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General Pathology > Class 3 > Flashcards

Class 3 Flashcards

1
Q

Etiology of inflammation

A

What caused it?

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2
Q

Duration of inflammation

A

how long is it?

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3
Q

Morphology of inflammation

A

What does it look like?

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4
Q

Acute inflammation

A

Sudden onset, short duration

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5
Q

Chronic inflammation

A

Longer duration (months/years)

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6
Q

Acute inflammation stops when?

A

Injurious stimulus removed

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7
Q

Acute inflammation initiated by

A

Local vessels near injury

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8
Q

Vessels initiate inflammation by

A

Alter permeability, allow leukocytes in

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9
Q

Inflammatory process stops by

A

Removal of stimuli

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10
Q

Mildest form of inflammation

A

Serous inflammation

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11
Q

Serous inflammation occurs?

A

Early stages and resolves easily

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12
Q

Inflammation rich in fibrin?

A

Fibrinous inflammation

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13
Q

Causes of Fibrinous inflammation

A

Bacterial infections

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14
Q

Inflammation caused by pus-forming bacteria

A

Purulent inflammation

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15
Q

Abscess

A

Local collection of pus

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16
Q

Inflammation of body surfaces or mucosa

A

Ulcerative inflammation

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17
Q

What ulcerative inflammation leads to

A

Ulceration or necrosis of epithelial lining

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18
Q

Ulcer is a ?

A

Defect in epithelium

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19
Q

Combination of ulcerative, fibrinous and purulent inflammation

A

Pseudomembranous inflammation

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20
Q

What is a pseudomembrane?

A

Exudate of fibrin, pus, cellular debris

21
Q

Diphteria

A

Pseudomembrane on throat

22
Q

Exudate containing monocytes, lymphocytes, macrophages, plasma cells is indication of?

A

Chronic inflammation

23
Q

Why does chronic inflammation perpetuate

A

Stimulate proliferation of fibroblasts &
Constantly recruit new inflammatory cells

24
Q

Chronic inflammation may lead to

A

Loss of function

25
Q

Chronic inflammation type not preceded by acute

A

Granulomatous Inflammation

26
Q

What accumulates at site of injury during Granulomatous inflammation

A

T-lymphocytes and macrophages

27
Q

t-lymphocyte and macrophage function in Granulomatous inflammation

A

Lymphocytes release cytokines
Cytokines turn macrophage into epithelioid cells

28
Q

Epithelioid functions during Granulomatous inflammation

A

Cells fuse together forming multinucleated giant cells

29
Q

Granulomas consist of

A

Lymphocytes, macrophages, multinucleated giant cells

30
Q

Leukocytosis

A

increase circulating WBC’s

31
Q

Continually dividing or mitotic cells (Tissue healing)

A

Stem cells divide and differentiate, replace lost cells

32
Q

Result of continually dividing cells (tissue healing)

A

Minimal tissue damage

33
Q

Quiescent Cells (tissue healing)

A

Don’t divide regularly, divide if necessary

34
Q

Outcome of quiescent cells (tissue healing)

A

Regeneration - replace w identical tissue

35
Q

Non-dividing cells (tissue healing)

A

Not able to divide and regenerate
Eg. neuron

36
Q

Outcome of non-dividing cells? (tissue healing)

A

Loss of functional capacity

37
Q

by first intention healing

A

would clean, necrotic tissue/edges close together

38
Q

by second intention healing

A

Large break, more inflammation, longer healing

39
Q

Leukocytes (WBC’s) PMNS

A

Scavenge initial injury site

40
Q

Connective tissue cells

A

Prod. scar tissue

41
Q

Epithelial cells

A

Divide and extend across wound

42
Q

Macrophages

A

Stay at healing site, produce cytokines/growth factors/meadiators

43
Q

Myofibroblasts

A

Smooth muscle cells and fibroblasts

44
Q

Angioblasts

A

blood vessel precursor

45
Q

Fibroblast produces?

A

prod. extracellular matrix

46
Q

Loss of function, deficient scar formation, excessive scar formation, infection are signs of?

A

Complications of healing

47
Q

Keloids

A

scar tissue overgrowth - excessive collagen

48
Q

Contracture

A

fixation/deformity of joint

49
Q

Adhesions

A

scar tissue bands joining 2 normally separated surfaces

General Pathology (12 decks)

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