Childrens Prthopaedics Flashcards

1
Q

What are the physis

A

Growth plates
The areas from which long bone growth occurs post natally

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2
Q

Bone development-how are flat and long bones formed

A

Flat bones
Intramembranous
Mesenchymal cells>bone

Long bones
Endochondral
Mesenchymal>cartilage>bone

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3
Q

Intramembranous ossification

A

Condensation of mesenchymal cells which differentiate into osteoblasts forming an ossification centre

Secreted osteoid traps osteoblasts which become osteocytes

Trabecular matrix and periosteum form

Compact bone develops superficial to cancellous bone. Crowded blood vessels condense into red bone marrow

Form cranial bones and clavicle

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4
Q

Endochondral ossification

A

All other long bone formation at both the primary and secondary ossification centre

Primary ossification centre
Sites of pre natal bone growth through endochondral ossification from the central part of the bone

Secondary ossification centers
Occurs post Nataly after the primary ossification centre and long bones often have several (physis)

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5
Q

Where is the first ossification centre

A

Middle of diaphysis of bone

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6
Q

Prenatal bone growth

A

Mesenchymal, differentiation at primary centre

The cartilage model of the future bony skeleton forms

Capillaries penetrate the cartilage. Calcification at the primary ossification centre spongy bone forms
Perichondrium transforms into periosteum

Cartilage and chondrocytes continue to grow at the ends of the bone

Secondary ossification centers develop

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7
Q

Secondary ossification centers

A

Long bone lengthening
Happens at physis (physeal plate)
Zone of elongation in a long bone
Contains cartilage
Epiphyseal side hyaline cartilage active and dividing to form hyaline cartilage matrix
Diaphyseal side cartilage calcified and dies and then replaced by bone

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8
Q

How are children’s and adults skeleton different

A

Elasticity
Physis
Speed of healing
Remodeling

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9
Q

Elasticity of children’s bone

A

Children’s bone bends more and is more elastic than an adults
Increased density of haversian canals therefore
plastic deformity (bends before breaks)
Buckle fracture (tarus like column)
Greenstick (like the tree,one cortex fractures but doesn’t break the other side)

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10
Q

Growth rate

A

Growth stops when physeal plates close
Gradual closure affected by puberty menarche and parenteral height

For girls complete at 15-16
For boys at 18-19

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11
Q

What can physeal injuries cause

A

Leads to growth arrest
This can lead to deformity
Are categorized by salter Harris

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12
Q

Common children congenital conditions

A

Developmental dysplasia of the hip
Club foot
Achondroplasia
Osteogenesis imperfecta

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13
Q

Developmental dysplasia of the hip

A

Group of disorder of the neonatal hip where the head of the femur is unstable or incongruous in relation to the acetabulum
A packaging disorder
Spectrum with dysplasia-subluxation-dislocation
(Ball not in socket)

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14
Q

Risk factors of developmental dysplasia

A

Female 6:1
First born
Breech
FH
Oligohydramnios
Native American/laplanders swaddling of hip
Rare in African American or Asian (less amniotic fluid)

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15
Q

Examination if developmental dysplasia of the hip

A

Picked up on baby check screening
Rom of hip -limitation in hip abduction,leg length (galeazzi)
In those 3 months or older barlow and ortalani are non sensitive

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16
Q

Ivx of developmental dysplasia of hip

A

US from birth to 4 months
After 4 months do X ray
If prior to 6 weeks needs to be age adjusted
Measures the acetabular dysplasia and the position of the hip

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17
Q

Treatment of developmental dysplasia of hip

A

Reducible hip and <6months
Palvik harness is 92% effective

Failed palvik harness or 6-18 months
-secondary changes in capsule and soft tissue
MUA and closed reduction and spica

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18
Q

Clubfoot and treatment

A

Congenital talipes equinovarus
Deformity of the foot
Highest in Hawaiians
50% bilateral
Genetic
5% likely of siblings
Familial in 25%
PITX1 gene

Treatment
Ponseti method is the gold standard
1.series of casts to correct deformity
2.many require operative treatment,soft tissue releases
3.foot orthosis brace
4.slme will require further operative intervention to correct final deformity

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19
Q

CAVE deformity

A

Cavus-high arch:tight intrinsic,FHL,FDL
Adductus of foot-tight tib post and ant
Varus-tight tendoachilies, tib post,tib ant
Equinous-tight tendoachiles

20
Q

Achondroplasia

A

Most common skeletal dysplasia
Autosomal dominant
G380 mutation of FGFR3
Inhibition of chondrocyte proliferation in the proliferative zone of physis
Results in defect in Endochondral bone formation
Normal cognitive development
Significant spinal issues
(Proliferative zone affected via fgfr3)

21
Q

Rhizomelic dwarfism

A

Humerus shorter than forearm
Femur shorter than tibia
Normal trunk
Adult height approx 125cm

Normal cognitive development
Significant spinal issues

22
Q

Osteogenesis imperfecta

A

Brittle bone disease
Hereditary-autosomal dominant or recessive
Decreased type 1 collagen due to decreased secretion,production of abnormal collagen

Insufficient osteoid production

23
Q

What does OI affect

A

Bones
-fragility fractures,short stature,scoliosis

Non orthopedic manifestation
Heart,blue sclera,dentinogenesis imperfecta (brown soft teeth,worminian skull,hypermetabolism

24
Q

Paediatric fractures

A

Pattern
Anatomy
Intra/extra articular
Displacement
Salter Harris

25
Q

Speed and remodelling

A

Depends on location and age of patient
Younger child heals more quickly
Physis and knee grows more
Physis at extreme of upper limb grows more

Inflammation—>repair/callus—>remodelling

26
Q

OI treatment

A

Supportive
Surgical
Medical

27
Q

Pattern of fractures

A

Transverse
Oblique
Spiral
Comminuted
Avulsion

28
Q

Anatomy of fractures

A

Proximal
Middle diaphysis
Distal
Avulsion

29
Q

Intra articular/extra articular

A

Primary bone healing
Heals by direct union
No callus formation
Preferred healing pathway in in Intra articular fracture as minimise risk of post traumatic arthritis

Secondary bone healing
Bone healing by callus

30
Q

Displacement

A

Displaced
Angulted
Shortened
Rotated

31
Q

Salter Harris

A

Classification of physeal injuries
1.physial separation
2.fracture traverses physis and exits metaphysis (above)
3.fracture traverses physis and exits epiphysis (lower)
4.fracture pssses through epiphysis physis and metaphysis
5.crush injury to physis

Risk of growth arrest increases from 1-5
Type 2 injuries most common

32
Q

Growth arrest

A

Injuries to physis can cause growth arrest
Whole physis-limb length discrepancy
Partial-angulation as the non affected side keeps growing

33
Q

Growth arrest treatment

A

Aim is to correct deformity
Minimise angular deformity
Minimise limb length difference

Limb length correction-shorten long side or longer the short side

Angular deformity-stop the growth of the unaffected side
Reform the bone (osteotomy)

34
Q

Fracture management

A

Resuscitated
Reduce
Restrict
Rehab

35
Q

reduce

A

correct the deformity and displacement
reduce secondary injury to soft tissue/nv structures

closed-reduce a fracture without making an incision eg traction and manipulation in a and e

open-making an incision and realignment of fracture under direct visualisation

36
Q

closed reduction

A

gallows traction
holding the skin the long bones of the lower limb can be reduced

closed reduction to correct deformity

37
Q

restrict

A

maintain fracture reduction
provides stability for the fracture to heal
children rarely have issues with bone not healing

external via splints or plaster

internal via plates and screws or intramedullary device

38
Q

what are used most commonly in paedeatric fractures

A

plasters and splints as it removes need for operative internal fixation

if operation needed consider the ongoing gorwth at the physis and metawork may be needed to be removed in the future

39
Q

rehabilitate

A

Children generally rehabilitate very quickly

Play is a great rehabilitator

Stiffness not as major issue as in adults

Use it, Move it and Strengthen!

40
Q

limping child

A

septic arthritis
transient synovitis
perthes
SUFE

41
Q

septic arthritis

A

orthopaedic emergency
can cause irreversible long term problems in the joint therefore needs surgical washout of the joint to clear the infection

history is key-duration,other recent illness,associated joint pain

42
Q

what classification can help determine septic arthritis

A

kochers classification
Non weight bearing
ESR >40
WBC >12,000
Temperature >38
the higher the score the more likely its present

43
Q

what can we diagnose is septic arthrits has been ruled out

A

transient synovitis (reactive arthritis)

supportive treatment with abx

44
Q

perthes disease

A

Idiopathic necrosis of the proximal femoral epiphysis

Usually in those 4-8 years old
Male 4:1 Female

Septic arthritis needs to be excluded first

Treatment is usually supportive in the first instance

45
Q

SUFE

A

Slipped upper femoral epiphysis

The proximal epiphysis slips in relation to the metaphysis

Usually obese adolescent male
12-13 years old during rapid growth

Septic arthritis needs to be excluded first

Treatment is operative fixation to prevent further slip and minimise long term growth problems