L2: CHPS Flashcards
1
Q
Def of CHPS
A
- Hypertrophy of the pyloric muscle that causes gastric outlet obstruction.
2
Q
Epidemeology of CHPS
A
3
Q
Epidemeology of CHPS
- Incidence
A
One in 250 live births 1: 250
4
Q
Epidemeology of CHPS
- Sex
A
Males 4 : Females 1
5
Q
Epidemeology of CHPS
- Family History
A
Positive
6
Q
Etiology of CHPS
A
7
Q
Etiology of CHPS
- Etiological Factors
A
8
Q
Pathophysiology of CHPS
A
9
Q
Pathophysiology of CHPS
- Infantile pyloric stenosis is characterized by persistent, non-bilious projectile vomiting, Due to gastric outlet obstruction.
A
β¦
10
Q
Prolonged vomiting leads to loss of large quantities of gastric secretions rich in H+ & Cl.
A
β¦.
11
Q
- As a result of dehydration, kidneys attempt to conserve Nat to maintain volume by exchanging Na* for K* and H* secreted as KHCO, & H,CO, β¦..
A
(paradoxical aciduria).
12
Q
Paradoxical Aciduria
A
There is Β«metabolic alkalosis, but instead of having an alkalotic urine, it is acidix.
13
Q
CP of CHPS
A
14
Q
CP of CHPS
- History (symptoms)
A
- Typically a full-term baby, 3-5 weeks old
- Persistent, gastric, never-bilious, projectile vomiting soon after feeds.
- Failure to thrive, constipation, seizures.
15
Q
CP of CHPS
- Signs
A
- Pyloric βtumorβ in right upper quadrant.
- Visible peristalsis.
- Dehydration late, presentation).
16
Q
CP of CHPS
- Metabolic Abnormalities
A
- Hypochloremic hypokalemic metabolic alkalosis.
- Paradoxical aciduria due to renal conservation of Na+ leading to loss of H*.
- CO, increase due to the respiratory compensation.
17
Q
INVx for CHPS
A
- Rads
- Labs
18
Q
INVx for CHPS
- Rads
A
- US
- Contrast Study
19
Q
INVx for CHPS
- US
A
20
Q
INVx for CHPS
- Contrast Study
A
21
Q
INVx for CHPS
- Labs
A
22
Q
TTT of CHPS
A
- Pre-operative
- Surgical TTT
23
Q
TTT of CHPS
- Pre-operative
A
24
Q
TTT of CHPS
- Surgical TTT
A
25
Q
Done
A
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