Chapter 12 Flashcards

1
Q

Cognitive communication disorder

A

Difficulty with any aspect of communication that is affected by disruption of congition

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2
Q

What are some difficulties that people have with a cognitive communication disorder?

A

Attention, memory, organization, problem solving, reasoning, executive functions

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3
Q

Can a cognitive communication disorder effect both verbal and non verbal communication?

A

Yes (speaking, listening, reading, writing, social interaction)

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4
Q

Why was there a sudden burst in neurogenic communication disorders?

A

WW2 many soldiers survived TBI’s so there was a burst in these disorders and a need to provide rehabilitation

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5
Q

What are the 3 types of disorders that neurogenic communication disorders affect?

A

Language, motor speech, and cognition

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6
Q

Acquired disorder (adult)

A

Had typical speech, language, communication before neurological incident

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7
Q

Developmental disorder (child)

A

Difficulty developing language, speech, or cognitive skills for communication

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8
Q

What is a cerebrovascular accident?

A

stroke (acquired)

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9
Q

What are the 3 types of strokes?

A

Thrombotic, embolic, hemorrhagic

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10
Q

Thrombotic

A

Clot from gradual accumulation of plaque in the brain

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11
Q

Embolic

A

Clot that moved from another part of the body

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12
Q

Hemorrhagic

A

Bleeding in the brain (an aneurysm/artery burst and there is blood in the brain)

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13
Q

What is the perisylvian fissure?

A

Area on the lateral left hemisphere near the sylvian fissure/lateral suclus

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14
Q

What may damage to the sylvian fissure result in?

A

Aphasia

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15
Q

What are some secondary results of CVA?

A

Edema or swelling in the brain, infarct or tissue death in the area of the stroke, or spontaneous recovery which is when the brain reorganizes and takes over the function that was lost in different areas

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16
Q

Aphasia

A

Disorder of language (receptive & expressive)

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17
Q

Receptive

A

Difficulty understanding language (listening & reading)

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18
Q

Expressive

A

Difficulty using language (talking & writing)

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19
Q

Heschel’s gyrus

A

Primary auditory cortex responsible for perception and discrimination of the auditory stimuli

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20
Q

Wernicke’s area

A

Responsible for sematics, storage/retrieval of mental representations of words, word meanings, grammar/linguistic rules (attaching meaning to language/understanding)

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21
Q

Arculate Fasciculus

A

White matter pathway connecting wernickes area to brocas area

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22
Q

Broca’s area

A

helps with grammar, responsible for planning/organizing speech movements, and articulation (production of speech)

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23
Q

What are the 4 lobes?

A

Frontal, parietal, occipital, temporal

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24
Q

What lobe is broacs in?

A

Frontal

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25
Q

What lobe is wernickes in?

A

Temporal

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26
Q

What is the hearing/auditory comprehension pathway?

A

Ears - auditory pathway - CN 8 - temporal lobe - Heschl’s gyrus - wernickes area

27
Q

What is the speaking pathway?

A

Wernickes - arculate fasiculus - broca’s area - primary motor cortex - brainstem - cranial nerves

28
Q

Why do we test repetition for aphasia?

A

Can the word get in & can the word get out (listening & producing)

29
Q

Brocas Aphasia

A

Fluency = impaired, auditory comprehension = good, repetition = impaired, naming = impaired

30
Q

Wernickes Aphasia

A

Fluency = good, auditory comprehension = impaired, repetition = impaired, naming = impaired

31
Q

Global Aphasia

A

Fluency, auditory comprehension, repetition, naming = impaired

32
Q

What is fluency?

A

Effortless speech, normal rate, grammar okay, syntax okay, intonation okay, uses all types of words

33
Q

What is non-fluency?

A

Effortful speech, slow rate (under 100 wpm), grammar reduced, syntax poor, monotone, leave out function words and rely on nouns

34
Q

Broca’s aphasia example

A

agrammatic speech (lots of nouns), loss of function words, nouns and verbs, misarticulating (dysarthria), frustration and awareness

35
Q

Wernicke’s aphasia example

A

verbal paraphasias (fork for spoon), right category but wrong word, neologisms (new words/jargon), get overall just of convo but could not understand specifics, could not follow instructions or comprehend reading

36
Q

Global aphasia example

A

stereotypical utterances, over learned phrases, single words, speech limited, can hardly respond to yes/no questions

37
Q

Does a person with slow laborious speech, grammatical, and phonemic errors have slow and laborious writing with grammatical errors as well?

38
Q

Motor speech disorders

A

Disorders of speech resulting from neurologic impairment affecting the motor programming and/or neuromuscular execution of speech (apraxia and dysarthria)

39
Q

Apraxia

A

Disorder of motor programming for speech (planning, not caused by paralysis, weakness, incoordination, or sensory. loss, damage to CNS, oral groping, inconsident, write fine

40
Q

Dysarthria

A

A group of speech disorders resulting from disturbances in muscle control of speech mechanism (neuromuscular execution), damage to CNS/PNS, speech problems result from paralysis, weakness, incoordination, sensory loss (drunk slurred speech), execution problem

41
Q

Problem with cognitive linguistic process (throughs translated to language)

A

Aphasia, right hemisphere disorder, cognitive communicative disorders in TBI

42
Q

Problem with motor speech planning/programming (language translated into motor movements)

A

Apraxia of speech

43
Q

Neuromuscular execution of motor speech (motor movement carried out by CNS and PNS)

A

Dysarthria

44
Q

Damage to wernickes

45
Q

Damaga to Brocas

A

Aphasia and/or Apraxia

46
Q

Damage to arcuate fasciculus

47
Q

Damage to motor strip

A

Dysarthria

48
Q

Damage to cranial nerves

A

Dysarthria

49
Q

Dementia

A

Cognitive Communication disorder

50
Q

Alzheimers

51
Q

Frontal lobe

A

Decision making, personality

52
Q

Primary progressive aphasia

53
Q

Cognition

A

Language, memory, attention, decision making

54
Q

TBI risk factors (more likely to get it if..)

A

Alcohol and drugs, poor school adjustment, socioeconomic status, personality type, pervious history of TBI, participation in sports

55
Q

First injury

A

2 seconds direct effect of the injury where the brain is accelerated, decelerated, or rotated

56
Q

Can first injuries occur twice?

A

Yes, hit head on windshield and then flung out of the car

57
Q

Second injury

A

Occurs in first few hours post onset, it is the physiological response to injury (could include oxygen deprivation to brain, blood loss, hematomas, increased inter cranial pressure, decreased blood pressure, swelling

58
Q

Open head injury

A

The skull is open/cracked, brain is exposed (gunshot)

59
Q

Closed head injury

A

Skull is closed/intact (falls)

60
Q

Mild brain injury

A

Brief or no loss of consciousness, the CT and MRI is negative (looks normal), symptoms could be vomiting, dizziness, lack of recall

61
Q

Moderate brain injury

A

Coma less than 24 hours, could see things on a CT or MRI, could be hemorrhage (bleeding) or skull fractures (tissue damage)

62
Q

Severe brain injury

A

Comas is greater than 24 hours

63
Q

Cognitive deficits following TBI

A

focusing problems, delayed response time, organization, difficulty multitasking, poor judgement, bad spacial functions, etc

64
Q

Language Cognitive deficits following TBI

A

Word finding is hard, prosody is flat, topic selection, maintenance, turn-taking, and consciousness are off