Cardiology ECG's Flashcards
What is an ECG?
Recording of electrical activity of the heart
How is an ECG recorded?
Attach electrode or leads to pt. Add conducting agents then hook to ECG machine.
Conducting agents
Alcohol
Ultrasound gel
Limb leads
Lead 1
Lead 2: most common
Lead 3
All make up “Einthoven’s triangle”
Augmented limb leads
- aVR
- aVL
- aVF
Patient placement for ECG:
Pt in R lateral recumbency (if possible) on Table on blanket or padded table
Pt breathing normal: avoid interference/artifact
Interference/artifact causes:
Pt movement: panting,trembling, etc
60-cycle: electrical interference
Lead placement on patient:
Right Axillary: White Lead Right Inguinal: Green Lead Left Axillary: Black Lead Left Inguinal: Red Lead \+/- Chest: Tan/brown Lead
P wave
Small, + deflection in lead 2.
SA node has fired and atria has depolarized.
Diastolic phase
PR Interval
Period of time where AV node is causing a delay in conduction.
QRS Complex
Q = -, R = +, S = - deflections.
Largest part.
Ventricular depolarization a.k.a contraction.
Systolic phase
T wave
+ or - deflection – uniform.
Should be 25% of the amp of the R wave.
Ventricular depolarization or relaxation.
ST segment
Period of time between ventricular contraction and relaxation.
RR Interval
Period of time between cardiac contractions.
What is an arrhythmia?
A disruption of normal electricity in the heart.
Causes of arrhythmia:
- Abnormal rate of rhythm
2. Abnormal site of origin
Intrinsic pacemaker rates:
- Dependent on size of animal/species
SA Node
70-120 bpm
Atrial muscle
0 bpm
AV Node
40-60 bpm
Bundle of HIS
40-60 bpm
Bundle branches
20-40 bpm
Purkinje fibers
20-40 bpm
Steps in evaluations: Step 1
Evaluate R-R Intervals – must be within a 10% variation.
Steps in evaluations: Step 2
Determine if impulses are sinus or not
Sinus
A,B,C
Normal conduction occurred = heart contracted normally
A. Evaluate P waves:
normal = small and positive deflection
Inverted P waves:
Impulse originates at the AV junction
Absent P waves:
Impulse originated somewhere at ventricles.
B. Look at relationship between P waves and QRS:
- Every P wave must have a QRS
- PR interval should be consistent
C. Evaluate T waves:
+ or - deflection– uniform. Should be 25% of the amp. of the R wave.
Steps in evaluations: Step 3
Look for anything unusual
How to obtain a heart rate:
- Ausculate
- Pulse – listen & palpate at the same time
- Palpate apex (apical apex)
- From an ECG
Normal Sinus Rhythm
Normal P, Q, R, S, T
Rate is normal for size of pt:
Cat=120-240 Dog=70-160
P waves are small and + R waves are tall and spiky. R to R are consistent
Sinus Bradycardia
Regular sinus rhythm with a heart rate below normal.
<120bpm – cat
Sinus Bradycardia:
Physiologic
Increased vegal tone (cranial nerve 10)
Lean, trim animals
Sinus Bradycardia:
Pathologic
Renal failure, increased levels of K+ (hyperkalemia) cardiac arrest
Sinus Bradycardia:
Drug induced
Phenoyl (Ace), Narcotics
Sinus Bradycardia:
Tx
Anti-colonergic drug – Glycopyrelate and Atropine
Sinus Tachycardia
Regular sinus rhythm with a increased rate
Dog- <240
Most common arrhythmia in practice
Sinus Tachycardia:
Physiologic
Pain, exercise, stress
SinusTachycardia:
Pathologic
Fever, shock, anemia, hyperthyroidism, CHF, hypoxia
Sinus Tachycardia:
Drugs
Anti-colonergics, epinephrine
Sinus Tachycardia:
Tx
Identify and control
Sinus Arrhythmia
Irregular sinus rhythm that originates in the SA node
Inconsistent R-R interval, alternating periods of a slow & fast heart rate associated with reap. Normal in dogs & horses, never normal in cats – pathology
Ventricular premature contractions (VPC)
Cardiac impulse originated in the ventricles rather than SA node
VPC Guidelines
Poor tissue perfusion d/t pulse deficits
Absent P waves
Wide & bizarre QRS complexes
+/- compensatory pause after VPC
VPC Causes
Cardiomyopathy Valve dz Feline hyperthyroidism Aorticstinosis Hypoxia (GDV)
VPC Signs
Exercise intolerance Weakness Syncopi (fainting) Pulse deficit, decreased perfusion Sudden death
VPC Tx
Anti-arrhythmic drugs (Litacane- K9, Procainamide- cat)
Bigeminy
One or two VPCs in a row
Consistent pattern of normal impulse and VPC
Ventricular Tachycardia
Three or more VPCs in a row
Ventricular Tachycardia Guidelines
Intermittent or sustained
Absent or buried in QRS complex- P wave
Fast heart rate <160 bpm
Ventricular Tachycardia Causes
Cardiomyopathy Valve dz Feline hyperthyroidism Aorticstinosis Hypoxia (GDV)
Ventricular Tachycardia Signs
Life threatening -> can convert to vfib
Ventricular Tachycardia Tx
Litacain - K9
Procainamide - feline
CRI -> constant rate infusion
Ventricular Fibrillation (vfib)
Ventricular depolarization without coordinated activity
Vfib Guidelines
No pulse = 0 cardiac output
Rapid, chaotic, irregular rhythm in ventricles
NO P, QRS
Vfib Causes
Severe systemic illness Shock Trauma Anesthetic reaction Severe hypothermia Untreated Vtach
Vfib Tx
Electrical cardio version 1st (dfib)
Anti-arrhythmic 2nd
Ventricular Asystole
Absence of pacemaker activity -> no depolarization= cardiac output = no pulse +/- escape beat - may see several
Ventricular Asystole Tx
CPR
ET-> 1. Intracardiac
2. Atropine
Atrial Fibrillation
Numerous disorganized atrial impulses that bombard the AV node
Atrial Fibrillation Guidelines
- Inconsistent R-R intervals
- Normal appearing QRS complex
- +/- Flutter P waves
Atrial Fibrillation Causes
Atrial enlargement
Dialated cardiomyopathy
Atrial Fibrillation Tx
Digoxin
Quinidine
Atrial Standstill
Absence of P waves with a regular escape rhythm, escape beat is slow (40-60 eb/min)
escape beat=no normal beats around
Atrial Standstill Guidelines
Slow rhythm >60 bpm
No P waves
Normal or wide appearing QRS
Atrial Standstill Causes
Atria distention
Cardiomyopathy
Hyperkalemia
Atrial Standstill Tx
Pacemaker (heart problem)
1st degree AV block
Delay in conduction through the AV node
1st degree AV block Guidelines
Prolonged P-R Intervals
1st degree AV block Causes
Geriatric pts
1st degree AV block Tx
Not needed
2nd degree AV block
Occasional P waves without corresponding QRS complexes
2nd degree AV block Guidelines
Regular occurring P waves
Occasional dropped QRS complexes
Common in horses d/t increased vegal tones
Not common in cats & dogs
2nd degree AV block Tx
Anti-colonergic
May be unnecessary
3rd degree AV block
“Atriaventrical dissociation”
Complete block
Cardiac impulse is completely blocked at AV node
3rd degree AV block Guidelines
Consistent P-P at normal rate
Consistent but slow escape rhythm (40-60)
3rd degree AV block Causes
VSD
Cardiomyopathy
3rd degree AV block Tx
Pacemaker