Chapter 38 - Complications of Otitis Media Flashcards
3 main routes of spread of OM
hematog
direct extension
thrombus propagation (thrombophlebitis of local perforating veins)
Bacteria involved with complicated AOM
Most common are still S Pneumo/H Flu/Morax, but may involve bacteria with increased resistance
MRSA, Pseudomonas, Klebsiella, P acnes, bacteroides species. Often polymicrobial
Which involved bony destruction and granulation tissue - AOM or CSOM?
CSOM
Which structural deformities increase risk of direct extension of infection in middle ear and mastoid
T bone trauma
Mondini
enlarged vestibular aqueduct
prior surgery
How ear infections lead to meningitis
Usually AOM
Hematogenous spread
HA, nausea, nuchal rigidity, photophobia, AMS, fever
Two critical steps to workup meningitis as AOM complication
CSF analysis
CT to r/o other complications
Examples of direct extension of ear infection
postauricular abscess, Bezold abscess, sigmoid sinus thrombosis, epidural abscess, subdural empyema
Bezold’s abscess
Infection erodes the mastoid cortex medial to SCM attachment, where digastric attaches
Extends to infratemporal fossa
What makes a Bezold’s abscess difficult to palpate?
Deep to cervical fascia enveloping SCM and trapezius
Name all extracranial/intratemporal OM complications
Acute mastoiditis, coalescent mastoiditis, chronic mastoiditis, postauricular abscess, bezold ascess, temporal abscess, petrous apicitis, labyrinthine fistula, facial paralysis, acute suppurative labyrinthitis, encephalocele, CSF leak, hearing loss (CHL/SNHL)
Name all intracranial complications of OM
Meningitis, brain abscess, subdural empyema, epidural abscess, lateral sinus thrombosis, otic hydrocephalus
Age at which OM is more likely to be complicated
60-80% episodes of complicated OM occur in 0-20 yo
Most common AOM complication
OME
Which nerve palsy can petrous apicitis present with?
VI
Where does the EAC tend to sag with cholesteatoma canal erosion?
posterior superior EAC