Approach to Neonate with Respiratory Distress Flashcards

1
Q

Manifestation of neonatal respiratory distress

A

Tachypnea

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2
Q

Neonatal tachypnea

A

> 60 breaths/min

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3
Q

T of F the presence of cyanosis necessarily means the newborn is respiratory distress

A

F

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4
Q

Cessation of breathing for 20 seconds

A

Apnea

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5
Q

Motor responses can be monitored and evaluated by measuring

A

EMG activity, intrathoracic pressure and lung function parameter

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6
Q

Sensors of thoracic expansion and alveolar respiration

A

J-receptors, irritant receptors and stretch reeptors

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7
Q

How can the baby conserve oxygen and energy

A

by not moving and not breathing

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8
Q

Best strategy to control hypoxemia in utero

A

APNEA

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9
Q

volume inspired above the tidal volume

A

Inspiratory reserve volume

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10
Q

Volume of a normal breath

A

TIDAL volume

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11
Q

Volume expired after expiration of tidal volume

A

ERV

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12
Q

volume that remains after the maximal expiration

A

Residual volume

FRC-ERV

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13
Q

volume expired after maximal inspiration

A

Vital capacity

TLC-RV

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14
Q

volume that lung can maximally hold

A

TLC
TLC= TV+ERV +RV

200ml in newborn

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15
Q

The tidal volume only uses __% or less of our TLC

A

10%

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16
Q

Tidal volume can increase by?

A

modifying insoiratory and expiratory

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17
Q

Ventilatory strategy of transient tachypnea

A

Increase frequency of breathing

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18
Q

Suprasternal retractions and head bobbing

A

Recruit more alveoli by increasing the use of accessory muscles

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19
Q

STrategy to recruit more functional respiratory units from ERV

A

Using inspiratory musles of laryngeal narrowing during expiration

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20
Q

Due to poor compliance and attempt to improve oxygenation

A

Grunting

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21
Q

only age group that exhibits grunting

A

Newborns

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22
Q

Involves closing glottis to keep the lungs a little inflated

A

Grunting

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23
Q

Observe in conditions with obstruction of airways and in most aspiration syndromes

A

Changing the size of alveolar gas reserve

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24
Q

Cardiac causes of respiratory distress

A

Left to right shunt
Congestive cardiac faiure
Obstruction to pulmonary Venous Flow

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25
Neurologic causes of respiratory distress
Post asphyxia state Subarachnoid hemorrhage Infections
26
Metabolic causes of respiratory distress
``` Metabolic acidosis Sepsis Hyperthermia/hypothermia Narcotic withdrawal Drugs ```
27
Fastest way to correct acidosis
blowing off CO2 rapidly
28
Pulmonary (Mechanical) causes
``` Ribcage anomalies Pneumothorax Pneumomediastinum Pleural effusion Chylothorax severe abdmonal distension ```
29
Pulmonary (Developmental) causes
``` TEF CCAM CDH sequestration Pulmonary hypoplasia lobar emphysema congenital lung cyst ```
30
Pulmonary (Airway abonarmalities)
``` Chonal stenosis, atresia Laryngeal web Laryngotracheomalacia Bronchomalacia Subglottic stenosis ```
31
Pulmonary (Parenchymal) causes
``` Transient tachypnea of the newborn Hyaline membrane disease Pulmonary edema Aspiration syndromes Pneumonia pulmonary hemorrhage ```
32
Persistent Pulmonary Hypertension of the newborn
Vascular disease - not parenchymal pulmonary vascular disease -> increased pulmonary vascular resistance
33
Also known as Respiratory distress syndrome Type I
Hyaline Membrane disease (developmental disorder)
34
conditions than can cause an even lower production of surfactant in an effort to decrease metabolic demand
Hypoxemia and hypercarbia
35
Deficiency of surfactant
``` Decrease FRC Atelectasis Increased R to L shunt Increased work of breathing V/Q mismatch` ```
36
collapse of an expanded lung especially in infants which is also a failure ofpulmonary alveolu to expand at birth
Atelectasis
37
Increases R toL shunt because of pulmonary hypoperfusion
Atelectasis
38
Potent vasodilators
high pH and low Co2 conditions
39
Increased pressure due to vasoconstriction and may lead to capillary endothelial damage
Pulmonary hypertension
40
composed of a high molecular weight proteinaceous substance
Hyaline membranes
41
May cause the development of bronchopulmonary dysplasia
hyaline membrane
42
Progressive atelectasis leads to worsening in the first ____hours
24 -28 hours
43
End result of HMD
Capillary damage | Alveolar necrosis
44
Radiologic findings of HMD
Reticulo-granular pattern Severe white lungs - semisolid Steepled appearance - some alveoli have air in it
45
bronchi visible as black lines
Air bronchogram sign
46
manangement of HMD
Continuous positive airway pressure Surfactant replacement therapy
47
How do you give surfactant to a baby who is not intubated>
INSURE PROCEDURE
48
Dosage for surfactant administration intrathecally
4 cc/kg (1cc/kg/quadrant)
49
How do the surfactant enter the 4lung quadrants
Use gravity
50
Also called respiratory distress syndrome type II
Transient tachypne of the newborn TTN
51
Most common cause of respiratory distress in newborns
Transient tachypnea of the newborn
52
Main pathology of the TTN
Delay in the resorption of fetal lung field
53
Rate of production of fetal lung fluid
20 ml/kg of fluid in the fetal lungs
54
What happens during labor?
there is plenty of beta adrenergic stimulation -> there is cessation of fetal lung fluid production
55
How many fluid is removed during labor
2/3
56
How many fluid is removed during baby's first breath
1/3
57
Fluid resorption begins with
child's first breath
58
Radiographic findings of TTN
Streaky linear hilar densities
59
Management of TTN
self limiting disease and is managed conservatively with supportive care Fluid electrolytes thermoregulati Oxygen if needed
60
Representative of all aspiration syndromes
Meconium Aspiration Syndrome
61
MAS is non existent among
Preterm neonates
62
MAS may result in
severe hypoxemia, acidosis, and respiratory failure
63
Air leak syndromesL Allows air to enter but not exit
Ball Valve Effect
64
Radiographic findings of MAS
cotton wool-like fluffy infiltrates or densities
65
Classification of PPHN
PPHN associated with pulmonary parenchymal disease PPHN with radiographically normal lungs and no evidence of parenchymal disease PPHN associated with hypoplasia of the lungs
66
Radiographic findings of PPHN
No vascular markings are observed because the vessels are constricted(oligemic x-ray)
67
Manangement of PPHN
Vasodilators
68
How to rule out pulmonary atresia
Passage of catheter through the nares