The child w/ respiratory dysfxn Flashcards

1
Q

Assessment

A

-Respiration: Tachypnea, Hyperpnea/hypopnea
-Associated Observations: Retractions, Nasal flaring, Head bobbing, Noisy breathing, Stridor, Grunting, Wheezing, Color changes of the skin, Chest pain, Clubbing, Cough

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2
Q

Dx tests

A

-Pulmonary Function Tests: Radiology & Other Diagnostic Procedures, Blood Gas Determination, Noninvasive monitoring: Pulse oximetry, Transcutaneous monitoring, End-tidal carbon dioxide monitoring (ETCO2)

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3
Q

Pulse ox

A

-Most helpful in determining extent of hypoxia
-Ensuring the sensor probe is properly placed is the
priority nursing action. (ATI)
-Good contact with skin, remove nail polish, ink on feet
-Pulse ox number should have an ocean-like wave next to it

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4
Q

RTIs

A

-Described according to area of involvement
-Spread due to contiguous nature of the mucous membrane
lining the entire tract
-Respiratory infections are cause of majority of acute illnesses in
children
-Upper respiratory tract
-Oronasopharynx, pharynx, larynx, and trachea
-Lower respiratory tract
-Bronchi, bronchioles, and alveoli
-Croup syndromes
-Infections of the epiglottis or larynx

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5
Q

Etiology of dysfxn

A

-Viruses: RSV, Parainfluenza
-Others: Group A beta-hemolytic streptococci, Staphylococci, Chlamydia trachomatis, Mycoplasma, pneumococci, Haemophilus influenzae

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6
Q

Age

A

● Infants younger than 3 months—maternal antibodies
● 3 to 6 months—infection rate increases
● Toddler and preschool-ages—high rate of viral infections
● Older than 5 years—increase in Mycoplasma pneumonia
and beta-strep infections
● Increased immunity with age

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7
Q

Size

A

● Diameter of airways is smaller: When edema, mucus, or bronchospasm is present, the capacity for
air passage is greatly diminished; Small reduction in the diameter of a child’s airway will result in an
exponential increase in resistance to airflow, causing increased
work or breathing
● Distance between structures is shorter, allowing organisms
to rapidly move down
● Short and open eustachian tubes

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8
Q

Resistance

A

● Immune system deficiencies
● Allergies, asthma
● Cardiac anomalies
● Cystic fibrosis
● Exposure to infections in daycare
● Exposure to second-hand smoke

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9
Q

Seasonal variations

A

● Most common during winter and spring
● Mycoplasmal infections more common in fall and winter
● Asthmatic bronchitis more frequent in cold weather
● RSV season considered winter and spring

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10
Q

Clinical s/s

A

○ Fever
○ Anorexia, vomiting, diarrhea, abdominal pain
○ Cough, sore throat, nasal blockage, or discharge
○ Respiratory sounds

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11
Q

Nursing interventions

A

● Ease respiratory effort: Effecting breathing patterns, Promote airway clearance and maintenance, Adequate gas exchange
● Fever management
● Promote rest and comfort
● Infection control
● Promote hydration and nutrition
● Family support and teaching
● Prevent spread of infection
● Provide support and plan for home care

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12
Q

URIs

A

● Acute viral Nasopharyngitis—“common cold” More frequent in the winter
● Caused by numerous viruses: RSV, rhinovirus, adenovirus, influenza, and parainfluenza viruses
● Clinical manifestations: Fever—varies with age of child, Irritability, restlessness, Decreased appetite and fluid intake, Nasal inflammation, Vomiting and diarrhea
● Therapeutic Management –> OTC Cold Preparations/Prevention

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13
Q

URI meds

A

● OTC pediatric cold remedies are not recommended for treating “common cold”
● Antihistamines ineffective in most cases
● Antipyretics for comfort from fever
● Cough suppressants for dry cough: Caution alcohol content
● Decongestant to shrink swollen nasal passages: Nose drops more effective than oral administration

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14
Q

Acute infectious pharyngitis

A

● AKA “acute streptococcal pharyngitis”
● Etiology and risks in children: Group A β-hemolytic streptococci (GABHS) infection of the upper airway (strep throat)
● Clinical Manifestations: Pharyngotonsillitis, headache, fever & abdominal pain, Inflamed tonsils and pharynx that are covered by exudate, Anterior cervical lymphadenopathy
● Diagnostic Evaluation –> 80 to 90% are viral: Rapid antigen testing, Culture
● Risk of rheumatic fever, acute glomerulonephritis
● Bacterial treatment: ABX as ordered, Change toothbrush after 24 hrs on ABX
● Viral treatment: symptom management

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15
Q

Strep meds

A

● Penicillin: Oral (Needs 10-day treatment to decrease risk of rheumatic fever and glomerulonephritis post strep), Issues with medication compliance
○ IM: Penicillin G: Resolves compliance issue (one injection), Painful injection, Penicillin G procaine is less painful injection, CANNOT give penicillin G by IV route
○ Erythromycin if penicillin allergy
○ Other antibiotics

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16
Q

Tonsillitis

A

● Enlarged tonsils can block the nose and throat & disrupt the function of the Eustachian tubes: Often occurs with pharyngitis
● Acute or chronic: Acute is self-limiting
● Enlarged tonsils can block the nose & throat, disrupt fxn of the eustachian tubes
● Commonly occurs with pharyngitis

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17
Q

Tonsillitis tx

A

● Therapeutic Management: Medical, Surgical –> tonsillectomy/adenoidectomy (Controversial, May be indicated with massive hypertrophy)
● Nursing Care Management:
○ Codeine contraindicated postoperatively, Food & Fluids, Monitor for “continuous swallowing” –> bleeding postoperatively

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18
Q

Tonsillectomy/adenoidectomy

A

● Indicated for recurrent throat infections
● Post-op: Assess for bleeding, Assess airway and VS, Comfort measures, Educate about post-op hemorrhage

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19
Q

Nursing Concerns-Post-Operative Tonsillectomy

A

● Airway: Positioning
● Bleeding: Observation—frequent swallowing, Prevention of recurrent bleeding, Maintain quiet environment, Minimize agitation/crying, NO suctioning

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20
Q

Infectious Mononucleosis (aka mono)

A

● Acute, self-limiting infection; common in younger than 25-year-olds
● Etiology & Pathophysiology –> Epstein Barr Virus (EBV)
-s/s: fever, pharyngitis, looks like strep throat (when given abx for strep –> rash)
-May cause spleen rupture (d/t splenomegaly)
● Diagnostic Evaluation: CBC –> atypical leukocytes, Monospot –> nonspecific heterophile antibody test

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21
Q

Influenza

A

● Flu A, B, & C
● 1-3 day incubation period
● Prevention: vaccine

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22
Q

Influenza meds

A

● Antivirals for children: Oseltamivir (Tamiflu), Zanamivir (RELENZA)
● Must start within 48 hr of symptom onset
● Avoid aspirin—possible link with Reye syndrome (damages brain and lever)

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23
Q

Prevention of influenza

A

● Vaccine recommended for children greater than 6mo
● New vaccine annually
● Types: Inactivated influenza vaccine, Live attenuated influenza vaccine which is
administered intranasally.
● United States –> quadrivalent: Contain 2 strains of influenza A & 2 strains of influenza B
● Can be given simultaneously with other vaccines
but at a separate site

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24
Q

Otitis media

A

● Streptococcus pneumoniae, H. influenzae & Moraxella catarrhalis, The two viruses most likely to precipitate OM are RSV & influenza
● Impact of passive smoke inhalation/crowded living conditions
● Therapeutic Management : Acute AOM/Recurrent AOM/AOM with Effusion (Myringotomy)

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25
Otitis media: infant feeding methods
* Breastfed infants have less OM than bottle-fed infants * Immunoglobulin A * Position in breastfeeding may decrease reflex in Eustachian tubes
26
Otitis media: abx therapy
* If over age 6 month --> “watchful waiting” up to 72 hours for spontaneous resolution * Antibiotics if less than 2 years with persistent acute symptoms of fever & severe ear pain * Antibiotics if less than 6 months * Topical relief heat or cold or benzocaine drops (Rx)
27
Otitis media w/ effusion (OME)
Prevention: pneumococcal vaccine, breastfeeding, preventing exposure to tobacco smoke
28
Otitis externa (OE)
* “Swimmer’s ear” * Infections of the external ear canal * Etiology: Staphylococcus or Corynebacterium * Acute Otitis Externa (AOE)
29
OM meds
● First-line antibiotics: Amoxicillin PO 80 to 90 mg/kg/day, divided twice daily ×10 days ● Second-line antibiotics: Amoxicillin-clavulanate (Augmentin), azithromycin, Cephalosporins IM (If highly resistant organism or noncompliant with oral doses, IM is painful --> Reconstitute with 1% lidocaine, w/o EPI, to decrease pain of injection) ● Analgesic–antipyretic drugs: Acetaminophen, Ibuprofen (only if > 6 m) ● No steroids, antihistamines, decongestants, antibiotic ear drops
30
Acute epiglottitis
● Serious obstructive, inflammatory process ● Clinical manifestations: Abrupt onset, Sore throat, pain, tripod positioning, retractions, Inspiratory stridor, mild hypoxia, distress, Fever ● Therapeutic mgmt: Potential for complete respiratory obstruction ● Nursing mgmt: position for comfort, no tongue blade, keep suction at bedside, keep emergency respiratory equipment at bedside, decrease anxiety ● Prevention: Hib vaccine
31
Croup syndromes
● Characterized by hoarseness, “barking” cough, inspiratory stridor & varying degrees of respiratory distress ● Affect larynx, trachea & bronchi ● Described by anatomic area primarily affected: Epiglottitis (or supraglottitis), laryngitis, laryngotracheobronchitis (LTB) & tracheitis
32
Acute laryngitis
● More common in older children & adolescents ● Usually caused by virus ● Chief complaint --> hoarseness ● Generally self-limiting & without long-term sequelae ● Acute laryngotracheobronchitis (LTB): Most common type of croup experienced by children admitted for hospitalization, Primarily affects children 6 to 36 months old
33
Acute Spasmodic Laryngitis
● Also called spasmodic croup, midnight croup ● Paroxysmal attacks of laryngeal obstruction ● Occurs chiefly at night ● Inflammation: mild or absent ● Most often affects children ages 1 through 3
34
Bacterial tracheitis
● Infection of the mucosa of the upper trachea ● Distinct entity, features of croup & epiglottitis in older children (5 to 7 years) ● s/s: Thick, purulent secretions, Respiratory distress & stridor, May develop life-threatening obstruction or respiratory failure ● Mgmt: Humidified O2, Antipyretics, abx, intubation
35
LRI
● Considered the “reactive” portion of the lower respiratory tract ● Includes bronchi and bronchioles ● Cartilaginous support not fully developed until adolescence ● Constriction of airways
36
Bronchitis
● AKA tracheobronchitis ● Inflammation of the large airways, frequently associated with URIs: Persistent dry, hacking, non-productive cough ● Usually viral ● Mild and self-limiting
37
Bronchiolitis
● Most often caused by respiratory syncytial virus (RSV) ● Primarily affects the bronchi and bronchioles: Occurs at bronchiolar level, Intraluminal obstruction from swelling leads to hyperinflation, obstructive emphysema, and areas of atelectasis ● Initially, URI symptoms which progress to increased WOB and hypoxia ● Infants are typically hospitalized ● Prevention of RSV: prophylaxis (palivizumab [Synagis])
38
RSV
-Copious secretions, tachypnea, apnea, cyanosis -Monitor O2 and respiratory status -Dx: PCR nasal swab -Tx: deep suctioning before eating, fluids, antipyretics -Droplet precautions
39
COVID-19
-Airborne precautions -Tx: veklury (remdesivir),
40
Multi-system inflammatory syndrome (MIS-C)
-Post-covid syndrome --> hyperinflammatory syndrome -Fluids, O2, BP meds, abx, blood clot meds
41
Pneumonia
● Causative organism varies greatly by age category: Young children at risk for aspiration PNA ● The most useful classification is etiologic agent: Viral, Bacterial, Mycoplasmal, foreign substances ● Hospitalization for O2 therapy, IV hydration, and IVAB may be necessary if s/s of respiratory distress: tachypnea, retractions, nasal flaring, low SaO2 ● s/s: Fever, malaise, rapid respiration, cough, Chest or abdomen pain, nausea ● Dx: X-ray --> Pulmonary infiltrates, Lobar consolidation, Pleural effusion ● Lab tests: Cultures --> sputum, blood cultures, lung aspiration, & biopsy; WBC ● Prevention: PCV vaccine
42
Asthma
● Most common chronic respiratory illness of childhood -Chronic inflammatory disorder of the airways: Can lead to airway remodeling ● Recurring episodic symptoms: Wheezing, Breathlessness,Chest tightness, Cough (especially at night), Limited air flow or obstruction that reverses spontaneously or w/ tx, Bronchial hyperresponsiveness
43
Neonatal pneumonia (not on test)
● Occurs in the immediate neonatal period ● Infected mother transmits to fetus via ascending infection or during delivery ● Multiple organisms ● Infant is afebrile with cough, tachypnea, rales ● X-ray ● Abx
44
Aspiration pneumonia
● Risk for child with feeding difficulties ● Types: lipoid, inhalation, hydrocarbon ● Mgmt: Prevention of aspiration, Feeding techniques, positioning ● Avoid these aspiration risks: Oily nose drops, petroleum distillates, Solvents, Talcum powder
45
Pneumonia pathogenesis
● Severity of lung injury depends on ingested substance ● Hydrocarbons: Petroleum distillates often contain heavy metals or other toxic chemicals, Often found in home or garage, Severe effect even with small amount ingested, Spread over surface of tissues & interfere with gas exchange
46
Hydrocarbon pneumonia s/s
● Coughing, vomiting, aspiration ● CNS: agitation, restlessness, confusion, drowsiness, and coma ● Elevated body temperature (37.8° to 40° C) ● Dyspnea, cyanosis, tachycardia, tachypnea, nasal flaring, retractions ● Hemoptysis, pulmonary edema, severe cyanosis & death within 24 hrs of aspiration
47
Pulmonary edema (PE)
-Types: cardiogenic (hydrostatic hemodynamic), noncardiogenic (factors that increase pulmonary capillary permeability) -Tx: PEEP, pulse ox, ventilation, intubation
48
Acute lung injury (ACL)/Acute respiratory distress syndrome (ARDS)
● Continuum of symptoms of ALI, with ARDS the most severe form ● Characterized as respiratory distress and hypoxia within 72 hours after serious injury or surgery in person w/ previously normal lungs -ICU, high mortality ● Animation: Bag Ventilation, dx criteria are based on the Berlin definition of ARDS ● Mgmt: oxygenation, pulmonary perfusion, tx of infection, adequate cardiac output, intubation
49
Suspicion for Inhalation Injuries
● History of exposure to flames whether burns are present ● Soot around nose or in sputum, singed nasal hairs, or mucosal burns of the nose, lips, mouth, or throat ● Hoarse voice & cough -Swollen neck, burned hair -FLASH (facial skin, lips, airway, sputum, hair ● Increased inspiratory & expiratory stridor ● Tachypnea, tachycardia, abnormal breath sounds
50
Inhalation Injuries: Pulmonary Complications
● Early carbon monoxide poisoning, airway obstruction & pulmonary edema ● ARDS 24 to 48 hours after injury ● Late complications: bronchopneumonia & pulmonary emboli ● Airway compromise from eschar after a severe burn
51
Smoke Inhalation Injuries
● Heat injury: Thermal injury to upper respiratory tract, Reflex glottis closure ● Local chemical Injury: Insoluble gases in the lower respiratory tract ● Systemic injury: CO causing tissue hypoxia ● Mgmt: 100% O2, bronchodilators
52
Environmental tobacco exposure
● Children exposed to passive or environmental smoke ● Legislation to prevent smoking in public places ● Use of e-cigs
53
Allergies vs cold: allergies
-Occur repeatedly -Seasonal -No fever -Itching in eyes and nose -Trigger constant sneezing -Accompanied by eye and ear problems
54
Asthma
● Chronic inflammatory disorder of airways ● Bronchial hyperresponsiveness ● Episodic ● Limited airflow or obstruction that reverses spontaneously or w/ treatment ● Atopy (genetic) ● s/s: Dyspnea, Wheeze, Cough ● Dx: PFTs most accurate, peak expiratory flow meter ● Nursing mgmt: education plan, severity classification, acute asthma care, avoid allergens, relieve bronchospasm, inhalers ● Risks: mother < 20 y, smoking on maternal side, African American ethnicity, allergic genes on chromosome 5, lack of access to medical care, prior life-threatening attacks
55
Asthma types and categories
● Types: Recurrent wheezing usually precipitated by a viral respiratory tract infection (e.g. RSV), Chronic asthma associated with allergy persisting into later childhood and often adulthood, Associated w/ girls who develop obesity and early-onset puberty by 11 y, Cough-variant asthma ● Categories: Intermittent, mild persistent, moderate persistent, and severe persistent
56
Asthma severity classification in children 5+ (not on test)
● Stage I—mild, intermittent asthma ● Stage II—mild, persistent asthma ● Stage III—moderate, persistent asthma ● Stage IV—severe, persistent asthma
57
Asthma meds
● Long-term control meds: Inhaled corticosteroids, cromolyn sodium and nedocromil, long-acting β2-agonists, methylxanthines, leukotriene modifiers (Advair or Pulmicort), theophylline, leukotriene modifiers, Mg sulfate, oral steroids ● Quick-relief (“rescue”) medications: Short-acting β2-agonists, anticholinergics, systemic corticosteroids AKA bronchodilators, EPI-pen
58
Asthma drug routes
● Inhaled for most medications: MDI with spacer, Nebulizer (Albuterol tx) ● Generally less effective in treating asthma: Oral, IV
59
Asthma interventions
-Breathing exercises and physical therapy -Chest physiotherapy (CPT) -Hyposensitization
60
Acute asthma care
● Calm nursing presence ● Monitor with pulse oximetry ● Assess breath sounds ● Allow older children to sit up if they are more comfortable in that position ● Safety precautions ● Allow parents to remain with children: Try to decrease parental stress
61
Signs of severe Respiratory Distress in Children with Asthma
● Remains sitting upright, refuses to lie down ● Sudden agitation ● Agitated child who suddenly becomes quiet ● Diaphoresis ● Pale
62
Status asthmaticus
● Life threatening episode of airway obstruction that is often unresponsive to common treatments: Can result in respiratory failure and death ● Medical Emergency!: Lack of air movement in lungs, Be prepared for intubation, Monitor SaO2 and ABGs carefully ● Humidified O2, continuous bronchodilator nebulizer tx, corticosteroid, Mg sulfate, sit the child up ● Emergency tx: EPI 0.01 mL/kg subq (max is 0.3 mL), IV Mg sulfate, IV ketamine, IV corticosteroids, heliox (breathing gas mix of He and O2)
63
Cystic fibrosis (CF)
● Autosomal recessive genetic disease ● Abnormal gene is located on the long arm of chromosome 7 ● 95% known cases occur in Caucasians ● Most common lethal genetic illness among Caucasian children ● Approximately 3% of US Caucasian population are symptom-free carriers
64
CF patho
● Characterized by several unrelated clinical features: Respiratory system, GI system (small intestine, pancreas, bile ducts, Growth patterns), Reproductive system and skin ● Increased viscosity of mucous gland secretion: Results in mechanical obstruction, Thick, inspissated mucoprotein accumulates, dilates, precipitates, coagulates to form concretions in glands and ducts, Respiratory tract and pancreas are predominantly affected
65
CF dx
● Early infancy screening ● DNA testing (autosomal recessive) ● Sweat chloride test (garbage bag sweatsuit, test level of Cl in sweat, 3x normal child) ● Chest radiography ● Pulmonary function tests (PFTs) ● Stool fat (fatty, stinky stools) and/or enzyme analysis ● History and physical exam
66
CF s/s
● Meconium ileus at birth ● Prolapse of the rectum ● Distal intestinal obstruction syndrome ● Pancreatic enzyme deficiency (d/t pancreatic fibrosis) steatorrhea ● Excretion of undigested food in stool: Stool is bulky, frothy, and foul smelling ● Failure to thrive/growth failure/wasting of tissues, hx of repeated respiratory infections ● Sweat gland dysfunction ● Increased weight loss despite increased appetite ● Gradual respiratory deterioration ● Wheezing respiration; dry, nonproductive cough ● Generalized obstructive emphysema ● Patchy atelectasis ● Cyanosis ● Clubbing of fingers and toes ● Repeated bouts of bronchitis and pneumonia ● Stasis of mucus increases the risk for life-threatening respiratory infections ● Delayed puberty in females ● Sterility in males ● Parents report that children taste “salty” ● Dehydration ● Hyponatremic or hypochloremic alkalosis
67
CF respiratory s/s
● Viscous secretions are difficult to expectorate and obstruct bronchi and bronchioles: Cause atelectasis and hyperinflation, Stagnant mucus leads to destruction of lung tissue, Stagnant mucus provides a favorable environment for bacteria growth ● Mgmt: Improving ventilation, removing mucopurulent secretions, and administering antimicrobial agents
68
CF respiratory progression
● Gradual progression follows chronic infection ● Bronchial epithelium is destroyed ● Infection spreads to peribronchial tissues, weakening bronchial walls ● Peribronchial fibrosis ● Decreased O2/CO2 exchange ● Chronic hypoxemia causes contraction and hypertrophy of muscle fibers in pulmonary arteries and arterioles ● Pulmonary hypertension ● Cor pulmonale (RHF, enlarged RV d/t high BP in lungs) ● Pneumothorax ● Hemoptysis
69
CF and GI tract
● Thick secretions block ducts → cystic dilation → degeneration → diffuse fibrosis ● Prevents pancreatic enzymes from reaching duodenum -Meconium ileus --> meconium is abnormally thick and sticky ● Impaired digestion/absorption of fat → steatorrhea ● Impaired digestion/absorption of protein → azotorrhea ● Endocrine function of pancreas initially stays unchanged ● Eventually pancreatic fibrosis occurs; may result in diabetes mellitus ● Focal biliary obstruction results in multilobular biliary cirrhosis ● Impaired salivation
70
CF respiratory mgmt
● Airway clearance therapies (ACTs): Chest PT, PEP, Active-cycle-of-breathing, forced expiration, mechanical vest ● Bronchodilator medication prior to ACT ● Dornase alfa (Pulmozyme): Decreases mucus viscosity ● Aggressive treatment of pulmonary infections: Home IV abx therapy, Aerosolized abx ● Pneumothorax ● Hemoptysis ● Transplantation: heart, lung, pancreas, and liver for adolescents who have advanced disease
71
CF GI mgmt
● Administration of pancreatic enzymes & ADEK ● High-protein, high-calorie diet, as much as 150% rDA increase fluids ● Reduction of rectal prolapse ● Salt supplementation ● Oral glucose-lowering agents or insulin injections as needed ● Treat gastroesophageal reflux: Place the patient in the upright position after meals, H2 Blockers
72
CF Endocrine mgmt
● CFRD (cystic fibrosis-related diabetes): Characteristics of both Type 1 & Type 2 DM, Increased risk for glucose mgmt problems ● Screening: oral glucose tolerance test at 10 years of age ● Monitor blood glucose levels: 3x/day, Insulin ● Diet ● Exercise ● A1C measurements ● Regular eye examinations: Retinopathy
73
CF prognosis
● Estimated life expectancy for a child born with CF is 40 yrs ● CF continues to be a progressive, incurable disease ● Organ transplantation has increased the survival rate: Heart–lung and bilateral lung transplantation, Liver and pancreas transplantation ● Maximize health potential: Nutrition, Prevention and early aggressive treatment of infection, Pulmonary hygiene
74
CF family support
● New research hope: Transplantation, Gene therapy, Bilateral lung transplants, Improved meds ● Child requires treatments multiple times each day: child may rebel, Depression & anxiety common ● Frequent hospitalizations: Nosocomial infections (pt may be on contact precautions)
75
Respiratory failure (RF)
● Respiratory insufficiency: Increased work of breathing (WOB), Hypoxemia and acidosis ● Respiratory arrest: Complete cessation of respiration ● Apnea: Cessation of breathing for > 20 sec ● Restlessness, tachypnea, tachycardia, diaphoresis ● Retractions, grunting, changes in resp pattern, CNS symptoms ● Supplemental O2, positioning, suctioning, CPAP/BiPAP, consider early intubation ● Resuscitative measures if respiratory arrest