Chapter 16 Flashcards

1
Q

Temperament

A

Any emotional predisposition or personality traits

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2
Q

Child psychopathology

A

The emotional and behavioral manifestations of psychological disorders in children and adolescents. Characteristics that signify mental illness and adults such as difficulty with emotional regulation often occur normally developing children. Additionally symptoms of some disorders are quite different and children compared to adults.

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3
Q

Prevalence rates of childhood disorders

A

31.9% reported symptoms of any anxiety disorder 19.1% demonstrated a behavior disorder and 14% reported symptoms of a depressive or bipolar disorder. Out of 10,000 US adolescence 13 to 18 years old almost half of it already experienced significant mental health concerns. Depressive and bipolar disorder symptoms caused the greatest distress. Females get more depression and posttraumatic stress reactions whereas males demonstrated more inattention and hyperactivity. 40% had more than one disorder.two thirds of those with mental illness receive no treatment. Low treatment rates for depression is the worst.

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4
Q

Rates of youth taking medication

A

Medication is most frequent treatment among children and adolescents who do not receive treatment. 7.5% of youth from 6 to 17 years old take medication for emotional or behavioral difficulties including 9.7% to boys and 5.2% of girls. Low income youth who qualify for subsidized healthcare of the most likely to take psychotropic medication

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5
Q

Internalizing disorders

A

Conditions involving emotional symptoms directed inward. Anxiety and depressive disorder’s are the most common internalizing disorders they often lead to substance-abuse and suicide. They include anxiety trauma and stressor related disorders early in life, and mood disorders early in life

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6
Q

Separation anxiety disorder

A

Severe distress or worry about leaving home being alone or being separated from primary caregivers

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7
Q

Selective mutism

A

Consistent failure to speak in certain social situations

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8
Q

Anxiety disorders early in life

A

Separation anxiety disorder, selective mutism, attachment disorders, posttraumatic stress disorder, nonsuicidal self injury. Anxiety disorders are the most prevalent mental health disorder and childhood and adolescent. Of 32% who has experience and anxiety disorder specific phobias 19% and social phobia 9% are the most common.if untreated can lead to adult anxiety disorders. Inhibited and fearful children are at higher risk for anxiety disorders and overprotective or controlling parent practices, no parent to warmth, or perceive parental rejection can make the issue worse.childhood anxiety disorders are most effectively treated with CBT. These children show exaggerated autonomic responses and are apprehensive in new situations preferring to stay at home or another familiar environments.

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9
Q

Attachment disorders

A

Infants and children raised and stressful environments that little predictable parenting and nurturing show difficulties with emotional attachments and social relationships. Symptoms must be a parent before age 5 and went early circumstances prevent the child from forming stable attachments. Changes in primary caregiver, persistent neglect of physical or psychological safety including physical abuse and environments the void of stimulation or affection can disrupt attachment.

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10
Q

Reactive attachment disorder or RAD

A

Show little trust that adults will attend their needs. Do not readily seek or respond to comfort attention or neutering. A trauma related disorder characterized by inhibited avoidant social behaviors and reluctance to seek or respond to attention or nurturing. Use avoidance as a psychological defense, and experience difficulty responding to orinitiating social or emotional interactions. Rarely show positive emotions and demonstrate irritability sadness or fearfulness when interacting with adults. Symptoms often disappear of children begin to receive protectable caretaking and nurturance

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11
Q

Disinhibited social engagement disorder DSED

A

They socialize effortlessly but indiscriminately and readily become superficially attached to strangers and casual acquaintances. Interact with unfamiliar adults in an overly familiar Man are both verbally and physically while moving away from caregivers. They often have a harsh punishment history or inconsistent parenting in addition to emotional neglect and limited attachment opportunities. Symptoms are often persistent and continue into adulthood.

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12
Q

Post dramatic stress disorder in early life

A

Youth experience recurrent distressing memories of a shocking experience and desperately want to avoid any cues associated with the event. Many entail a)distressing dreams, b) intense psychological or physiological reactions to thoughts or cues associated with the event, c) episodes of play acting the event sometimes without apparent distress and D)dissociative reactions in which the child appears to re-experience the trauma or seems unaware of present surroundings. They also seem socially withdrawn show few positive emotions or seem disinterested in activities previously enjoyed. According to DSM behavioral evidence of PTSD includes Angry aggressive behavior or temper tantrum’s, difficulty sleeping or concentrating, and exaggerated startle response or vigilance for possible threats. 8% for girls and 3.2% for boys lifetime prevalence. CBT helps.

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13
Q

Nonsuicidal self injury

A

Intentional self inflicting harm intended to provide relief from negative feelings or to induce positive feeling; can also involve a preoccupation with engaging in self harm. Often there is a desire to resist the impulse to self-injure. This is a diagnostic category undergoing further study. For diagnosis individual must display he’s intentional behaviors at least five times over one year. Pain produces relief from uncomfortable feelings or a temporary sense of calm and well-being. A secondary motivation for some is that the self injurious behavior serves as a form of self punishment. Those who self injure tend to be highly self critical and have difficulty expressing in regulating emotions.risk of substance-abuse or attempted suicide. A negative cognitive style a negative self talk are associated with increased frequency of it and increased likelihood of suicide. Males more likely hit or burn themselves females more frequently cut themselves. 14 to 17% haveengaged at least once. Only few do it repeatedly. DBT is effective.

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14
Q

Childhood and abuse and a neglect

A

678,810 youth were victims of child neglect or physical or sexual abuse in 2012. 1640 died. Majority of deaths from abuse involve children three or younger and 80% of cases the perpetrator is one or both parents. Factors are poverty parental immaturity and lack of parenting skills. Many adults will be used for themselves abused as children many of the parents involved are young high school dropouts and under severe stress many of personality disorders and low tolerance for frustration or abuse alcohol and other substances. And child sexual abuseperpetrators are often friends or other family members in the parent is unaware that the abuse occurred

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15
Q

Mood disorders early in life

A

Depression most prevalent among females an older adolescence. Environmental factors are a frequent cause of depression in childhood where is genetic or other biological factors are more of an influence during adolescence. Increased by conditions such as childhood physical or sexual abuse, parental mental or physical illness, or loss of an attachment figure. Use with depressive disorder’s have a negative self-concept and are more likely to engage in self blame and criticism. Evidence-based treatment include individual or group CBT family focused therapy and programs focused on building resilience based on positive psychology principles strong association between depressive disorder is an adolescent suicide ideation and suicide attempts. SSRIs despite warnings may be helpfulin severely depressed

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16
Q

Disruptive mood dysregulation disorder

A

DM DD is characterized by chronic irritability and severe mood dysregulation including recurrent episodes of temper triggered by common stressors such as interpersonal conflict or being denied a request. And her reactions are extreme and both intensity and duration and may involve verbal rage or physical aggression. This is a depressive disorder even though behavioral symptoms are directed outward they reflect an irritable angry or sad mood state. For the diagnosis the mood between temper Appisodes must be irritable or angry most of the day nearly every day. Outbursts are present in at least two settings and occur at least three times a week for most months over a year. Cannot make the diagnosis until the child is at least six and symptoms must be evident before 10.symptoms are overlapping with bipolar disorder and this must be rolled out interventions are quite different for these two disorders. Ranges between. 8 to 8.2% many have comorbid disorders associated with emotional dysregulation such as depressive disorder’s or oppositional defiant disorder. Prevalences to the 5% more often in boys.

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17
Q

Pediatric bipolar disorder PBD

A

A serious disorder involving depressive and energize episode similar the mood swings seen in adults mood changes depressive episodes and the Departure from individuals typical functioning. May involve diminished need for sleep distractibility talkativeness or inflated self-esteem also may distribute recurring depressive episodes or periods of on characteristic irritability that alternate with the energized episodes can develop gradually are suddenly. Change from child’s normal mode or temperament. Youth often demonstrate rapid cycling of moods combined with difficulties in regulating behavior and social emotional functioning elevated neural logical responsiveness to environmental stimuli and various brain abnormalities. The new DM DD category is hope to help for greater diagnostic accuracy since some experts believe this prevalence rates are inflated. In adolescence a estimated to be 3% lifetime prevalence. Medications therapeutic techniques and psychosocial interventions are similar to those for adults use of lithium an antipsychotic medications does concern some mental health professionals family focused interventions help children learn to regulate their symptoms. Diagnosis usually occurs around age 10 through adolescence about five years later then DM DD. Prevalence is 1% affects boys and girls equally

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18
Q

Externalizing disorders

A

Sometimes called disruptive behavior disorders, include disruptive, impulse control and conduct disorders. Conditions associated with symptoms that are distressing to others. Can be distressing to parent and result in negative parent child interaction’s, high family stress and negative feelings about parenting. diagnosis of a disruptive impulse control or conduct disorder requires a persistent pattern of behavior that : is atypical for the child’s culture, gender, age, and developmental level; and is severe enough to cause distress of the child or others,or negatively affect social or academic functioning. They include oppositional defiant disorder intermittent explosive disorder and conduct disorder. Externalizing behavior disorders in thechild are associated with the development of depressive disorder’s in adulthood.

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19
Q

Oppositional defiant disorder

A

A childhood disorder characterized by negativistic argumentative and hostile behavior patterns that continues for at least six months directed at parents teachers and others in authority. At least four symptoms involving short tempered resentful blaming spiteful or hostile behavior’s must be present it’s mild if they occur and only one setting and severe if the behaviors occur in threeor more settings. Symptoms can resolve especially with intervention although some youth begin the demonstrate the more serious rule violations associated with conduct disorder. ODD has two components, one involving negative affect an emotional dysregulation such as angry irritable mood, and the other involving defiant and oppositional behavior. Negative affect predicts future depressive symptoms whereas oppositionalbehaviors are more productive of delinquency and conduct disorder. Half of those with ODD display inattention and hyperactivity

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20
Q

Intermittent explosive disorder or IED

A

Condition involving frequent lower intensity outbursts or low-frequency high intensity outbursts of extreme verbal or physical aggression. A diagnosis involves it’s under treated and underdiagnosed category. Diagnosis involves recurrent outbursts of extreme verbal or physical aggression that occur twice weekly for at least three months or three outbursts occurring within one year. That involve damaged injury people animals or property low-frequency high intensity outbursts. Outbursts occur suddenly in response to mine or provocation and do not involve premeditation they are exaggerated angry or impulsive reactions that caused distress or impairment interpersonal functioning. The moon is normal between outbursts unlike DM DDmust be at least six years old to be diagnosed. It’s associated with early exposure to family aggression and violence and enter personal trauma maybe diagnosed and individuals with ADHD, conduct disorder, or ODD if the outbursts of her and meet the criteria for IED. 63% of adolescents had experience angry outbursts however 7.8% met the criteria

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21
Q

Conduct disorder

A

A persistent pattern of behavior that violates the rights of others, including aggression, serious rule violations, and illegal behavior reflects dysfunction in the individual rather than a pattern of behavior within the person subculture, and include serious violations of rules and social norms and disregard for the rights of others. Diagnosis requires at least three behaviors involving: a) deliberate aggression such as bullying physical fights use of weapons cruelty to people or animals aggressive theft or sexual conduct; b) destruction of property including fire setting; c) theft or deceit such as stealing, forgery home or car invasion or conning others; or D)serious violation of rules such as staying out at night truancy running away. Disorderly condo at increases or becomes more serious with age. Boys with CD are often involved in confrontational aggression whereas girls are more likely to display truancy substance abuse or lying. 2 to 9% of youth meet diagnostic criteria for CD half also have inattention and hyperactivity.

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22
Q

Limited prosocial emotions

A

According to DSM-V some youth diagnosed with cd have this they display minimal guilt or remorse and are consistently I’m concerned about the feelings of others, their own wrongdoing, or poor performance at school or work. They are good at manipulating others and may appear superficially polite and friendly when they have something to gain. Cruelty aggression and a pervasive lack of remorse or common characteristics of the subgroup. They show limit Limited neural responsiveness and brain regions associated with empathy when presented with pictures of people in pain. In an MRI adolescents with CD and Calais traits demonstrated strong pleasure responses to video clips of people experiencing pain and distress. These traits are at high-risk for continuing criminal behavior and for receiving a diagnosis of antisocial personality disorder or later inLife

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23
Q

Etiology Of externalizing disorders

A

Biological factors appear to exert the greatest influence on the development of conduct disorder. Antisocial behavior has been linked to brain abnormalities associated with deficits in social information processing as well as reduced activity in the amygdala in situations associated with fear. These deficits appear to decrease the ability to learn from rewards and punishments. Risk ofconduct disorder is increased when carriers of the genotype low activity MAOA, an allele associated with fear regulating circuitry in the amygdala are subjected to childhood maltreatment. Reduced activity of the autonomic nervous system and an increased need for stimulation to achieve optimal arousal is also associated with conduct disorder in males; may account for risk-taking behaviors associated with the disorder. Elevated stress hormones cortisol have been associated with the symptoms of impulsive aggression where as low cortisol occurr in youth with callous and unemotional traitsand predatory aggression

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24
Q

Family and social context role in the development of externalizing disorders

A

Child’s environment moderates the relationship between individual vulnerability and the age at which antisocial behavior emerges. Parents and teachers able to exert more influence on the behavior of children with antisocial tendencies during childhood compared to adolescence. In adolescence peer influences dominate. Aggressive behaviors associated with harsh or inconsistent discipline, a punitive inconsistent or impatient man or in response to typical childhood misbehaviors leads to disruptive behavior power struggles confer their intensify behaviors.

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25
Q

Classical psychological behavioral model of disruptive behavior

A

Based on the following pattern of parental reaction to miss behavior: 1) the parent addresses miss behavior or makes in unpopular request 1)the child responds by arguing or counterattacking and 3)the parent withdraws from the conflict or gives into the Childs demands. Child does not learn to respect rules or authority. Alternate pattern sometimes occurs is a vicious cycle of harsh punitive parental responses to miss behavior resulting in defiance and disrespect on the part of the child and further coercive parental behaviors. Limited parental supervision, permissive parenting and avoidance of conflict, excessive attention for negative behavior, inconsistent disciplinary practicesand failure to teach prosocial skills or use positive management techniques can further exacerbate the stripped of behavior

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26
Q

Treatments of externalizing disorders

A

Interventions that address the family and social context of behaviors as well as deficits in psycho social skills can improve externalizing behaviors. Cognitive behavioral parent education teach parents to regulate their own emotions which increases positive interactions with children establishment of appropriate rolls and consistently improving child behavior and parental mental health. Psychosocial interventions focused on teaching youngsters assertiveness and anger management techniques and building skills and empathy communication and social relationships and problem-solving. Mobilize an adult mentors who demonstrate empathy warmth and acceptance is effective. Conduct disorder is particularly difficult to treat best before patterns of antisocial behavior or firmly established. Incarcerationproduce additional behavioral or psychological difficulties rather than rehabilitation especially if incarceration is an adult facilities

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27
Q

Elimination disorders

A

Enuresis and encopresismany exhibit significant distress and apprehension, sensitivity to real or imagined parental disapproval, and with drawl from peer relationships. On sympathetic or impatient responses from caregivers can increase anxiety and distress making the problem worse

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28
Q

Enuresis

A

Periodic forwarding of urine into one’s clothes or bad or onto the floor during the day or night. Usually involuntary although in rare situations may be intentional. Most likely to occur during sleep bedwetting. Child must be at least five and void inappropriately at least twice per week for three months. Temper cent of five-year-olds and 5% of 10-year-olds. 1% of adolescents. Psychological and biological factors are associated. Life situation such as death of parent birth of a new sibling family dysfunction or the presence of other emotional problems can increase risk. Severe nocturnal enuresis is usually due to heredity. Biological influence maybe genetics delays and physical maturation and I hypersensitive or smallletter. Provided you Acacian and support for parents and child set up rewards and use a bedtime your own alarm. Medication can also be used

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29
Q

Encopresis

A

Defecating onto ones close the floor or other inappropriate places child must be at least four years old and must have defecated inappropriately at least once a month for three months. .7 to 4. For prevalence in children. Intermittent episodes can last for years usually pattern is a child with constipation resulting in painful defecation and subsequent withholding of bowel emptying leads to National constipation fickle leakage an involuntary swelling. Inattention and hyperactivity or common. Treatment includes proper medical valuation increasing fluid intake and and parent and child education about toileting regimes

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30
Q

Neurodevelopmental disorder’s

A

Conditions involving impaired development of the brain and central nervous system that are evident early in the child’s life symptoms include difficulty with learning communication and behavior becoming increasingly evident as child grows and develops includes tip disorders attention deficit hyper activity disorder autism spectrum disorder’s and intellectual and learning disorders

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31
Q

Tick

A

And involuntary repetitive movement or vocalization. Motor tics involve various physical behaviors including blinking grimacing tapping jerking the head flaring the nostrils and contracting the shoulders. Vocal tics include coughing grunting throat clearing sniffling and son repetitive stereotyped outbursts of words.

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32
Q

Tick disorder

A

If the tech is present for less than a year diagnosis of provisional to disorders given chronic motor or VocalTec disorder refers to text lasting more than a year. Texas usually peak prior to puberty in the Kleiner disappear through adolescence. Neurological studies suggest symptom improvement maybe due to compensatory neuroplastic brain we organization that allows affect teens to suppress and eventually controlticks. Short term suppression of a tip is sometimes possible but results the subsequent increases in the tech. Many people report feeling tension before I took followed by a sense of relief after.2 to 5% four times as common in males. Have a genetic basis. Prenatal factors associated include alcohol and cannabis use in adequate maternal weight gain. You psychotherapies or habit reversal or antipsychotic medications for severe text

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33
Q

Tourette’s disorder TD

A

Characterized by multiple motor tics and one or more vocal tics. Motor tics involving self harm such as punching oneself or coprolalia occur and 10% of those with Tourette’s disorder. Tics are present for at least one year although not necessarily concurrently. motor tics include blinking grimacing shrugging jerking of the header shoulders. vocal tics such as repetitive throat clearing sniffling or grunting..77% had Tourette’s much more common in boys. has a genetic basis. Prenatal factors include maternal alcohol and cannabis use an in adequate maternal weight gain. TD is highly comorbid with obsessive-compulsive disorder therefore similar neurochemical abnormalities in brain structures are likely involved. Stress, negative social interactions, anxiety, excitement or exhaustion can increase the frequency of ticks. Psychotherapy can help as well as behavioral techniques such as habit reversal. Sometimes antipsychotic medications are used.

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34
Q

Habit reversal

A

A therapeutic technique in which a Clynus talk to substitute new behaviors for habitual behavior such as the tic

35
Q

Corprolalia

A

Involuntary utterance of obscenities or inappropriate remarks like her and 10% of those with Tourette’s disorder. Comorbid with poor and her control attention deficit hyper activity disorder obsessive-compulsive disorder impulse of behavior and poor social skills often interfere with quality of life even more than the tix

36
Q

Attention deficit hyper activity disorder

A

Childhood onset disorder characterized by persistent attentional problems and or impulsive and hyperactive behaviors there atypical for the child’s age and developmental level. Symptoms begin before 12 persistently six months and interfere with social and academic functioning. They can have problems involving inattention, hyperactivity and impulsivity, or a combination. Most frequently diagnosed disorder in preschool and school-age children. 11% of children between 417 Eversave this diagnosis. Diagnoses have increased by 5% per year over the last decade boys 13% more than twice as likely as girls at 5.6% to receive ADHD.30% experience continued symptoms of inattention disorganize Asian or impulsive action in adulthood

37
Q

Symptoms of ADHD and difficult situations

A

Children have most difficulty in situations that are unstructured or involve insufficient stimulation or tedious activities that require sustained attention. Peer relationships are challenging. Two thirds of those with ADHD had other mental health conditions including conduct disorder oppositional defiance disorder anxiety or depression or learning disabilities or other neurodevelopmental disorder’s.

38
Q

Inattention symptoms

A

Careless mistakes, poor attention to detail, difficulty sustaining attention, does not appear to be listening, failure to follow instructions, difficulty organizing tasks, avoidance of sustained mental effort, missed placing of important objects, distractibility and forgetfulness.

39
Q

Hyperactivity and impulsivity symptoms

A

Fidgeting, restlessness, excessive movement, excessive loudness, excessive talking, blurting out answers, difficulty waiting for a turn, interruption of orange version on others, and impatience. A diagnosis requires the presence of at least six characteristics involving in attention or hyperactive impulsivity. The characteristics must be evident before age 12 be present in at least to settings persist for at least six months and interfere with social academic or work functioning.

40
Q

Biological dimension of ADHD

A

80% of symptoms explainable by genetic factors. Involving multiple genes each with smaller Fox and subsequent Jean asked environment interactions. 1st° relative’s have increased risk of both bipolar and schizophrenia.

41
Q

Neurological mechanisms of ADHD

A

1)Functional abnormalitiesin frontal brain regions associated with executive functions attention and inhibition of responses. Reduce inhibitory mechanisms in the prefrontal cortex can affect impulsivity organization and attentional processes. Frontal lobe abnormalities involving networks associated with sustained attention appear to persist into adulthood even one symptoms subside. 2) Brain structure in circuitry irregularities in regions such as the frontal cortex cerebellum and parietal lobe’s. Smaller frontal lobe’s in children with ADHD. Reduce brain connectivity in regions associated with the tensional skills and goal directed actions. Some show slower development of the cerebrum particularly pre-frontal region is associated with the tension and motor planning. May explain why some children outgrow disorder 3)reductions and neurotransmitter such as dopamine in GABA that affect signal flow to and from frontal lobe’s associate it with difficulty inhibiting behavioral impulses. Many medications used to treat ADHD target these neurotransmitters

42
Q

Other biological factors implicated in ADHD

A

Prematurity, perinatal oxygen deprivation, very low birth weight, exposure to lead and PCB, viral infections, meningitis, and encephalitis. Maternal smoking or drugs or alcohol use during pregnancy. Food additives can contribute but do not cause

43
Q

Psychological social and social cultural dimensions of ADHD

A

Family stress, severe marital discord, poverty, family conflicts, maternal psychopathology, parental criminality, maternal mental disorder, and foster care placement have all been associated with ADHD. Negative reactions from parents and peers causes more interpersonal conflict and can result in psychological reactions such as depression and low self-esteem and rebelliousness and lack of opportunities to socialize with peersfactors that further exacerbate symptoms. Different cultural and regional expectations, can explain regional differences and ADHD which is prevalent and southern states and some Midwestern states and lowest and Western states. Encouraging exercise and outdoor activities help prevent children from getting over rows and reduce symptoms

44
Q

BiologicalTreatment of ADHD

A

Stimulants such as methylphenidate Ridellan. 30% do not improve. 69% of children take medication. Stimulants work by normalizing neurotransmitter functioning and increasing neurological activation in the frontal cortex thereby increasing attention and reducing impulsivity.

45
Q

Behavioral and psychosocial treatment for ADHD

A

Parent education classroom management strategies behavioral rewards or self-management are highly effective in producing both short term and long term reductions and symptoms. Parent behavior training teaching parents to use affect of disciplinary practices should be used before considering medication especially with preschool children. Modify the environment to allow movement or ensuring the school work is challenging.provide opportunities for moderate exercise can reduce impulsivity and improve academic performance. One and five receives no treatments

46
Q

Hi income families and ADHD

A

Children from high and come families have an increased risk of ADHD diagnosis. Percentage of children diagnosed his highest in southern states a 12.6% and lowest and Western states 8.1%

47
Q

Autism spectrum disorder AST

A

Characterized by significant impairment in social communication skills and a display of stereotyped interest and behaviors. ST is designated to spectrum disorder because the symptoms vary significantly, occurring along the continuum from mild to severe, and affecting each person in different ways. One out of 68 children have this. Has increased at 123% between 2002 and 2010. Five times more frequently and boys compared to girls. Complex symptoms

48
Q

Leo Kanner

A

A child psychologist who in 1943 identified a triad of behaviors that of come to define essential features of ASD: extreme isolation and inability to relate to people, I need for sameness, and significant difficulties with communication. Kanner called the syndrome infantile autism from the Greek autos for self to reflect the profound aloneness and detachment of these children. At its core ASD involves pervasive deficits in social communication.

49
Q

Deficits in social communication and social interaction with ASD

A

Persistence of1) atypical social emotional reciprocity. Interesting social interaction maybe limited or totally lacking. Milder symptoms are one-sided communication focused on their own interests or the failure to understand the back-and-forth of typical conversations. 2) atypical nonverbal communication. Little or no eye contact and absence of meaningful just yours or facial expressions. Mild symptoms may include unusual nonverbal communication such as pushing people side as if they were objects, or poor social boundaries involving interest behaviors or awkward interactions3) difficulties developing and maintaining relationships there may be a lack of interest in others or failure to recognize other peoples identity or emotions including treating people as objects or feeling to see contact from caregivers milder symptoms may have no interest in imaginative play be socially inapt or have difficulty adjusting their behavior to the social context.

50
Q

Repetitive behavior or restricted interests or activities involving at least two of the following

A

1)Repetitive speech movement or use of objects including banning the head flap in the arms rocking the body spinning object swirling in circles or rhythmically moving fingers sometimes stacking are spinning objects to move them from side to side maybe echolalia or echoing what is heard incessant repetition of sounds words phrases or nonsensical word combinations are one-sided conversations. 2) intense focus on rituals or routines and strong resistance to change. 3) intense fix stations or restricted interests4) atypical sensory reactivity such as lack of reactivity to pain heat or cold or over reactivity to sensory input or unusual focus on sensory aspects of objects

51
Q

Diagnosis of autism spectrum disorder happens when a trained professional documents persistent evidence of both deficits in social communication and social interaction, and repetitive behavior or restrictive interest or activities in involving at least two of the following

A

both 1deficits in social communication and social interaction, and 2repetitive behavior or restrictive interest or activities in involving at least two of the following

52
Q

Levels of severity in ASD

A

One half have average or above average cognitive skills and are considered high functioning, one third have a significant cognitive impairment. Some with a low intellectual functioning display splinter skills. These children are referred to as autistic Savant level of impairment varies from severe to mild

53
Q

Splinter skills

A

Some autistic children with low intellectual functioning do well on isolated tasks such as drawing puzzle construction musical ability or wrote memory but perform poorly on tasks requiring language skills and some Bolick thinking these children are referred to as autistic Savant’s

54
Q

Recent eye tracking technology

A

Candy tech decreases and I contact between two and 24 months of age I’m on some infants a higher risk of ASD in contrast to normally developing children who showed progressive increases and I contact those later diagnosed with ASD show progressive declines and I gaze with most rapid the clients occurring among those with develop more severe symptoms. These differences were evident as early as two months of age and generate optimism about earlier diagnoses. Earlier diagnoses Mayhall or slow down the cascade of events that begin early in life.

55
Q

Diagnosis of ASD is often delayed

A

In some children there is a period of apparently normal social and intellectual development before AST symptoms appear with deterioration of skills beginning around 12 months of age or even later. Children with this pattern of regression referred to as regressive autism often develop more severe symptoms compared to autistic children without this pattern

56
Q

Biological factors of ASD

A

Biological factors play the most critical role. Concordance rates are much higher for monozygotic twins then dizygotic twins with heritability of AST about .73% for males and .87% for females. Much higher prevalence of ASD up to 19% a bunk siblings of individuals with ASD compared to the rest of the population. Autistic traits are highly heritable. Some siblings who do not develop ASD show atypical social development and communication patterns. Psychological theories pointing to deviant parent child interaction is as the cars have been proven wrong autism is Nate. Biological factors are the primary cause

57
Q

Neurological findings of research of ASD

A

Genetic factors involving multiple brain regions including cerebellum and frontal and temporal lobe’s. Unique pattern of metabolic brain activity, reduced gays towards the eye regions of faces combine with elevated activity in the middle lot in response to human faces, abnormally high levels of serotonin especially in males with AST and those who are high functioning, hyperconnectivity throughout The brain, accelerated growth of amygdala in early childhood appears long before autistic symptoms appear in regressive autism around 4 to 6 months of age. Accelerated had growth may be an Endo phenotype. Males later diagnosed with ASD exhibited a pattern of rapid her grow from 6 to 9 months after birth

58
Q

Extra head growth in ASD

A

. Of accelerated head growth in infancy and multiple regions of the brain proceeds and overlaps with the onset of autistic symptoms. Conversely the increasingly severe autistic symptoms display by children with ASD in the second year of life correspond with the subsequent. Of the Excelerator growth within the brain. Some boys later diagnosed with ASD showed overall growth acceleration in the first year of lifeincreased stature was associated with more severe symptoms of AST and increased likelihood of seizures

59
Q

Mitochondrial dysfunction and ASD

A

Mitochondrial dysfunction affects the energy producing capacity of cells , A process critically important to neural development. Some biomarkers of mitochondrial dysfunction correlate with severity of autism symptoms in fact some children with ASD appear to have genetically based mitochondrial disease.

60
Q

Study involving analysis of postmortem brains of children with ASD

A

Evidence of patchy areas of disrupted and Ronald development occurred during normal cellaring process and all six layers of cortex. Abnormalities most prevalent in early developing layers of frontal and temporal cortex areas associated with social emotional communication skills. This brand we abnormalities begins her in pregnancy when the brain is forming. Because it’s patchy may explain why some children recover

61
Q

Genetic mutations associated with ASD

A

These mutations coming to play in specific brain regions during fetal development between 10 and 24 weeks after conception these genes influence cell development in brain regions associated with symptoms of a STD. Twins with ASD show environmentally driven epigenetic alterations in comparison to the other twin without it.

62
Q

Environmental factors contributing to the development of ASD

A

Environmental toxins associated with the development of ASD include exposure to air pollutants such as lead and mercury and other heavy metals, certain pesticides, maternal smoking, poor indoor ventilation and PVC flooring. Children with ASD metabolize lead and mercury differently. Could it possibly explain the geographic difference?

63
Q

Other factors associated with ASD

A

Nutritional deficiencies, immune system dysregulation, prematurity, and closely spaced pregnancies,. Biological mechanisms may also account for the fact that artistic sometimes sometimes improve and then I’ll probably return when a child with ASD has a fever.

64
Q

Intervention and treatment of ASD

A

Most diagnosed retain their diagnosis and require support throughout their lifetime. Some with milder symptoms may be self-sufficient and sufficiently employed and function reasonably well in adulthood all those social awkwardness restrictive interests or a typical behaviors often persist. Higher cognitive adaptive functioning fare better than those who have a intellectual disability and severe autistic symptoms. Some children have made remarkablerecovery after receiving intensive intervention early especially children with higher cognitive and language skills. Comorbid conditions such as depression anxiety inattention and hyperactivity often remain after ASD symptoms have remitted.

65
Q

Children born less than one year after the birth of a sibling

A

Are 300% more likely to develop ASD compared to children born at least four years after a sibling

66
Q

Medical treatment of ASD

A

Only two medications the antipsychotics risperidone and aripiprazole,.have received FDA approval for treatment of symptoms of aggression and irritability associated with ASD. Some studies have found that administration of Oxytocin , A naturally occurring hormone that affect social bonding, can increase Eye gaze and emotional recognition skills in children and adolescents with mild ASD. Oxytocin increases neural activity in the amygdala and in social circuits of the brain there by facilitating social attunement. Oxytocin is the only biological intervention to address a core symptom of ASD social communication rather than the behavioral challenges.

67
Q

Specialized program for children with ASD often include

A

High degree of structure through predictive routine, visual activity schedules, and clear physical boundaries to minimize distractions; intensive systematically plan developmentally appropriate educational activities; behavior modification procedures to eliminate echolalia and repetitive behaviors and increase attending behaviors verbalizations and social play; parent education regarding behavior management and enhancing communication; and opportunities to practice learned skills in new environments including interactions with typically developing peers. Social communication reinforcement of appropriate responses and prevention of repetitive behaviors produce the most significant gains.important for children with ASD to have social learning such as interactions with normal peers in normal social contacts.

68
Q

Pivotal response treatment PRT

A

I’ll play bass child initiated therapeutic approach focuses on reducing self stimulating behaviors and developing communication and social skills by targeting pivotal behaviors such as motivation responding to social cues and initiating social reaction. Use the items of interest of the child is natural reinforcers rather than contrived awards.

69
Q

Intellectual disability

A

A disorder characterized by limitations in intellectual functioning and adaptive behaviors formally referred to as mental retardation is characterized by significant limitations intellectual functioning an adaptive behaviors. including a significantly below average intellectual functioning interpreted as an IQ score of 70 or less ;and deficiencies in adaptive behavior such as self-care, understanding health and safety issues, ability to live ,work or plan leisure activities and use community resources greater than would be expected based on age or cultural back around. ID is diagnosed only one low intelligence is accompanied by impaired adaptive functioning. Less than 1% of students in public schools in the US have an intellectual disorder. Lower to middle income countries have double the prevalence. Many with ID have consistent condition such as ASD. One fourth have seizure disorder

70
Q

For distinct categories of intellectual disability based on IQ scores

A

Mild IQ score 50 to 55 up to 70, moderate is IQ score from 35 to 55 severe 20 to 40 and profound IQ score below 20 to 25

71
Q

Adaptive behavior

A

Performance on tasks of daily living including academic skills, self-care, and the ability to work or live independently

72
Q

Causes of intellectual disability

A

Mild intellectual disability is often idiopathic having no known cause more pronounced intellectual the disability is often associated with genetic factors brain abnormalities or brain injuries. Most common perinatal birth conditions associated with ID are prematurity and low birth weight. During post natal. Factors such as head injuries, brain infections, tumors and prolong malnutrition can cause brain damage and consequent ID. Exposure to toxins including lead is associated with both ID and hyperactivity

73
Q

Genetic factors of ID

A

In 80% of cases of ID underlying cause unknown. Genetic factors have not yet been identified which may be responsible. Higher prevalence of ID in males. 80% of the 40 jeans that have been identified as contributing to ID are in fact on the X chromosome. Genetic factors that exert in an influence on ID include both genetic variations and genetic abnormalities. Some simply fall on the lower end of the normal range of intelligence of 72 130 IQ with no specific physiological abnormalities. Genetic abnormalities associated with ID include chromosomal variations as well as conditions resulting from inheritance of a single gene.although some genetic abnormalities can result in varying degrees of ID many individuals with genetically based ID have significant impairment the most common inherited form of ID is fragile X syndrome. Down syndrome is the second most common

74
Q

Fragile X syndrome

A

And inherited condition involving limited production of proteins required for brain development resulting in mild to severe intellectual disability. Males with this usually have more impairment. Autistic behavior and hyperactivity occur in some individuals with fragile X

75
Q

Down syndrome DS

A

The most common and most easily recognize chromosomal disorder resulting in intellectual disability. And extra copy of chromosome 21 originates during gamete development involving either the eighth or the sperm; this extra call Mazzone produces physical and neurological characteristics associated with the condition. 1 in 691 births. Increases significantly with maternal age. One in 10 at age 49. Distinctive physical characteristics include a single kris across the palm of the hand, slanted eyes, a protruding tongue, and a harsh voice. They have significantly increased incidence of leukemia and infectious disease is, hearing loss, congenital heart disease, and premature aging also have increased risk of early dementia including early onset Alzheimer’s disease

76
Q

Amniocentesis

A

A parental screening procedure involving with drawl of amniotic fluid from the feral sack. Perform between 14th and 18th week of pregnancy involve some risk and candy tacked down syndrome. Due to cultural differences in use of genetic screening and decisions regarding termination of pregnancy this has produced Hispanic mothers being most likely to give birth to a child with down syndrome

77
Q

Non genetic biological factors of ID

A

Prenatal viruses and infections such as tuberculosis or German measles, drugs and alcohol, radiation and poor nutrition can cause ID. Iron deficiency during pregnancy or during early infancy can impair her own till actual development. Phenyl ketonuria is an inherited condition affecting metabolism can you have prenatal postnatal affectsif pregnant women with PK you and just protein or artificial sweeteners result and build up of a substance called phenylalanine can cause significant intellectual impairment in the developing fetus. PKU in an infant can cross post natal brain damage

78
Q

Fetal alcohol spectrum affects

A

A continuum of detrimental neurological and behavioral effects resulting from maternal alcohol consumption during pregnancy. The greatest concern is for those children with fetal alcohol syndrome. Include reduce cognitive functioning, attentional difficulties, slower information processing, and poor working memory.

79
Q

Fetal alcohol syndrome FAS

A

A condition resulting from maternal alcohol consumption during just station that involves central nervous system dysfunction and altered brain development. Less than 1% of live births. Results and restricted growth, facial abnormalities, insignificant this function of the central nervous system and brain. Both fAS and spectrum have difficulty with attention learning memory regulation of emotions and executive functioning all associated with frontal lobe of brain. Market delay development in adaptive behavior particularly skills of daily living is common

80
Q

Neural behavioral disorder associated with prenatal alcohol exposure

A

DSM five propose diagnosis undergoing study. Children with FAS and those with fetal alcohol spectrum facts. This encompasses impairment in neurocognitive behavioral and adaptive functioning associated with prenatal alcohol exposure. 2 to 5%

81
Q

Learning disorders

A

And academic disability characterized by reading writing or math skills that are substantially below levels that would be expected based on the persons age, intellectual ability, and educational background. Specific ones include dyslexia dyscalculia. 5% of students in public schools in the US have LTE twice is off an invoice. Metairie have Coke current disorder such as ADHD specially boys. Little is known about the precise causes. Some children have slower me brain maturation and eventually catch up others have lifelong differences in neurological processing ofinformation. Possibilities may include many of the same biological explanations for ID and 80 HD. LD tends to run in families suggesting a genetic component

82
Q

Dyslexia

A

Condition involving difficulties with reading skills either with accuracy or fluency and reading.

83
Q

Dyscalculia

A

Significant difficulties and understanding mathematical skills or concepts quantities number symbols or Basic arithmetic equations