2.23 PPS Flashcards
Who developed first polio vaccines?
Salk and Sabin
polio transmission
fecal/oral
iron lung
creates negative pressure to help them breathe
Salk’s vaccine
- dead virus (currently, we get this one in first world)
- not as stable, not a good shelf life
Sabin’s vaccine
- attenuated virus
- used more in underdeveloped areas now
- takes 3 doses to get full immunity
What structure is affected with polio? Why is this problematic?
- anterior horn cells affected
- during growth, need muscle growth to help strengthen bones and make them grow
Prior to vaccines, what was treatment for polio?
- treatments initially didn’t do well, mostly wait and see
- used a lot of bracing
What do we usually treat nowadays?
post polio syndrome, not true polio
If a post polio patient is below 3/5 strength, what must be done for treatment?
- must go slower (isometric, AAROM)
- don’t just make them do a ton right off the bat, can do a lot of damage
- SLOWLY build up strength
results of polio
- scoliosis
- a lot of postural issues
Why did people get strong in certain muscles after having polio?
peripheral nerve regeneration
With polio, what happens when there’s a nerve still working, but it’s not getting enough activation?
collateral sprouting of nerves » giant motor units (GMUs)
Problem with collateral sprouting and GMUs in polio survivers
- these begin to fail for polio survivors, don’t know why
- like putting too many plugs into one outlet, overloaded
- may or may not have an effect on the person
What must be considered with any new weakness in a polio survivor?
post polio syndrome
orthopedic issues with polio survivors
There will be a lot of orthopedic issues (aside from weakness) depending on how bad the polio was the first time
- Long term bracing, crutch use
- carpal tunnel, hand deformities, shoulder laxity, etc.