lecture 19: CN XI and XII Flashcards
which of the following nuclei is not shared by Cn IX and X
inferior salivatory nucleus
sensory trigeminal nucleis
nucleus ambiguous
nucleus solitaries
inferior salivatory nucleus
a patient presents with a uvular deviation to the left when saying ahh and an absent gag reflex unilaterally, which of these structures is damaged
left vagus n
right vagus n
left glosso n
right glosso n
right vagus n
what is CN XI
accessory n
is the accessory n motor, sensory or mixed
motor
what is the main function of the accessory n
voluntary motor innervation of sternocleidomastoid and trapezius
what does the sternocleidomastoid muscles do
contralateral rotation and ipsilateral flexion of the neck
true or false: the SCM does ipsilateral rotation and ipsilateral flexion of the neck
false, contralateral rotation
what does the trapezius muscle m do
scapular movement, upper limb support and posterior stability
how does the accessory n enter the skull
via foramen magnum
why does the accessory n need to enter the skull thru foramen magnum
because it starts in cervical region of the spine
how does the accessory n exit the skul
via jugular foramen
the accessory n exits skull via via jugular foramen with what other two nerves
glosso and vagus
where is the accessory nuclei located
in the lateral portion of the anterior grey horn (spinal root from c1-c5/6
true or false: the accessory nuclei is located in the lateral portion of anterior grey horn
true
spinal root for accessory nuclei is what cervical levels
c1-c5./c6
true or false, accessory nuclei get corticonuclear input
true
is the accessory nuclei somatic or branchial motor
branchial motor
is there a cranial component to the accessory nuclei
yes it is associated with nucleus ambigguus
(cranial root believed to join the vagus nerve en route to muscles of palate, larynx and pharynx
explain the UMN control of the XI n
1) UMN in primary motor cortex (neck area)
2) corona radiata
3) internal capsule (gene)
4) cerebral peduncle
5) basal pons
6) pyramids (decussation)
=ipsialteral for SCM
=contralateral for trap
7) synapse with LMN in accessory motor nucleis
explain what happens at the pyramids for cortciconuclear control of the CN XI
UMN stays ipsialtearl for the SCM
UMN goes contralateral for trapezius
where do UMN synapse with LMN for the CN XI
in accessory nuclei
is hypoglossal n purely motor, sensory or mixed
motor
what is the general function of the hypoglossal n
voluntary motor innervation of intrinsic tongue muscles (change the shape of the tongue)
voluntary motor innervation of all extrinsic tongue muscles (except palatoglossus CN X)
=movement of the tongue
what do the intrinsic muscles of the tongue do to the tongue
change the shape of the tongue
what do the extrinsic muscles of the tongue do
movement of the tongue (retraction. elevation)
what are some examples of external tongue muscles
genioglossus
hyoglossus
styloglossus
(palatoglossues)
what is the only extrinsic tongue muscle not innevated by the hypoglossal
palatoglossus (CN X)
which n innervates palatoglossus
CN X
how does the hypoglossal n exit the skull
via the hypoglossal canal (at the edge of the foramen magnum)
the hypoglossal nucleus is what type of nucleus
somatic motor
true or false; the hypoglossal nucleus carries motor innervation to all muscles of the tongue
false, all muscles except palatoglossus
where does the hypoglossal nerve emerge from in the brainstem
emerges from the medulla ventral to the olives
(between pyramids and olives)
true or false, the hypoglossal n emerges from the medulla dorsal to the olives with CN IX and X
false, ventral
what is the level of the brainstem where we would find hypoglossal nucelus
rostral medulla
what is the characteretisc symptom for a trapezius problem
shoulder droop
what is the characteretisc symptom for a SCM problem
weak SCM (Difficulty turning to opposite side)
will the symptoms for a trapezius issue always be contralateral to the lesion
no contralat for UMN
ipsilat for LMN
will the symptoms for a SCM issue always be contralateral to the lesion
false, always IPSI
an UMN of spinal accessory will cause what deficits
deficits to the contralateral trapezius
deficits to the ipsilat SCM (difficulty turning to opposite side)
person lesions the UMN of the spinal accessory at the right cerebral peduncle, what is the symptoms
deficits to the left trapezius (left shoulder drop)
deficits to the right SCM (difficulty turning to left side)
an LMN of spinal accessory will cause what deficits
deficits to the ipsilateral trapezius
deficits to the ipsilat SCM (difficulty turning to opposite side)
person lesions the LMN of the left spinal accessory nucleus, what are the symptoms
deficits to the left trapezius (left shoulder drop)
deficits to the Left SCM (difficulty turning to right side)
a person has an issue with their right trapezius and right SCM at the same time, is the lesion UMN or LMN
LMN
true or false: the hypoglossal and spinal accessory nucleus are both. branchial motoro
false
accessory=branchial
hypoglossal=somatic
explain pathway for UMN (corticinucleuar) for hypoglossal n
1) UMN in primary motor cortex (tongue region)
2) corona radiate
3) IC (genu)
4) cerebral peduncle
5) basal pons
6) synapse with LMN in the hypoglossal nucleus
-contraltaeral for the genioglossus only
-bilateral for all other tongue muscles
explain the synapse with LMN in the hypoglossal corticunuclear pathway (ie> explain the decussation)
-contraltaeral for the genioglossus only
-bilateral for all other tongue muscles
what is the function of the genioglossues muscle
tongue protrude
true or false and why: the majority of the tongue muscles will be unaffected by an UMN lesion
true because they receive bilateral innervation
explain the deficits seen in an UMN nerve palsy for hypoglossal n
UMN lesion results in tongue deviation to the contralateral side (paralyzing contralateral genioglossus)
an UMN lesion results in tongue deviation to the BLANK side (paralyzing Blank genioglossus)
contrlatearl for both
an UMN lesion results in tongue deviation to the contralateral side (paralyzing BLANK genioglossus)
CONTRLATERAL
If i lesion my right UMN in hypoglossal n pathway, what will be the deficits
tongue will deviate towards contralateral side/left side (paralyze my left genioglossues)
If i lesion my right LMN in hypoglossal n pathway, what will be the deficits
tongue will deviate towards ipsilateral side/right side (paralyze my right genioglossues)
rightside of tongue will looks fucked up (fasciualtions)
explain the deficits seen in a LMN nerve palsy for hypoglossal n
LMN lesion results in tongue deviation to the ipsilateral side (paralyzing ipsialteral genioglossus)
tongue will not be symmetrical (ie facsiculatinons, weird appearance on one side since all muscles affected)
a LMN lesion results in tongue deviation to the BLANK side (paralyzing Blank genioglossus)
ipsualtearl for both
a LMN lesion results in tongue deviation to the ipsialteal side (paralyzing BLANK genioglossus)
ipsialteral
add slides on medial medullary syndrome
what is the associated foramen of the accessory n
enter skull thru foramen magnum
exit through jug foramen
true or false, the spinal accessory innervates the SCM and the lat dorsi
false, the SCM and trap