Chap 5&6 Flashcards
Anxiety disorder
Unfounded fear or anxiety that interferes with day-to-day functioning and produces clinically significant distress or life impairment
Fear circuitry in the brain
Amygdala triggers state of fear,starts the hypothalamus pituitary adrenal axis. Slower pathway hippo campus and pre-frontal cortex evaluate danger and can stop the fear response
Primary fear circuit
Amygdala triggers the hypothalamus pituitary adrenal axis to prepare for immediate action fight or flight
Secondary fear circuit
Stimulus simultaneously activates hippo campus and prefrontal cortex which processes the sensory input and evaluates danger associated with the situation if no threat, overrides initial fear response
Amygdala2
Structure associated with processing expression and memory of emotions especially anger and fear
Hippo campus
Part of brain involved in forming organizing and storing memories
GABA
Gamma amino butyric acid an inhibitory neurotransmitter involved in inducing sleep and relaxation. Reduction in GABA receptors in hippo campus and amygdala link to anxiety and fear.
Serotonin to
A neurotransmitter associated with mood sleep and appetite and impulsive behavior.
Allele
The gene pair responsible for a specific trait
Behavioral inhibition
Shyness
Negative appraisal
Interpreting events as threatening, even ambiguous ones
Anxiety sensitivity
Trait involving Fear of physiological changes within the body.
Reappraisal
Minimizing negative responses by looking at the situation from various perspectives. Fewer anxiety symptoms
Phobia
A strong persistent and unwarranted fear of a specific object or situation
Social anxiety disorder
Social phobiainvolves an intense fear of being scrutinized in social or performance situations.fear of doing something embarrassing or humiliating in the presence of others, is out of proportion to the circumstances, and results in avoidance of the situation or intense fear or anxiety when in during the situation. Self-conscious 8.7 % adults in year.
Performance only type of social anxiety disorder
Only in situations where must speak or perform in public
Specific phobia
Extreme fear of a specific object or situation, exposure to stimulus nearly always produces intense panic or anxiety out of proportion to danger. 8.7% adults in Year.
Primary types of specific phobias
Animal or living creatures, natural disasters, blood injections or injury, situational factors or environment. 8.7% of pop.
Agoura phobia
Intense fear of at least two of the following: a being outside the home alone b)traveling via public transportation c)being an open spaces or D)being in stores or theater or E)standing in line or being in a crowd feared because help may not be readily available. Often have anxiety sensitivity. 1% of adults. Can be late onset 11% 65. 31% heritability
Panic attack
Episode of extreme fear accompanied by physiological symptoms. Can cause Agoura phobia
Heritability of phobias
All phobias you have a 31% genetic component
Biological component of phobias
Increased responsiveness of the amygdala and other areas of brain associated with fear. Neural imaging shows phobias have increased physiological responses in reaction to phobia stimuli
Preparedness
Alternate biological view of fear reactions. Fears do not develop randomly. Easier for humans to develop veers to which they are physiologically predispose such as fear of heights or snakes. May have been necessary for human survival,can appear without exposure to conditioning experiences.
Psychological dimension of phobias
Classical conditioning perspective, observational learning perspective, negative information perspective, cognitive behavioral perspective
Classical conditioning perspective
Phobias are conditioned fear responses evolved from psychologist John Watson experiment with little Albert. Childhood fears can be retriggered in adulthood if we are faced with sound smells or events that bring up memory of those fears
Observational learning perspective
Watch the video in which a man received uncomfortable shock response to stimulus. After viewing the video shown the stimulus associated with the shock respondents reacted with fear. Fear response document in neural imaging scans of amygdala. In children whose parents showed anxiety before spelling bee,reported higher anxiety levels more anxious thoughts and greater avoidance of spelling test done than in those with relaxed parent.
Negative information perspective
Parents giving negative description of strange animal caused the children to react with more fear than those who receive positive or ambiguous information
Cognitive behavioral perspective
Catastrophic thoughts and cognitive distortion’s including overestimating the threat may cause strong fears to develop thinking a spider will take attack or take revenge increased phobias
Social dimension of phobias
Over protection of socially withdrawn children and lack of support for independence can increase sense of insecurity. Children prevented from developing emotional regulation and coping skills. Negative family interactions at age 3 and family stress in middle childhood both associated with social anxiety symptoms. Punitive maternal parenting style linked with increased tendency to have fearful believes. Victimization by peers in childhood can increase social anxiety
Benzodiazepines
Increase activity of neurotransmitter GABA. They can produce dependence, withdrawal symptoms and paradoxical reaction such as increased talkativeness excessive movement and even hostility and rage and dementia an older adults
SSRI for anxiety
Begin to alter brain chemistry after first dose but require 4 to 6 weeks before they reduce symptoms
Beta blockers
Propranolol or Inderal can reduce physical symptoms of anxiety disorders especially social phobia. These treat high blood pressure and heart conditions
D cycloserine
Drug to treat tuberculosis sometimes used in combination with psychotherapy. Appears to affect brain regions associated with the unlearning of fear. If it doesn’t work it may actually reconsolidate fear and strengthen it
Cognitive behavioral treatments of phobias
Exposure therapy systemic desensitization cognitive restructuring like changing irrational or anxiety arousing thoughts, and modeling therapy
Exposure therapy
Treatment involves gradual increasingly difficult and counters with the feared situation
Apply tension
For individuals who show the physiological pattern of a sudden drop in blood pressure like in blood injury fear by tightening the muscles repeat 5 times can prevent fainting the fear becomes extinguished
Systemic desensitization
Uses muscle relaxation to reduce anxiety associated with phobias While being exposed to item feared while in relaxed state.
Cognitive restructuring
Unrealistic thoughts believed to be responsible for phobias are altered.normalize social anxiety by encouraging to interpret emotional and physical tension as normal anxiety and redirect attention away from themselves in social situations
Modeling therapy
Individual with phobia observes the model of a person coping with our responding appropriately to fear producing situation.
Panic disorder 2.7% per yr.
Recurrent unexpected panic attacks in combination with a)apprehension over having another attack or worry about the consequences of an attack or B)changes in behavior or activities designed to avoid another panic attack. must be present for one month or more 11.2% have them in a year. More attacks with comorbid depression, GAD or substance abuse.
Concordance rates concordance rates
Percentage of relative sharing the same disorder.is higher. Heritability is estimated to be 32% in panic disorder.
Brain in neurochemistry of panic disorder
Brain structures such as amygdala are involved in anxiety disorder, incl panic disorder. Neuroimaging Fewer serotonin receptors results in decreased availability of Serotonin
Psychological demension a panic disorder
Heightened anxiety sensitivity and heightened fear responses to bodily sensations
Cognitive behavioral perspective of panic disorder
Physiological change such as faster breathing increased heart rate occurs; then catastrophic thought development ,thoughts result in increased fear, resulting in more physiological changes. circular pattern develops. Interoceptive conditioning. Has research basis
Interoceptive conditioning
Classical conditioning process in which fear is associated with the perception of bodily changes. Pairing of bodily changes with fear. As this assoc. strengthens, somatic changes can automatically cause panic attack.
Social and social cultural dimensions to panic disorder
Stressful childhood involving separation anxiety, family conflicts, school problems or loss of a loved one and bullying.
Biological treatment of panic disorder
Benzodiazepines and antidepressants also beta blockers to reduce symptoms such as sweating heart palpitations and dizziness. High relapse rates after cessation of drugs therapy.
CBT therapy for panic disorder
Teaching coping statements such as the feeling is not pleasant but I can handle it, help the client identify the antecedents of the panic like what stress am I facing, teaching the clients to self-induce physiological symptoms of panic to extinguish the Interoceptive conditioning that has occurred, correcting catastrophic thinking, and encouraging the client the face symptoms both within the session and the outside world, educated the client about misconceptions regarding the symptoms of panic disorder
Prefrontal cortex
Region of the cortex responsible for executive functioning allows us to manage our attention behavior and emotions
Role ofmedication and anxiety disorders
Directly decrease activity in the amygdala and thus normalize anxiety reactions.