chapter 2 Flashcards

1
Q

Recent evidence of cerebral dominance says….

A

both hemipsheres contribute to language and non-language functions

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2
Q

left hemisphere dominance

A

approx. 95% of right handed people & 70% of left handed people are left hemisphere dominant

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3
Q

bilateral dominance

A

approx. 25-30%

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4
Q

right hemisphere dominance

A

5%

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5
Q

recent view of lang. functions

A

different neural networks control different functions meaning all structures work together

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6
Q

broad classifications of aphasia

A

nonfluent, fluent, subcortical, mixed

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7
Q

nonfluent aphasia are characterized by

A

relatively good auditory comprehension and deficits in lang expression; broca’s, transcortical motor, global

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8
Q

fluent aphasia are characterized by

A

relatively fluent speech with deficits in auditory comprehension;wernicke’s, conduction, anomic, transcortical sensory

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9
Q

subcortical aphasias are characterized by

A

borderline aphasias; deficits in both expression and comprehension but severity varies.

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10
Q

mixed aphasias

A

presence of 2+ aphasias at same time

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11
Q
A
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12
Q

lesions limited to Broca’s area can have what deficits?

A

mild deficits in prosody, mild agraphia with occasional anomia; dysarthria

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13
Q

aphemia (broca’s)

A

unable to articulate words BUT can comprehend and write

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14
Q

Broca’s aphasia is a nonfluent type of aphasia with

A

deficits in language expression and relatively intact comprehension

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15
Q

transcortical motor aphasia - most commonly affected areas

A

supplementary motor cortex and areas connecting Broca’s area

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16
Q

transcortical motor aphasia - most commonly affected blood supply

A

-anterior cerebral artery
-anterior branches of middle cerebral artery

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17
Q

transcortical motor aphasia is a non-fluent type of aphasia with

A

intact repeitition skills

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18
Q

global aphasia - most commonly affected areas

A

lesions affecting both divisions of MCA and left fronto-parieto-temporal regions (including zone of language)

19
Q

global aphasia

A

most severe form of aphasia with a generalized effect on communication skills, deficits in both comprehension and expression

20
Q

wernickes aphasia - most commonly affected areas

A

posterior 3rd of superior-temporal gyrus (near wernicke’s area)

21
Q

wernickes aphasia - when it effects the primary temporal lobe lesions will result in…

A

deficits in understanding indvidual, isolated spoken words but can understand words with context (in a sentence)

22
Q

wernickes aphasia - when it effects the posterior temporal lobe lesions will result in more….

A

more changes in vision, more deficits in written language, less in language comprehension

23
Q

wernicke’s aphasia

A

fluent type of aphasia with poor comprehension

24
Q

conduction aphasia - most commonly affected areas

A

supramarginal gyrus, underlying white matter pathways, wernicke’s area, left insula, and auditory cortex

25
Q

conduction aphasia lesions…

A

isolated lesions in inferior parietal lobe or combo of lesions that affect left primary auditory cortex

26
Q

types of conduction aphasia

A

afferent and efferent

27
Q

afferent conduction aphasia

A

temporal lobe; impaired repetition and memory deficis

28
Q

efferent conduction aphasia

A

parietal and insular regions - phonemic deficits

29
Q

conduction aphasia

A

fluent type of aphasia with impaired repeitition skills

30
Q

anomic aphasia - most commonly affected areas

A

angular gyrus and second temporal gyrus

31
Q

anomic aphasia - damage to left inferior frontal region will have what deficits

A

action word deficits

32
Q

anomic aphasia - damage to left temporal region will have what deficits

A

deficits in naming nouns

33
Q

anomic aphasia

A

fluent type of aphasia with anomia as the primary symptom

34
Q

transocrtical sensory aphasia - most commonly affected areas

A

posterior parietal-temporal regions (excluding wenicke’s) and bilateral lesions

35
Q

transcortical sensory aphasia

A

can co-occur with alzheimers disease; fluent type of aphasia with intact repetition skills

36
Q

borderline/subcortical aphasia

A

-often associated with damage to thalamus and basal ganglia

37
Q

anterior damage to internal capsule and putamen

A

severe form of dysaarthric articulation, mild repetition problems, naming, word finding, auditory comprehension problems, severe writing deficits, moderate reading difficulties

38
Q

posterior capsular putamen damage

A

severe auditory comprehension problems, fluent speech, significant naming/word finding problems, moderate reading and writing problems

39
Q

anterior and posterior damage to thalamus

A

global aphasia, nonfluent and extremely limited spontaneous speech, severe auditory comprehension issues, significant reading and writing, repetition, an naming difficulties

40
Q

posterior cerebral artery damage

A

parietal lobe (deficits in written lang)

41
Q

middle cerberal artery damage

A

deficits in oral language expression

42
Q

internal carotid artery damage

A

deficits in spoken and written language

43
Q

MCA both inferior and superior dvisions

A

more likely to cause aphasias with repetion deficits