SA neuro IM diseases, spinal cord, intracranial Flashcards

1
Q

what are the three major variants of MUE

A

GME, Necrotizing encephalomyelitis (NME, NLE) and EME

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what is the most common dog type, and age associated with GME

A

female, toy breeds around 4.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what are the dog types, and major things associated with NME

A

pugs. rapidly progressive, forebrain signs, 2.5 y/o (seizures)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what tissue is NME in?

A

grey matter and meninges

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what tissue is GME in?

A

white matter of the brain. multifocal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

NLE necrotizing leukoencephalitis what dogs?

A

chihuahua and yorkies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

EME eosinophilic meningoencephalitis. which dogs?

A

rare. middle aged large breeds. causes any signs. they do better clinically.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

how does cat MUE compare to dog MUE

A

cats is more uncommon, cats respond well to steroids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is the most common cause of cranial neuropathies

A

idiopathic etiology. none are life threatening. most are self limiting.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

signs of trigeminal neuropathy

A

cant close mouth, one half of head muscles are caved in

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

eitologies for unilateral trigeminal neuropathies

A

nerve sheath tumor, meningioma, lymphoma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

no menace, no palpebral, bilateral facial paralysis, lip droop.. eitologies?

A

most common: idiopathic facial nerve paralysis. otitis media/interna

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

peripheral vestibular lesion vs central vestibular lesions. how to differentiate.

A

peripheral will have normal postural reactions and may include only CN VII

central vestibular will have postural reactions on ipsilateral to the lesion. and could include CN V-XII.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

dog presents with fever, poorly localized pain when palpating spine. what are three ddx

A

meningitis, polymyositis, polyarthritis.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

SRMA: signallment

A

B breeds. fever, young large breeds.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

acute vs chronic form of SRMA

A

acute: lethargy, fever, normal neuro exam.
chronic: waxed and waned for months. relapsing hyperpathia.

17
Q

which fibers in the spinal cord are most susceptible to injury? least?

A

most: large myelinated. proprioception is first to go
least: small, central fibers. nociception is last to go

17
Q

difference between grade 3 and grade 4 severity

A

3: nonambulatory, can move legs some if supported but cant walk.
4: completely paraplegic but has intact deep pain.

18
Q

if presenting schiff sherington posture, where is the lesion likely located?

A

T3-L3 lesion including border cells. when supported, can use thoracic limbs normally, but stiff when in lateral recumbancy

19
Q

chondrodystrophic dogs, progressive, painful, common in T3-L3. good to excellent prog with surgery

A

IVDE

20
Q

IVDP: breeds, presentation, treatment?

A

larger, older dogs. chronic and progressive but minimally painful. MRI is helpful. medical management or sx

21
Q

AANPE. presentation, diagnosis, treatment

A

nonprogressive after 48 hr, my have hyperalgesia, may occur during exercise. MRI is best to see liquid and contusion. SC

22
Q

difference between AANPE and HNPE? HNPE physiology, history

A

compressive lesion where AANPE is not, seagull shaped material. usually nonpainful, usually cervical myelopathy. (example shows dog with proprioceptive deficit in thoracic limb) could also present tetraplegic.

23
Q

miniature schauzer, peracute onset, nonprogressive after 24hr, nonpainful on presentation. tx: is SC and may not fully recover

A

FCE. with nociception present 70% recover.

24
Q

tetraplegic, normal CN, reflexes N, intact deep pain. localize?

A

C1-C5 UMN to all limbs. all reflexes intact.

25
Q

german shephard, working dog, trouble taking the stairs, lumbosacral pain, +- paresis of pelvic limbs and tail. most do medical management as surgeries are many and controversial

A

Degenerative lumbosacral stenosis, multifactorial. 50-75% improve with managment

26
Q

corgi, boxer. localized T3-L3 myelopathy. progressive. nonpainful. doagnosis? tx?

A

degenerative myelopathy: mutation of SOD1 gene. axonal degeneration. can do genetic testing and MRI to rule out other causes. ultimately fatal and progressive.

27
Q

doberman, weimaraner. apparently painful in caudal cervical region. as neck moves there is difference in severity. diag? prog?

A

cervical spindylomyelopathy. disc associated. try medical management first. surgery is invasive

28
Q

young giant breed dog. mastiff, great dane. presents painful. in cervical region. CT and MRI show squared off spinal cord. Diag?

A

cervical spondylomyelopathy. likely congenital.

29
Q

AAI: breeds, signalment, treatment? prog?

A

small. chihuahua, yorkie. episodic neck pain. reluctant to move neck. acute onset of diffuctuly walking. postural reactions absent in all four limbs. sx is best treatment >70% success rate.

30
Q

pugs, bullys. insidious onset of T3-L3 myelopathy. incontinence.

A

Caudal articular process dysplasia. no treatment. CAP

31
Q

rottweilers and pugs. chronic progressive myelopathy. medical managment helps and surgery is not good for the long term.

A

SAD. spinal arachnoid diverticulum. mri and myelogram best because fluid accum.

32
Q

FIP viruses FIV

A

fip: coronavirus, vasculitis in all organs
FIV: lentivirus. infects T cells. immunodeficient. fighting male cats