3.2.4 Dysfunctional Families Evaluation (Sch) Flashcards

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1
Q

(lack of research support) many studies highlight what?

A

communication difficulties

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2
Q

(lack of research support) there is lack of research to support what theory?

A

double bind communication

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3
Q

(lack of research support) Liem gave structured tasks to parents of who?

A

11 sons with schizophrenia

11 sons without

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4
Q

(lack of research support) Liem found what between the parents communication with schizophrenic and non schizophrenic sons?

A

communication was no more disordered

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5
Q

(lack of research support) Liem’s research caused him to question what?

A

cause or effect relationship

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6
Q

(lack of research support) does double bind cause schizophrenia or does?

A

schizophrenia cause double bind

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7
Q

(lack of research support) if the theory was correct what would you expect?

A

all/most children in the family to develop schizophrenia

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8
Q

(origins) to be a complete explanation what needs to be offered?

A

the origins of dysfunctional communications

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9
Q

(origins) Koopmans - double bind communication arises at times of what?

A

family disruption

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10
Q

(origins) Koopmans - what is there little evidence of?

A

empirical research support between disruption and double bind communication

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11
Q

(origins) Koopmans - there is also an ethical issue of what?

A

blaming patients/their families

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12
Q

(origins) it has been suggested double bind communications are a symptom of what?

A

mental illness/pathology

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13
Q

(origins) it may be pathology that is a greater issue than what?

A

communications

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14
Q

(origins) what does the lack of origins do to this explanation?

A

weakens it

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15
Q

(origins) Bateson didn’t claim double bind was a whole explanation rather he said it was what?

A

a risk factor

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16
Q

(EE research support) there is a lot of evidence to support the role of expression of emotion in what?

A

relapse of schizophrenia

17
Q

(EE research support) Vaughan and Leff - in high EE homes with high contact and no medication, what was the rate of relapse?

A

90%

18
Q

(EE research support) Vaughan and Leff - in low EE homes what was the rate of relapse?

A

10%

19
Q

(EE research support) Vaughan and Leff - in high EE homes what was the rate of relapse?

A

50%

20
Q

(EE research support) in prospective studies what were high risk children?

A

those with a parent with schizophrenia

21
Q

(EE research support) high risk children who developed schizophrenia were more likely to come from where?

A

high EE homes characterised by negative relationships

22
Q

(EE research support) Marcus - Israeli high risk study - none of the high risk children with good parenting did what?

A

developed schizophrenia

23
Q

(EE research support) Kavanagh - people with schizophrenia in high risk families were how many times more likely to relapse than those in low EE families?

A

4x

24
Q

(EE research support) not all research supports EE as a predictor of what?

A

relapse

25
Q

(EE research support) McCreadie and Philips failed to find what?

A

increase in 6 and 12 month relapse rates among individuals with schizophrenia living in high risk homes

26
Q

(EE research support) McCreadie and Philips research suggest although EE may be a significant relapse factor in some, it is not what?

A

the only one

27
Q

(genes or environment) what is it difficult to separate?

A

whether it is the result of families having the same genes or same environment

28
Q

(genes or environment) the schizophrenia working group of psychiatric genomics consitorium have found what?

A

108 genetic loci associated with schizophrenia

29
Q

(genes or environment) which explanation has more supporting evidence?

A

genetics

30
Q

(genes or environment) it may be dysfunctional communication is what?

A

a CV

31
Q

(genes or environment) genetics leads to poor mental health which then leads to what?

A

poor communication

32
Q

(conclusion) what would be a better way to look at schizophrenia?

A

diathesis stress model

genes and environment both contribute