Chapter 34 - Tinnitus Flashcards

1
Q

Percentage of people with tinnitus

A

15%

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2
Q

Hypothesized reason for tinnitus

A

change in peripheral auditory function –> central neural hyperexcitability, cortex reorganization

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3
Q

Percentage of tinnitus pts w/ NL hearing on audiogram

A

10%

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4
Q

2 potential causes of pulsatile tinnitus

A

vascular malformation

neoplasm

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5
Q

Causes of clicking tinnitus

A
Mandibular condyle (TMJ)
palatal or stapedial myoclonus
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6
Q

Type of HL associated with tinnitus caused by high dose salicylates

A

reversible mild to moderate SNHL (flat)

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7
Q

somatosounds

A

objective tinnitus, may be audible to examiner
myoclonus of tensor tympani
pulsatile variation in vessels near ear

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8
Q

phantom limb concept of tinnitus

A

In animal studies, auditory cortex reorganizes after sensory deprivation similar to how somatosensory cortex reorganizes after amputation

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9
Q

Vascular tinnitus

A

glomus tumor
dehiscent jug bulb, ICA
AVMs between occipital a (medial to mastoid) and transverse sinus
loop compressing CN VIII

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10
Q

Venous hum tinnitus - causes

A

Impingement of jugular vein by C2

High output heart (anemia, exercise, pregnancy, thyrotoxicosis)

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11
Q

Causes of pulsatile tinnitus other than vascular

A
otitis media
otosclerosis
Paget disease
IIH
MS
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12
Q

EAC pathologies causing tinnitus

A

cerumen
foreign body
hair, insect, other small objects

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13
Q

Muscles involved in palatal myoclonus

A

TVP, LVP, salpingopharyng, superior constrictor

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14
Q

How to evaluate palatal myoclonus

A

Nasopharyngoscopy, as opening mouth wide could cause the myoclonus to stop

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15
Q

Systemic diseases causing tinnitus

A

MS, stroke, tumor, trauma, syphilis, malaria, psychogenic, other degenerative processes

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16
Q

Objective findings in patients with tinnitus and normal audiometric thresholds

A

smaller wave I ABR amplitude

suggests early cochlear damage –> reduced neuronal input

17
Q

Can noise exposure lead to tinnitus

A

Isolated loud noise tinnitus resolved in hours to days

Repeated episodes –> may be permanent

18
Q

Relationship between tinnitus and hyperacusis

A

May be same central cause (decreased cochlear input)

Almost all hyperacusis pts eventually develop tinnitus

19
Q

How to salicylates cause hearing loss

A

competitive inhibitors of chloride at anion binding site of prestin (motor protein of outer hair cell) –> reversible alteration of outer hair cell function
Reverse within 1-3 days of d/c

20
Q

List of drugs causing tinnitus

A

ACEi, anesthetics (lidocaine, bupivacaine, dyclonine), aminoglycosides, imipenem, Vanc, bactrim, aztreonam, erythromycin, cipro, antidepressants, phenergan, sudafed, anti-malarials, B blockers, CCB, diuretics, NSAIDs, sedatives
Misc: albuteraol, carbamazepine, flexeril, cyclosporine, benadryl, flecainide, isotretinoin, lithium, methergine, nicorette, prazosin, omperazole, Hep B vaccine, sodium nitroprusside, tocainide

21
Q

Percent of acoustic neuromas with tinnitus

A

10% present this way

80% eventually have it

22
Q

Treatment of tinnitus with auditory stimulation

A

fan, radio, generator, TV
Hearing aid (if frequency in amplification range)
custom devices

23
Q

Tinnitus retraining therapy

A

Counseling to educate on cause of tinnitus, decouple tinnitus from emotional response
Sound therapy to gradually habituate and thus reduce strength of tinnitus

24
Q

Meds for tinnitus

A

None better than placebo
Can treat stress/anxiety accompanying it with TCA, SSRI, benzo
Botox for myoclonus

25
How IV lidocaine stops tinnitus
augments natural central inhibition of auditory firing (which may be lost and then cause tinnitus, one theory)
26
Transcranial magnetic stimulation for tinnitus
low level electric brain currents which inhibit hyperexcited regions of brain
27
Treatment of tinnitus associated with bilateral profound HL
CI - cures tinnitus in 86% of cases
28
Treatment of myoclonus tinnitus
botox | tympanostomy tubes