 11

75 mEq sodium Bicarbonate IV

Question 12

Atropine is administered for organophosphate overdoses. By what mechanism does it affect S/S?

A. By freeing glucose from glycogen.
B. Competes with ACh on muscarinic receptors to reduce secretions and increase heart rate.
C. Stabilizing the cardiac membrane by yet unknown mechanism.
D. Overcoming calcium channel blockade/increases force of contraction.

Answer 12

B. Competes with ACh on muscarinic receptors to reduce secretions and increase heart rate.

Question 13

By what MOA does calcium chloride treat calcium channel blocker overdose?

A. By freeing glucose from glycogen.
B. Competes with ACh on muscarinic receptors to reduce secretions and increase heart rate.
C. Stabilizing the cardiac membrane by yet unknown mechanism.
D. Overcoming calcium channel blockade/increases force of contraction.

Answer 13

D. Overcoming calcium channel blockade/increases force of contraction.

Question 14

By what mechanism does glucagon treat calcium channel overdose?

A. By freeing glucose from glycogen.
B. Competes with ACh on muscarinic receptors to reduce secretions and increase heart rate.
C. Stabilizing the cardiac membrane by yet unknown mechanism.
D. Overcoming calcium channel blockade/increases force of contraction.

Answer 14

C. Stabilizing the cardiac membrane by yet unknown mechanism.

Question 15

By what mechanism does glucagon treat beta-blocker overdose?

A. By freeing glucose from glycogen.
B. Competes with ACh on muscarinic receptors to reduce secretions and increase heart rate.
C. Stabilizing the cardiac membrane by yet unknown mechanism.
D. Overcoming calcium channel blockade/increases force of contraction.

Answer 15

C. Stabilizing the cardiac membrane by yet unknown mechanism.

Question 16

By what mechanism does glucagon treat insulin overdose?

A. By freeing glucose from glycogen via enzymatic activity (activation of PKA creates a step-wise series of reactions to cleave liver glycogen)
B. Competes with ACh on muscarinic receptors to reduce secretions and increase heart rate.
C. Stabilizing the cardiac membrane by yet unknown mechanism.
D. Overcoming calcium channel blockade/increases force of contraction.

Answer 16

A. By freeing glucose from glycogen via enzymatic activity (activation of PKA creates a step-wise series of reactions to cleave liver glycogen)