chapter 2 Flashcards

1
Q

Recent evidence of cerebral dominance says….

A

both hemipsheres contribute to language and non-language functions

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2
Q

left hemisphere dominance

A

approx. 95% of right handed people & 70% of left handed people are left hemisphere dominant

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3
Q

bilateral dominance

A

approx. 25-30%

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4
Q

right hemisphere dominance

A

5%

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5
Q

recent view of lang. functions

A

different neural networks control different functions meaning all structures work together

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6
Q

broad classifications of aphasia

A

nonfluent, fluent, subcortical, mixed

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7
Q

nonfluent aphasia are characterized by

A

relatively good auditory comprehension and deficits in lang expression; broca’s, transcortical motor, global

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8
Q

fluent aphasia are characterized by

A

relatively fluent speech with deficits in auditory comprehension;wernicke’s, conduction, anomic, transcortical sensory

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9
Q

subcortical aphasias are characterized by

A

borderline aphasias; deficits in both expression and comprehension but severity varies.

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10
Q

mixed aphasias

A

presence of 2+ aphasias at same time

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11
Q
A
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12
Q

lesions limited to Broca’s area can have what deficits?

A

mild deficits in prosody, mild agraphia with occasional anomia; dysarthria

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13
Q

aphemia (broca’s)

A

unable to articulate words BUT can comprehend and write

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14
Q

Broca’s aphasia is a nonfluent type of aphasia with

A

deficits in language expression and relatively intact comprehension

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15
Q

transcortical motor aphasia - most commonly affected areas

A

supplementary motor cortex and areas connecting Broca’s area

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16
Q

transcortical motor aphasia - most commonly affected blood supply

A

-anterior cerebral artery
-anterior branches of middle cerebral artery

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17
Q

transcortical motor aphasia is a non-fluent type of aphasia with

A

intact repeitition skills

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18
Q

global aphasia - most commonly affected areas

A

lesions affecting both divisions of MCA and left fronto-parieto-temporal regions (including zone of language)

19
Q

global aphasia

A

most severe form of aphasia with a generalized effect on communication skills, deficits in both comprehension and expression

20
Q

wernickes aphasia - most commonly affected areas

A

posterior 3rd of superior-temporal gyrus (near wernicke’s area)

21
Q

wernickes aphasia - when it effects the primary temporal lobe lesions will result in…

A

deficits in understanding indvidual, isolated spoken words but can understand words with context (in a sentence)

22
Q

wernickes aphasia - when it effects the posterior temporal lobe lesions will result in more….

A

more changes in vision, more deficits in written language, less in language comprehension

23
Q

wernicke’s aphasia

A

fluent type of aphasia with poor comprehension

24
Q

conduction aphasia - most commonly affected areas

A

supramarginal gyrus, underlying white matter pathways, wernicke’s area, left insula, and auditory cortex

25
conduction aphasia lesions...
isolated lesions in inferior parietal lobe or combo of lesions that affect left primary auditory cortex
26
types of conduction aphasia
afferent and efferent
27
afferent conduction aphasia
temporal lobe; impaired repetition and memory deficis
28
efferent conduction aphasia
parietal and insular regions - phonemic deficits
29
conduction aphasia
fluent type of aphasia with impaired repeitition skills
30
anomic aphasia - most commonly affected areas
angular gyrus and second temporal gyrus
31
anomic aphasia - damage to left inferior frontal region will have what deficits
action word deficits
32
anomic aphasia - damage to left temporal region will have what deficits
deficits in naming nouns
33
anomic aphasia
fluent type of aphasia with anomia as the primary symptom
34
transocrtical sensory aphasia - most commonly affected areas
posterior parietal-temporal regions (excluding wenicke's) and bilateral lesions
35
transcortical sensory aphasia
can co-occur with alzheimers disease; fluent type of aphasia with intact repetition skills
36
borderline/subcortical aphasia
-often associated with damage to thalamus and basal ganglia
37
anterior damage to internal capsule and putamen
severe form of dysaarthric articulation, mild repetition problems, naming, word finding, auditory comprehension problems, severe writing deficits, moderate reading difficulties
38
posterior capsular putamen damage
severe auditory comprehension problems, fluent speech, significant naming/word finding problems, moderate reading and writing problems
39
anterior and posterior damage to thalamus
global aphasia, nonfluent and extremely limited spontaneous speech, severe auditory comprehension issues, significant reading and writing, repetition, an naming difficulties
40
posterior cerebral artery damage
parietal lobe (deficits in written lang)
41
middle cerberal artery damage
deficits in oral language expression
42
internal carotid artery damage
deficits in spoken and written language
43
MCA both inferior and superior dvisions
more likely to cause aphasias with repetion deficits