Section 4: Feeding Management of Horses (Warren) Flashcards
Percentage breakdown of stomach, small intestine, and large intestine for horses
stomach: 8%
small intestine: 30%
large intestine: 62%
fxs of stomach
- start protein digestion
- mechanical churning
- NO absorption
2 parts of stomach
- non-glandular (esophageal region)
- glandular (cardiac, fundic, pyloric regions)
secretions of glandular stomach
HCl, pepsinogen, mucus
fxs of SI
- enzymatic digestion of starch protein, fat
- absorption of sugars, aa, NEFA, vits/mins
fxs of LI
microbes for fiber digestion
4 segments of LI
cecum, large colon, small colon, rectum
non-digestible carb
lignin
insoluble fibers. How are these digested?
hemicellulose, cellulose, ligno-cellulose. slowly fermented in LI –> acetate, butyrate
Resistant starches and soluble fibers. How are these digested?
fructans, pectins, gums, micilages, galactans. Rapidly fermented in LI –> lactate, propionate
how are monosaccharides, disaccharides, oligosaccharides, starches digested?
enzymatic hydrolysis in SI –> glucose
NDF
neutral detergent fiber. Comprised of insoluble fiber and lignin
see table top of slide 3
:)
5 common digestion disorders
1) impaction
2) gas build-up
3) hindgut acidosis
4) laminitis
5) gastric ulcers
impaction is caused by:
blockage in intestines, usually at sternal/pelvic flexures or ileocecal jx
normal products of microbial digestion of fiber in LI
VFA, B-vits, vit K, heat, gas
what happens in gas build-up? What is produced?
rate of gas prod. exceeds rate of gas removal with rapid fermentation of NSC (non-structurable carb). lactic acid, drop in pH, lots of gas produced.
gas build-up can secondarily cause:
colic, stomach rupture, torsion/displacement of intestine
hindgut acidosis
low pH in hindgut due to lactic acid prod. from rapid ferm. of NSC. Results in:
- microbe death
- endotoxins/inflamatory mediators –>diarrhea, laminitis
- metabolic changes
- reduced gastric motility –> colic
- reduced digestive efficiency
laminitis
disruption of intricate network of blood supply to feet. Can be caused by nutrition and other factors
founder
can happen secondarily to laminitis from tendons pulling up and weight of horse pushing down
nutrition-related laminitis is probably due to:
excess NSC intake. Causes metabolic aberrations and hidgut acidosis
most common site of gastric ulcers
non-glandular mucosa b/c it doesn’t have protective mechs from HCl prod. by gland. mucosa
Correct way to feed horses
- majority of diet = roughages (high fiber, energy, protein)
- small, frequent meals
- don’t overfeed NSC
- high quality protein sources
- gradual changes to diet
most starch comes from:
grains (i.e.oats, corn)
limit for starch digestion in SI
0.3% BW as starch per meal, and 0.5% BW as grain per meal
does microbial protein have value to horse?
NO. It needs to be met by diet
good sources of protein
soybean meal, skim milk, whey, casein
cool season grasses
timothy, orchardgrass, fescue, bromegrass
warm season grasses
bermudagrass, bahia, stargrass, pangolagrass
3 main forages used to feed horses
grass, legume, hay. Grass highest fiber, legume highest protein and Ca
legumes are high in:
protein, N, Ca
What is straw very high in?
fiber. Low in basically everything else