Hydrocephalus and raised ICP Flashcards

1
Q

What are the two types of hydrocephalus?

A

communicating and non communicating

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2
Q

Describe non communicating hydrocephalus?

A

obstruction to the flow of CSF occurs within the ventricular sytem - before the CSF flows out into the ventricuar space

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3
Q

Describe communicating hydrocephalus?

A

obstruction to the flow of CSF is outside of the ventricular system, the dilation of the ventricles is constant throughout - eg in subarachnoid space or arachnoid granulations

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4
Q

At what age do the cranial sutures close?

A

2/3 years

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5
Q

What happens if there is hydrocephalus before 2/3 years?

A

cranial enlargement

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6
Q

What happens if there is hydrocephalus after 2/3 years?

A

get expansion of the ventricles and increased cranial pressure

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7
Q

What is hydrocephalus ex vacuo?

A

dilation of the ventricular system and a compensatory increase in CSF volume secondary to loss of brain parenchyma

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8
Q

What is the equation that links MAP, ICP and CPP?

A

MAP - ICP = CPP

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9
Q

What does a CPP <30 mean?

A

comatosed

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10
Q

What is a normal MAP?

A

90

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11
Q

What is a normal ICP?

A

10 (can be negative while lying down)

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12
Q

What is a normal CPP?

A

80

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13
Q

What is cushings triad?

A

decreased resp rate
increased CPP
MAP >100

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14
Q

What is the Monroe-Kellie Doctorine?

A

three components in the skull - brain tissue, CSF, intracranial circulating blood

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15
Q

What is pressure autoregulation of blood pressure?

A

arterioles dilate or constrict in response to changes in BP or ICP

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16
Q

What is metabolic autoregulation of blood pressure?

A

arterioles dilate in response to chemicals - lactic acid or CO2

17
Q

What can cause problems with CSF flow?

A

obstruction of flow - masses and chiari malformations
increased production - choroid plexus papilloma (produces CSF)
decreased absorption - communicating hydrocephalus - due to SAH, meningitis or malignany meningeal disease

18
Q

What are the early signs of CSF flow issues?

A
drowsy 
headache
pupillary dysfunction
nausea and vomiting
blurring
visual field defects
decreased level of conciousness
19
Q

What are the later signs of CSF flow issues?

A
coma
fixed dilated pupils
hemiplegia
cushings triad
increased urinary output
20
Q

What is the treatment for raised ICP?

A

maintain midline and neutral postition of the head
loosen tube ties, collars, jewellery - prevent gagging/coughing
head of the bed at 30-45 degrees
maintain normocarbia - hyperventilating the patient
maintain fluid and electrolytes

21
Q

What medical treatment can be given for raised ICP?

A

diuretics - mannitol, hypertonic saline, furosemide urea
antiepileptics - prevent energy expenditure
surgical decompression

22
Q

What causes communicating hydrocephalus?

A

meningitis
post trauma
post SAH

23
Q

What causes non communicating hydrocephalus?

A

tumours
cysts
aqueductal stenosis - enlarged frontal horns

24
Q

What causes sunsetting eyes?

A

midbrain compression

25
How do you treat hydrocephalus?
VP shunt
26
What is the presentation of normal pressure hydrocephalus?
idiopathic disease of the elderly | Hakims triad - abnormal gait, urinary incontinence, dementia
27
How is normal pressure hydrocephalus investigated?
CT and lumbar drain | test their gait, do a lumbar drain test, then test gait again and it should have improved
28
What is the risk factors of idiopathic intracranial hypertension?
young women fat
29
What is the presentation of idiopathic intracranial hypertension?
``` debilitating headaches double vision blurry vision tinnitus radicular pain papilloedema ```
30
What are the possible causes of idiopathic intracranial hypertension?
CSF imbalance hormones - oestrogen venous pressure transverse/sigmoid sinus stenosis
31
What is the first line treatment of idiopathic intracranial hypertension?
Acetazolamide/Topiramide - carbonic anhydrase inhibitor | AND WEIGHT LOSS
32
What else can be done for idiopathic intracranial hypertension?
CSF inversion - LP or VP shunt | intraventional radiology - intracranial venous shunt/stenting
33
What investigations are done for idiopathic intracranial hypertension?
LP CT/MRI head fundoscopy and opthamology review