TBI Flashcards

1
Q

Risk factors for TBI

A

Ages 15-24 & 64+, men >women, ETOH, drug abuse, prior brain injury, SES/education, unemployment, divorce, LD/ADHD, psych illness, heart disease & HTN

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2
Q

Glasgow Coma Scale (GCS)

A

Assesses degree of impaired consciousness via responsiveness level in eye opening, motor mvmt, & verbal communication

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3
Q

What are the advantages of using GCS score for measuring TBI severity?

A

scores can be determined with 1st 24 hours, predictive of early important outcomes & later functional outcomes

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4
Q

What are the disadvantages of using GCS score for measuring TBI severity?

A

which GCS score to use (post-resuscitation, ER admission, etc.), can be affected by early management (intubation, sedating meds), limited in pts w/ aphasia or facial injuries

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5
Q

What are factors that complicate the use of initial GCS as a prognostic indicator?

A

Drugs/ETOH, sedation, intubation, facial injuries, echymosis, delayed deterioration (e.g., slow bleeding SDH)

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6
Q

Time to follow commands following TBI is predictive of

A

Global outcomes, NP functioning, personal independence, employment

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7
Q

What are the advantages to using time to return to conscious state as a marker for TBI severity?

A

takes into account early complications, can be obtained during early stages of recovery

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8
Q

What are the disadvantages to using time to return to conscious state as a marker for TBI severity?

A

interval can affected by sedation, difficult to interpret behaviors & fluctuations in MS, not immediately available for early prediction of outcome, no commonly agreed upon classification scheme, often not available to later-treating physicians

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9
Q

Posttraumatic amnesia

A

Phase of recovery from TBI during which the patient is responsive, but acutely confused, disoriented, & unable to form & retain new memories

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10
Q

PTA is predictive of

A

NP outcome, independent living states, return to work

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11
Q

Using duration of unconsciousness, what is a mild, mod, sev TBI?

A

Mild: 24 hrs

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12
Q

Using GCS scores, what is a mild/mod/sev TBI?

A

Mild: 13-15
Mod: 9-12
Sev: 3-8

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13
Q

Using duration of PTA, what is a mild/mod/sev TBI?

A

Mild: 7 days

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14
Q

Areas of the brain that are most vulnerable to diffuse axonal injury

A

Frontal & temporal lobes, BG, perinventricular zones, CC, brainstem fiber tracts, superior cerebellar peduncles

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15
Q

Glasgow Outcome Scale

A

Death
Vegetative state
Severe disability (requires assistance to meet basic needs)
Moderate disability (unable to return to non-sheltered work or resume other major roles)
Good recovery

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16
Q

Factors predictive of functional outcome after TBI

A

Pre-injury employment status, demographic variables, injury severity, CC atrophy, physical impairments, early cognitive status, post-injury depression, impaired self-awareness, early fx status, family support, post-acute brain injury rehab

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17
Q

Risk factors for substance abuse following TBI

A

pre-injury history, onset of depression since injury, better physical functioning, male gender, young age, uninsured, single

18
Q

Negative predictors of return-to-work following TBI

A

severity of injury, age >40, low education, greater physical or cognitive impairment, personality change, substance abuse

19
Q

Positive predictors of return-to-work following TBI

A

multidisciplinary team approach during acute rehab, socially inclusive work environment, environmental modifications, focus position on strengths of the individual

20
Q

ACRM mild TBI definition

A

A traumatically induced physiological disruption of brain fx: any LOC, any loss of memory for events before/after accident, any alteration in MS at time of accident, focal neuro deficits that may/may not be transient

Severity of injury does not exceed: LOC of 30 mins or less, initial GCS of 13-15 after 30 mins, PTA not >24 hrs

21
Q

Psychological causes of chronic postconcussion syndrome

A
Attributions/misattributions
Diagnosis threat
Expectation as etiology
"Good old days" bias
Iatrogenesis
Nocebo affect
22
Q

Tetany

A

Repetitive muscle stimulation by electricity that prolongs contact during electrocution

23
Q

Second Impact Syndrome

A

When a 2nd TBI is sustained before the physiological reactions to the prior injury have resolved; diffuse swelling within hours of even mild injury that is likely the result of compromised cerebral autoregulation

24
Q

What constitutes primary, secondary, tertiary, & quaternary damage secondary to blast injuries?

A

Primary - blast wave
Secondary - flying debris
Tertiary - blast winds throw individuals into other things
Quaternary - anything else, like burns, toxic inhalations, crush injuries

25
Q

The younger the child at the time of TBI, the more likely to display

A

Brain swelling, hypoxic-ischemic insult, diffuse rather than focal injuries

26
Q

Academic performance in older children following TBI

A

Academic decline in behavioral terms with increased risk of retention, special ed placement, etc.

Achievement tests tend to be insensitive to their academic changes

27
Q

Academic performance following TBI in younger children

A

More likely to demonstrate deficits on standardized achievement tests than older children

28
Q

Academic performance following TBI in children is predicted by

A

Premorbid academic fx, postinjury NP fx, postinjury bx adjustment, family environment, age at injury (preschool injury assoc. w/ school failure & slower acquisition of academic skills)

29
Q

Factors related to pediatric TBI outcome

A

Severity, pre- & post-injury family status, treatment received, premorbid fx

30
Q

Social functioning following pediatric TBI

A

Rated as less socially competent & lonelier than peers, poor social outcomes persist over time, relationship b/t injury & social outcome moderated by poor family fx, lower SES, lack of family resoources

Children w/ frontal lobe injury more likely to have problems

31
Q

There is empirical support for what treatments following pediatric TBI?

A

Operant conditioning in decreasing aggressive behaviors, school-based social interventions, cognitive remediation for attention skills, involvement of family member as member of treatment team

32
Q

Galveston Orientation & Amnesia Test (GOAT)

A

Test of orientation; starts at 100 & failing items of orientation result in subtractions

33
Q

Contusions are seen most in what areas?

A

Orbitofrontal cortex, anterior temporal lobe, posterior portion of superior temporal gyrus & adjacent parietal opercular area

34
Q

Diffuse axonal injury (DAI)

A

Breaking/shearing/stretching of myelinated axons due to acceleration/deceleration & rotational injuries

35
Q

Excitatory amino acids that are produced after DAI have an affinity for receptors that are especially prevalent in the _____ & ______.

A

Hippocampus & thalamus

36
Q

Increase in risk in dementia with aging after history of severe TBI

A

4-5x higher

37
Q

Risk of 2nd TBI following 1st

A

8x

38
Q

Low velocity GSW injuries create contusions at

A

Entry & contrecoup points

39
Q

High velocity GSW injuries are more likely to create

A

Diffuse injury secondary to shock waves & pressure effects

40
Q

Pathway of electrical current most likely to result in CNS disturbance

A

Head to foot

41
Q

Most damage caused by primary blast wave injuries occurs in

A

Organs that contain air (ears), fluid (stomach/intestines), tissue/air, tissue/fluid interface