ABCN Deck 6 Flashcards

1
Q

Because of the anatomical arrangement of the brain and skull focal injury is most common in the ___ and ___ lobes.

A

frontal and temporal.

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2
Q

Diffuse axonal injury is more prominent at the ____.

A

gray-white matter junctions.

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3
Q

Reasons for risk of severe TBI for ages 0-7

A

falls & child abuse

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4
Q

Reasons for risk of severe TBI for ages 15-19

A

motor vehicle related injuries

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5
Q

Reasons for risk of severe TBI for ages 65+

A

falls

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6
Q

What is complicated mTBI? functional outcome tends to be similar to ____.

A

mTBI patients with positive neuroimaging. moderate TBI.

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7
Q

MRI in the subacute period (< 3 months) is more reliable than CT in identifying ___.

A

structural changes

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8
Q

When the patient demonstrates a measurable level of continuous memory for ___ to ___ consecutive days, it is typically appropriate to perform a brief NP eval to establish a post injury baseline.

A

2 to 3

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9
Q

A brief NP battery ___ following mild to severe TBI provided incremental value and was predictive of outcome above and beyond functional and injury severity variables.

A

one month

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10
Q

Persistence of symptoms __ months following uncomplicated mild TBI are uncommon.

A

3

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11
Q

Recovery after moderate to severe TBI often takes more than ____ months.

A

12

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12
Q

For moderate to severe TBI, over time, ___ and ___ tend to have more influence on functional recovery than injury severity.

A

social support, access to resources

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13
Q

____ in problem solving and compensatory techniques tends to be the most effective treatment for mod to sev TBI.

A

broad-based, real-world

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14
Q

These areas are unlikely to be affected following mod to sev TBI:

A

general fund of info, vocabulary (unless language centers damaged)

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15
Q

the majority of patients with mild TBI experience rapid improvement on performance-based tests within __ to ___.

A

days to weeks.

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16
Q

These areas are commonly affected following mod to sev TBI in adults:

A

novel problem solving, processing speed, attention/concentration, multitasking, language pragmatics, memory, depression, anxiety

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17
Q

These areas are commonly affected following mod to sev TBI in kids in particular:

A

long-term intellectual/academic challenges, secondary ADHD, visuospatial skills, psychomotor speed

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18
Q

the ___ principle suggests that younger brains have more plasticity and therefore a better prognosis following injury.

A

Kennard

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19
Q

Neuropathological changes described in CTE are not unique to that condition, and a sizable percentage of persons who have such changes will NOT develop ___ or commit ___.

A

dementia or suicide

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20
Q

As a direct result of repetitive concussive or subconcussive injuries, a unique distribution of p-tau accumulates in the depths of the cortical sulci.

A

CTE

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21
Q

___ was initially included in several concussion grading systems but has been de-emphasized in recent years because of its inconsistent relationship with outcome.

A

loss of consciousness.

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22
Q

erroneous assumption based on the concept of the magnitude of the stressor (the dose) and the subsequent response of the receptor.

A

dose-response fallacy

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23
Q

Relative to other outcome predictors in TBI, ____ is considered to be the most robust.

A

length of PTA

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24
Q

According to Little Black Book, ___ is related to severity of TBI.

A

Retrograde Traumatic Amnesia

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25
Q

for the Glasgow Coma scale, the ___ the score, the worse the injury

A

lower

26
Q

Commonly used classification system for mild, moderate, and severe TBI include 3 areas:

A

duration of unconsciousness, GCS, PTA

27
Q

Clinical presentation of this type of head injury involves an initial LOC, followed by return to broadly normal function for a few hours, followed by rapid deterioration of function (hours to a day). (2 kinds)

A

epidural hemorrhage due to ruptured middle meningeal artery OR subdural hemorrhage due to ruptured bridging vein.

28
Q

type of herniation involving the mesial temporal lobe

A

transtentorial or uncal

29
Q

type of herniation involving the brainstem and/or mesial temporal lobes

A

central

30
Q

type of herniation involving the cingulate gyrus

A

subfalcine

31
Q

type of herniation involving brain stem & cerebellum through foramen magnum

A

tonsillar

32
Q

Triad of clinical features associated with uncal/transtentorial herniation

A

hemiplegia, dilated (blown) pupil, coma

33
Q

__ herniation often leads to respiratory failure, cardiovascular dysfunction, and death.

A

tonsillar

34
Q

Traumatic axonal injury typically occur in 4 areas:

A

gray-white matter interfaces, long fibers of the internal capsule, corpus callosum, upper brainstem

35
Q

impaired recognition of previously meaningful stimuli

A

agnosia

36
Q

lack of concern for serious neurological impairments without denying their existence

A

anosodiaphoria

37
Q

Long-term cognitive impairment following mod to severe TBI are most notable in (4)

A

attention/concentration, working memory, processing speed, memory

38
Q

following simple commands within the first hour post injury have significantly better outcomes than those who took longer than 24 hours to follow simple commands. true or false?

A

true

39
Q

DSM-V criteria for neurocognitive disorder due to TBI?

A

Look up!

40
Q

____ patients rarely develop signs of cerebral cortical dysfunction such as seizures or aphasia. The cerebral cortical gray matter is relatively spared.

A

MS

41
Q

More than __ different illnesses produce symptoms of dementia.

A

50

42
Q

__% of all causes of dementia are reversible.

A

5

43
Q

Acute exposure to pesticides can results in the following symptoms (8)

A

headaches, blurred vision, restlessness, anxiety, depression, mental slowing, slurred speech, ataxia.

44
Q

chronic exposure to pesticides can result in (6)

A

irritability, confusion, depression, attention, memory, response speed.

45
Q

Persons with depression are less likely than those with AD to show impaired (3)

A

naming ability, verbal fluency, visuospatial ability.

46
Q

When hydrocephalus occurs, the ventricles expand in a __ to __ direction, and white matter is damaged. Thus, the ___ regions of the brain may be particularly susceptible.

A

posterior to anterior. posterior.

47
Q

Lesions of the temporal lobe can cause ___ due to interruption of the lower portions of the optic radiations. This is sometimes referred to as ___ phenomenon.

A

contralateral superior quadranopia. pie in the sky.

48
Q

Lateral cerebellar lesions affect ___ and ___. Medial cerebellar lesions affect ___, ___, and ___.

A

distal limb coordination and motor planning. trunk control, posture and balance, gait.

49
Q

The __ hemisphere has more white matter, less gray matter, more association cortex, and more interconnections.

A

Right

50
Q

___ refers to the period of time following a TBI in which new memories cannot consistently be formed.

A

Post traumatic amnesia.

51
Q

Patients with Parkinson’s have more trouble with ___ while AD patients have problems with ___ in memory.

A

retrieval; consolidation

52
Q

The ___ is involved with habit learning and OCD.

A

caudate

53
Q

___ palsy is dysarthria, dysphagia, and hypoactive jaw/gag reflex.

A

Bulbar

54
Q

Infarction of the inferior portions of the __ or ___ can cause locked in syndrome.

A

pons or medulla

55
Q

Neurotransmitter found in the neurons of the raphe nuclei.

A

serontonin

56
Q

_____ is characterized by a greater deficit in sounding out pseudowords than
reading real words

A

phonological alexia

57
Q

part of the frontal lobe associated with response initiation and inhibition.

A

orbitofrontal

58
Q

which areas of the brain are myelinated first? motor or sensory?

A

sensory

59
Q

___ presents with Parkinsonian motor symptoms but the cognitive functioning is more similar to AD.

A

LBD

60
Q

Galveston Orientation and Amnesia Test cut off scores

A

76-100 normal
66-75 borderline
< 66 impaired

61
Q

GCS cut offs

A

Severe < 8
moderate 9-12
mild 13-15

62
Q

_____ refers to an abnormality in visual perception and discriminative process, despite the absence of elementary visual deficits. These people are unable to recognize objects, draw, or copy a figure. They cannot perceive correct forms of the object, although knowledge of the object is intact

A

Apperceptive visual agnosia