readings Flashcards
heart failure
effects of beta blockers on HR and BP with rest and exercise
HR and BP both decrease with rest and exercise
heart failure
effects of nitrates of HR and BP with rest and exercise
rest: increase HR; decrease BP
exercise: increase/no change HR; decrease/no change BP
(Dilates vessels)
heart failure
effects of calcium channel blockers of HR and BP with rest and exercise
CC blockers: end with “dipine”
both decrease with rest and exercises
heart failure
effects of digitalis of HR and BP with rest and exercise
HR: decrease in pts with aFib and possibly HR
BP: no change with rest and exercise
heart failure
effects of diuretics of HR and BP with rest and exercise
HR: no change with rest and exercise
BP: no change or decrease with rest and exercise
heart failure
effects of vasodilators of HR and BP with rest and exercise
HR: increase/stay the same with R and E
BP: decrease with R and E
heart failure
effects of ACE inhibitors, Angiotensin II Blockers, and Alpha Adrenergic Blockers of HR and BP with rest and exercise
HR: no change with R and E
BP: decrease with R and E
heart failure
effects of nicotine of HR and BP with rest and exercise
HR: increase/stay the same with R and E
BP: increase with R and E
heart failure
left sided heart failure
- pathology of the LV reduces cardiac outpu leading to a backup of fluid into the LA and lungs.
- the increased fluid in the lungs produces 2 hallmark pulmonary signs of left sided HF:SOB and cough
right sided heart failure
- occurs from direct insult to the RV caused by conditions that increase PA pressure.
- increased oressure within the PA subsequently increases afterload, thereby placing greater demands on the RV and causing it to go into failure
- blood is not effectively ejected from the RV and backs up into the RA and venous vasculature, producing 2 hallmark peripheral signs: jugular venous distention and peripheral edema
ejection fraction norm range
EF: 55-75%
terminology difference of compensated vs decompensated HF
compensated: the pt’s congestive symptos can be relieved by medical intervention
decompensated: shows s/s of congestion and requires medical and pharmacological readjustment
common s/s of CHF
fatigue
dyspnea
edema (pulmonary and peripheral)
weight gain
presence of S3 heart sound
renal dysfunction
other symptoms:
- paroxysmal nocturnal dyspnea
- orthopnea
- bendopnea
describe S3 heart sound
low frequency heart sound heart in early diastoleand occrs due to poor ventricular compliance and sibsequent turbulence of blood within the ventricle
- correlated with increase left ventricular end diastolic pressures and pulmonary capillary wedge pressure
paroxysmal nocturnal dyspnea vs orthopnea vs bendopnea
- paroxysmal nocturnal dyspnea: sudden episodes of SOB occuring in the night
- orthopnea: increased SOB in the recument position
- bendopnea: presence of SOB when the pt bends fwd
NYHA heart failure classifications
- class 1: pts with cardiac disease but without resulting limitations of physical activity. ordinary physical activity does not cause undue fatigue, palpitation, dyspnea or anginal pain
- class 2: pts with cardiac disease resulting in slight limitation of physical activty. they are comfortable at rest. ordinary physical activity results in fatigue, palpitations, dyspnea, and aginal pain
- class 3: pts with cardiac disease resulting in marked limitations of physicalactivity. they are comfortable at rest. less than ordinary physical activity causes fatigue, palpitations, dyspnea or anginal pain
- class 4: pts with cardiac disease resulting in inablity to carry on any physical activity without discomfort. symptoms of cardian insuffiencey or of the anginal syndrome may be present even at rest. if any physical activity is undertaken, discomfort is increased
dyspnea scale
0- no dyspnea
1- mild, noticeable
2 - mild, some difficulty
3 - mod difficulty but can continue
4 - severe difficulty, cannot continue
angina scale
0 - no angina
1 - light, barely noticeable
2 - moderate, bothersome
3 - severe, very uncomfortable: preinfarction pain
4 - most pain ever experienced; infarction pain
hypoxemia vs hypercapnea
hypoxemia- decreased amount of oxygen in the aterial blood to the tissue
hypercapnea - increased amount of co2 within the arterial blood will develop
cor pulmonale
increased pulmonary vascular resistance 2/2 capillary wall damage and reflex vasoconstriction in the presence of hypoxemia results in pulmonary HTN and RV hypertrophy
most common type of restrictive lung disease
pulmonary fibrosis
AKA
usual interstitial pneumonia
classic signs and symptoms of interstitial lung disease
symptoms: dyspnea w activity and persistant non productive vough
signs: rapid shallow breathing, limited chest expansion, inspiratory crackles, especially over the lower lung fields, digital clubbing and cyanosis
clincal significane for a 6MWT
25-35 meters
clinical significance for gait speed
0.05m/sec
common congenital disease of lymphedema
milroys disease
pitting edema
pressure on the edematous tissues with the fingertips causes an indentation of the skin that persists for several seconds after the pressure is removed.
this reflects significant but short duration edema with little or no fibrotic changes in the skin or subcutanous tissue
brawny edema
pressure on the edematous areas feels hard with palpation. this reflects a more severe form of interstitial swelling with progressive, fibrotic changes in subcutaneous tissues
brawny as in the paper towel guy - hes hard/tough
weeping edema
represents the most severe and long duration form of lymphedema. fluid leaks from cuts or sores; would healing is significantly impaired. exclusively in the LE
rare
stemmer sign
positive - indication of late stage 2 or stage 3 lymphedema
- indicitive of worsening condition
stage 1 pressure injury
non blanchable erythema of intact skin
- intact skin w/ a localised area of non blanchable erythema which may appear differently in darkly pigmented skin
- presence of blanchable erythema or changes in sensation, temperature, or firmess may precede visual change.
- colors do not include purple or maroon - this may indicate deep tissue pressure
stage 2 pressure injury
partial thickness skin loss with exposed dermis
- partial thickness loss of skin with exposed dermis
- the wound bed is viable, pink, or red, moist and may also present as an intact or ruptured serum filled blister
- granulation, slough and eschar are NOT present
stage 4 pressure injury
full thickness skin and tissue loss
- with exposed or directly palpable fascia, mm, tendon, ligament, cartilage, or bone in the ulcer.
unstageable pressure injury
obscured full thickness skin and tissue loss
- obsucred by slough or eschar
stage 3 pressure injury
full thickness skin loss
- adipose tissue is visible in the ulcer and granulation tissue and epibole are often present
- slough and eschare may be present
- undermining may occur
- fascia, mm, tendon, ligament and cartilage, bone are not exposed
ASIA A
complete
- no motor or sensory function presered in the sacral segments S4-S5
ASIA B
sensory incomplete
- sensory but not motor function is preserved below the neurological level and includes the sacral segment S4-S5
- no motor function is preserved more than 3 levels below the motor level on either side
ASIA C
motor incomplete
- motor fx is preserved at the most caudal sacral segments for voluntary anal contraction OR the patient meets the criteria for sensory incomplete status and has some sparing of motor function more than 3 levels below the ispsilateral motor level on either side of the body
- less than half of key mm functions below have a >=3
ASIA D
motor incomplete
- motor fx is preserved at the most caudal sacral segments for voluntary anal contraction OR the patient meets the criteria for sensory incomplete status and has some sparing of motor function more than 3 levels below the ispsilateral motor level on either side of the body
- **more than half of key mm functions below have a >=3 **
brown sequard syndrome
stab or gunshot wound
ipsilateral side:
- sensory loss (proprioception, light touch, vibratory) due to DCML tract paralysis
- paralysis due to the CST (lateral) tract
contralateral side
- loss of sense of pain and temp due to STT tract damage
- the loss begins several dermatome segments below the level of injury
can achieve good functional gains during inpatient rehab
anterior cord syndrome
related to flexion injuries to the c/s
- loss of motor function (CST) below the level of injury
- loss of sense of pain and temp (STT) below the level of injury
- DCML is preserved (priorprioception, light touch, vibratory)
longer length of inpatient rehab
central cord syndrome
most common syndrome
- occurs from hyperextension in the c/s or congenital/degenerative
- UE > LE
- motor > sensory
- normal sexual, b/b are retained
typically recover ambulation
cauda equina injuries
areflexic b/b and saddle anesthesia
considered peripheral nerve injuries (LMN)
conus medullaris syndrome
mixture of LMN and UMN
occurs very distal portion of spinal cord gets damaged
symptoms of autonomic dysreflexia
HTN
bradycardia
HA
profuse sweating
increased spasticity
vasodilation (flushing) above the level of lesion
constricted pupils
nasal congestion
piloerection (goosbumps)
blurred vision
normal BP for a person with an SCI
above T6 level
systolic: 90-110 mmHg
what level of SCI can drive with adaptive controls
C6
differences between fibromyalgia and myofascial pain syndrome
fibro
- tender points at specific sites
- no referred patterns of pain
- no tight bands of mm
- fatigue and waking unrefreshed
myofascial pain
- trigger points in mm
- referred patterns of pain
- tight band of mm
- no related fatigue complaints
characteristics of fibromyalgia
- first symptoms can occur at any age but usually appear during early to middle adulthood
- pain is reported in the scapula, head, neck chest and low back
- fluctuations of symptoms
Most common characteristics of an ACA
Contra lateral hemiparesis
Sensory LE>UE