Spinal Cord Injury Flashcards
Tetraplegia
Lesions in the cervical (C) region
Paraplegia
Lesions in the thoracic (T), lumbar (L) and sacral (S) regions
Spinal cord injury results in
- tetraplegia
- paraplegia
- complete or incomplete loss of somatic, sensory, and autonomic functions below the lesion level
- spinal bifida
- physiologic impairment
- autonomic troubles
- segmental neuromuscular troubles
SCI C4
artificial support for breathing
SCI C5-C8
Control of shoulder, elbow and wrist, decreased hand function
SCI T1-T6
- Autonomic dysreflexia (an uncoordinated, spinally mediated reflex response called the mass reflex)
- Poor thermoregulation
- Orthostatic hypotension
- Diminished breathing capacity
SCI T6-L2
Respiratory and motor control depends on abdominal muscles
SCI L2-S2
Lack of voluntary control
Normal upper extremities
SCI statistics
50% of those with SCI have tetraplegia
80% are men
SCI’s that are traumatic often occur at an early age
Individuals with SCI have a high risk for the development of secondary complications
Secondary complications of SCI
shoulder pain, urinary tract infection, skin pressure ulcers, osteoporosis, chronic pain, problematic spasticity, depression, CVD, obesity, Type 2 DM
Causes of SCI
Motor vehicle accidents, falls, sports, violence, surgical complications, infection, tumor
Primary Injury
an acute SCI
Secondary Injury
- Systemic injury to neurons and cells nearby
- Cellular Injury 1
- Cellular Injury 2
Cellular Injury 1
Reperfusion of ischemic area leads to free radicals, excitotoxicity, neuronal malfunction, death
Cellular Injury 2
Neutrophils, macrophages, pro-inflammatory cytokines glial scars
(These are barriers to regeneration and recovery)
Systemic injury to neurons and cells nearby
hypotension, hypoxia
Pathophysiology of primary injury
Neuronal damage/disruption -spinal cord, axon, demyelination, cell death Cytokine release -pro-inflammatory -ATP -Potassium Hemorrhage
Primary Injury Processed
Inhibitory Proteolgycan scar
Limited Scwann cell remyelination
Restricted axonal regrowth
Restricted Cycle of Deconditioning
SCI-Muscle paralysis-less physical activity-lower fitness/less mobile-change in body (more fat, less muscle)-high risk factors/chronic diseases
recurring arrow from high risk factors and then even less physical activity
associated complications for management of SCI includes
skin, bones, orthopedic, stabilization, handgrip or foot placement, bladder and bowels, illness, hypotension or hypertension, pain
Exercise Response to SCI
Limited mobility,
Restricted peak values (approx 1/2) of power output, oxygen consumption, cardiac output
Orthostatic and exercise hypotensive
peak HRs typically do not exceed 120 bpm
Considerations for Exercise testing
Purposes of the exercise test
Level of the SCI
The physical fitness level
To optimize equipment and protocol selection
Initial Exercise Testing
A functional assessment should be taken like
trunk ROM, wheelchair mobility, transfer ability, upper and lower extremity involvement
Cardiorespiratory endurance training
arm ergometer, cycle ergometer, wheelchair ergometer, rowing