CV monitoring Flashcards
Diagnosis of right atrial hypertrophy
Initial component of P larger in V1
Height > 2.5mm in any limb lead
Precordial limb lead placement
V1 4th intercostal space, right of sternum
V2 4th intercostal space, left of the sternum
V3 between V4 and V2
V4 5th intercostal space, left of sternum- mid clavicular line
V5 5th intercostal space, left of sternum- anterior axillary line
V6 5th intercostal space, left of sternum- mid axillary line
dx Left atrial hypertrophy
Terminal portion of diphasic P in V1 larger
Occurs with mitral stenosis, systemic HTN
Right ventricular hypertrophy dx
Right ventricular wall very thick; more depolarization toward V1
QRS in V1 positive, R waves get smaller
Left ventricular hypertrophy dx
Large S wave V1; larger R wave V5
Depth of V1 and height of V5= 35mm
Myocardial ischemia
Reduced supply of O2 from the coronary arteries
Inverted, symmetrical T wave
Myocardial injury
st elevation
Myocardial infarct (transmural) dx
Q indicated necrosis and makes diagnosis of “old” infarction
Must be significant…1mm wide or 1/3 QRS tall and 2 related leads
energy source and electrical circuits
Generator
Components of a pacemeaker
pulse generator and electrode leads
Reasons to have a pacemaker
Elderly….SSS
Anti-bradycardic treatment
insulated wire from generator to electrode
Lead
exposed metal end in contact with endocardium
Or epicardial leads
Electrode
neg electrode in chamber; positive electrode (grounding)
More sensitive to EMI
Unipolar
both electrodes in chamber being paced
Common; uses less energy
Bipolar
multiple electrodes within 1 lead but multiple chambers
Multipolar
Generic Code for Pacemaker Function
1 = chamber paced
2 = chamber sensing
3= Response to sensing
4= Rate modulation
5= multisite backing
Requirement for Bi-V pacing
Moderate/severe heart failure
EF 30-35%
Intraventricular conduction delays (BBB)
History of cardiac arrest
What is Bi V pacing
3 chambers; Ra, LV, RV.
2 Lead with two electrodes, one in Ra and one in RV and then a 3rd wire to the septum to the LV, risk of tamponade and bleeding because going through septum.
if intrinsic activity is perceived, chamber is not paced
inhibited
What is the purpose of Bi V Pacing
“cardiac resynchronization” (CRT)
Improves RV-LV activation time
Increases EF%
Very sick pts.
Done for bad conduction and the ventricles stop working together. Goal is to resynchronize = improve
what is CVP dependent on
Highly dependent on blood volume and vascular tone
Where is CVP measured
Pressure measured at the junction of vena cava and right atrium
pacemaker discharges if intrinsic activity IS sensed; used currently only for testing of devices
Triggered
Normal CVP for awake/ spont breathing
1-7 mmHg
Rate modulation occurs with: (4)
Vibration
Motion
Minute ventilation/ respiratory artifact
Right ventricular pressure
Indications for CVP lines
CVP monitoring
PA catheter placement
Transvenous cardiac pacing
Temporary hemodialysis
Drug administration
Rapid infusion of fluids/blood
Aspiration of air emboli
Inadequate peripheral access
Repeated blood testing
Multisite pacing is used for what pts
Atrial fib or dilated cardiomyopathies?
CVP A wave
Atrial contraction
Occurs after “P” wave
Increases atrial pressure
Provides “atrial kick”
Perioperative care for PM
Turn on pacmaker mode on monitor
move grounding pags away from PM
Interrogation pre/post op
CVP C wave
Interrupts the decreasing atrial pressure
Isovolumetric contraction of the ventricle
Tricuspid valve closed and ventricle bulges toward the atria
Follows “R” wave
Monopolar bovies and PM
EMI interference a Monopolar; bovie -> grounding to pad will cause more emi