Unit 2 Week 5 PAD Flashcards

1
Q

what is the cycle of PAD?

A

PAD ➙Leg pain ➙Decreased activity ➙Deconditioning ➙ PAD

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2
Q

what are the factors leading to critical limb ischemia?

A
  • Diabetic microangiopathy
  • Atherosclerosis usually secondary to DM, HTN, or smoking
  • CHF with severely decreased cardiac output
  • Vasospastic diseases (Raynaud’s phenomenon, prolonged cold exposure)
  • Smoking and other tobacco use
  • Infection (abscess, cellulitis, osteomyelitis)
  • Skin and soft tissue injuries
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3
Q

what is critical limb ischemia characterized by?

A
  • Pain at rest
  • ABI usually ≤ 0.4
  • Non-healing wounds and gangrene
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4
Q

Interpretation of ABI Values
* ≥1.3
* 1.00 - 1.29
* > 1.1
* 0.91 - 0.99
* 0.41 - 0.90
* ≤0.4

A
  • ≥1.3 are non-compressible; likely PAD; measure toe pressures
  • 1.00 - 1.29 are considered WNL
  • > 1.1 with risk factors and hx should be viewed with suspicion
  • 0.91 - 0.99 are considered borderline PAD
  • 0.41 - 0.90 are considered moderate to mild PAD
  • ≤0.4 are considered severe disease correlating with critical limb
    ischemia
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5
Q

what is the Segmental Pressures test?

A

BP measured along the LEs to localize an area of decline in systolic pressure
Relative lack of blood flow during diastole indicative of arterial disease

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6
Q

what is exercise ABI testing?

A
  • Confirms the PAD diagnosis
  • Assesses the functional severity of claudication
  • May detect PAD in the presence of normal resting ABI
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7
Q

what is plantar flexion exercise ABI?

A
  • Used if exercise ABI is impractical
  • May be able to reproduce a treadmill-derived fall in ABI
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8
Q

what vascular lab testing can be done for PAD?

A
  • Color duplex ultrasonography: non-invasive
  • Magnetic resonance angiography (MRA)
  • Computed tomographic angiography (CTA)
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9
Q

what is medical management for PAD?

A
  • Smoking cessation
  • Intensive antihypertensive therapy
  • Clopidogrel (plavix):antithrombotic, blocking ADP receptors on platelets
  • Aspirin: antithrombotic, less effective than plavix
  • Cilostazol (pletal): phosphoesterase inhibitor produces vasodilation, inhibits platelet aggregation, not to be used for pts with CHF
  • Pentoxifylline (trental): phosphoesterase inhibitor that improves blood flow through occluded areas and inhibits platelet aggregation, but does not improve walking distance as much as pletal
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10
Q

what interventions are important for PAD?

A
  • Education
  • Exercise protocol for PAD
  • Home exercise
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11
Q

what should be a patient with PAD be educated on?

A
  • Effect of elevating legs
  • Energy conservation
  • Signs of other arterial disease: CAD, CVA, CRI
  • Exercise program
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12
Q

what is the exercise protocol for PAD?

A
  • 5-10 minute warm-up and cool-down each
  • Treadmill or track walking
  • Resistance exercise complementary, but not a substitute for walking
  • Intensity: sufficient to cause claudication within 3-5 minutes
  • Time: until moderately severe claudication reached
  • Rest until claudication resolves in either sitting or standing
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13
Q

what is the FITT plan for those with PAD?

A
  • Frequency: 3–5 supervised sessions/week
  • Intensity increased by either speed or grade to reach claudication in 3-5 min
  • Time: 35 building to 50 minutes of walking each session + warm-up/cool down
  • Up to 6 months
  • Type: treadmill or track walking
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14
Q

what are the surgical interventions that can be done for PAD?

A
  • Critical limb ischemia or failure of conservative options
  • Endovascular: placement of stent graft, usually for aneurysms
  • Angioplasty with/without stents
  • Thrombolysis
  • Bypass grafting
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