psychopathology Flashcards

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1
Q

what is statistical infrequency

A

abnormality is defined as those behaviours that are extremely and statistically rarest therefore found in few people for example An Iq result under 70 is seen to be abnormal

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2
Q

evaluation of statistical infrequency

A
  • some abnormal behaviour is desirable eg an IQ over 150 is seen as “abnormally intelligent” TMB using SI the define abnormality means that we are unable to disguish desirable and undesirable behaviour
  • cut off point is every subjective eg why is 71 classed as normal and not 69 on an IQ test TMB the lack of definity between abnormality and normalative
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3
Q

deviation from social norms

A

abnormal bahaviour is seen as a deviation frm unstated rules about how we ought to behave. Anything that violets these rules is considered abnormal eg in the past homosexuality was classed as abnormal

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4
Q

evaluation of DFSN

A

+ breaking norms isnt always a bad thing eg homosexuality accepted now TMB historical be for the better
- deviance is related to context and degree eg summer and winter clothing TMB social deviance cant offer a full definition of abnormality because it is inevitably related to both context and degree

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5
Q

failure to function adequately

A

people are judged on their ability to go about daily life. If they cant do this and are also experiencing distress then it is considered a sign of abnormality.

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6
Q

examples of failure to function adequately

A

The DSM considers 6 factors ed getting along with people and self care on a scale of 1-5 generating a quantitive number out of 180 for functioning. eg maladaptiveness self harm and observer discomfort

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7
Q

evaluate FTFA

A

-who judges?
eg individaul might be content with their situation for example an old man staying at home and leaving when only necessary. TMB the judgement might be subjective based on who is deciding.
-cultural relativism
eg different diagnoses when applied to different cultures TMB the model is limited by different cultural relativisms.

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8
Q

deviation from ideal mental health

A

abnormality is defined in terms of mental health behaviours that are associated with competence and happiness. ideal mental health would include a positive attitude towards self, resistance to stress and accurate perception of life.

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9
Q

evaluation of deviation from ideal mental health

A
  • positive approach
    ed doesn’t describe abnormality as undesirable and influenced the positive psychology movement TMB postive outlook
  • unrealistic criteria
    eg hard to assess environmental mastery
    TMB not really usable for identifying abnormality
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10
Q

what is depression

A

a mood disorder where an individual feels sad and or lacks interest in their usual activities. Further characteristics irrational negative thoughts, raised/lowered activity levels difficulties concentrating, sleep and eating

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11
Q

characteristics of depression

A

emotional- sadness, feeling empty, low self esteem lost of pleasure in usual hobbies and anger
behavioural- shift in activity levels, sleep and appetite
cognitive- negative schemas that attract negative thoughts that are irrational eg “ i failed my exam i am stupid”

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12
Q

what is OCD

A

an anxiety disorder where anxiety arises from both obsession (persistent thoughts) and compulsions (repetitve behaviour) are a response to obsessions and the person believes the compulsions will reduce anxiety.

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13
Q

characteristics of OCD

A

emotional- obsessions and compulsions are a source of anxiety and distress. suffers are aware their behaviour is excessive which causes embarrassment and shame.
behavioural- compulsive behaviours are performed to reduce anxiety they are compelled because they believe something awful will happen
cognitive- obsessions are recurrent and perceived to the sufferer as inappropriate or forbidden

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14
Q

definition of phobias

A

a group of mental disorders characterised by high levels of anxiety in responce to a phobic stimulus. This is life interfering

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15
Q

characteristics of phobias

A

emotional- excessive dear panic and high levels of anxiety
behavioural - avoiding phobic stimulus
cognitive - mental processes in claustrophobia such as “ the small space is out to get me”

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16
Q

what is a phobia

A

a phobia is an irrational fear that produces conscious avoiding behaviours eg claustrophobia

17
Q

describe Mowrer’s model in explaining phobias

A

process 1 - initiation classical conditioning for example a phobia of dogs.
when associated with a bite (UCS) turns into the CR of fear to seeing a dog CS which was previously a NS
process 2 - maintenance operant conditioning for example avoiding the phobic stimulus reduces fear and is therefore rewarding making it more likely for a individual to avoid their phobia (negative reinforcement)

18
Q

evaluate mowrers 2 way process model

A

+ supporting study little albert by watson and raynor created a phobia of rats in 11 month old TMB it suggests that phobias can be created from a unfortunate event therefore increases the validity
- biological preparedness seligman suggests we have ancient fears such as heights and strangers which have come from evolution and survival TMB not all phobias derive from a traumatic experience
-diathesis stress model for example mowrers model cant explain generic vulnerability diathesis (every one born with genetic vulnerability) stress (environmental factors) that trigger the genetic vulnerability

19
Q

what is systematic desensitisation?

A

when a client is gradually exposed their phobic stimulus under relaxed conditions until te anxiety is extinguished

20
Q

what 3 components makes up for systematic desensitisation

A
  • fear hierarchy gradual steps determined by patient and therapist from least to most fearful
    -relaxation training patient taught deep muscle relaxation and breathes cant move onto next stage until fully relaxed
    -reciprocal inhibition based on the theory that no 2 opposite emotions can be felt a once
21
Q

what is flooding

A

a client is exposed to the phobia at its worst. A long session until the anxiety reaction has disappeared after being taught how to relax. this can either be vivo (real) or virtual reality

22
Q

evaluation of systematic desensitisation

A

+ supporting Mcgrath study 75% responded to treatment
- not appropriate for all phobias ohman suggested SD might be as useful for evolutionary survival components eg fear of the dark TMB it suggests SD only effective for some phobias

23
Q

evaluation for flooding

A
  • individual difference flooding isnt an appropriate treatment for everyone for example it is highly traumatic process therefore would limit the effectiveness of the therapy
    +effectiveness craske et al in 2008 found SD and flooding were equally useful TMB it suggests that flooding is an effective treatment
24
Q

2 explainations for depression

A
  • becks negative triad
    -Ellis ABC model
25
Q

describe Ellis’ ABC model

A

A- activating event
B- beliefs (irrational)
C- consequence eg unhealthy emotion
musturbatory thinking - thinking that certain ideas must be true in order for an individual to be happy eg i must be accepted by people to be happy

26
Q

describe Becks negative triad

A

depressed people have acquired a negative schema these led to cognitive biases for example labelling oneself “i am worthless”
negative triad - negative expectations about the self , world and future led to depression

27
Q

evaluation for explainations of depression

A

+ supporting study Bates et al he gave depressed participants negative automatic statements he found that they became more depressed TMB it supports the view that negative thinking can led to depression
- ignores other factors such as low levels of serotonin additionally Zhang et al found a gene 10 times more common in depressed patients TMB it can be argued the explainations arnt full
+real world application led to the treatment of CBT TMB it is helping people therefore suggests its effective

28
Q

describe CBT

A

-challenging irrational thoughts
Disputing / challenging
Effects of disputing
F new feelings that are produced
-Homework assignments that test irrational beliefs against reality
- behavioural actions encouraging patients to be active and peruse usually hobbies
-unconditional positive regard facilitates a change in attitude by therapist showing their value as a person

29
Q

evaluation of CBT

A

+ supporting study Ellis found 90% effective with 27 sessions supporting research
- individual differences takes a lot of effort for patient to do homework and do a thought diary TMB individual differences effect its effectiveness
+ support for behavioural action babyak et al studied 156 either assigned either sports drugs or all. the ones with all showed significant improvement over 4 months TMB change in behaviour can be benificial to treating depression

30
Q

genetic explaination to OCD

A

-Genetic explanations for OCD suggest that individuals inherit specific genes that cause OCD. Two genes that have been linked to OCD are the COMT gene and SERT gene. The SERT gene (also known as the 5-HTT gene) affects the transport of serotonin and can cause lower levels of serotonin, which is also associated with OCD. COMT is responsible for dopamine and low activity of the COMT gene is also associated with OCD. It is also believed that OCD is a polygenic condition, which means that several genes are involved.
- diathesis stress model gentic predisposition of genes + environmental factors trigger OCD

31
Q

Neural factors to OCD

A

-high levels of dopamine induce sterotypical movements resembling compulsive behaviour found in OCD patients
-low levels of serotonin found in OCD
-basal ganglia when damaged affects the coordination of movement which induce sterotypical movements resembling compulsive behaviour found in OCD patients
-obitofrontal cortex coverts sensory information into actions/ behaviours if it has higher activity may lead to intrusive thoughts and obsessions

32
Q

evaluation to explainations to OCD

A

+ supporting twin study Nestadt et al found that monozygotic twins had a concorance rate of 68% whereas dizygotic twins had 31% TMB the fact that MZ share 100% genes shows there is a genetic componant to OCD
+ real life application women can have eggs screened and turn certain genes off TMB suggests that specific genes can cause OCD
-biological approach iggnores the diathesis stress model could be caused by classical conditions eg dirt NS and illness UCS TMB not a full explaination

33
Q

drugs therapy for OCD

A

-antidepressants SSRIs (prozac)
-tricyclics brand name anafranil
-anti anxiety valium

34
Q

how do SSRIs treat OCD

A

SSRI’s block the re-uptake of serotonin at the pre-synaptic membrane increasing the serotonin concentration at receptor sites on the post synaptic membrane

35
Q

how do tricyclics treat OCD

A

work the same as sSRI’s but also do the same with Noradrenaline a nuerotransmittor involved in autonomic system eg heart rate these have big side effects

36
Q

how do anti anxiety drugs work to treat OCD

A

slows down central nervous system and enhances activity of GABA neurotransmitter therefore quietening effect of nuerons on the brain. it increases the flow of chloride ions making it harder for nueron to be stimulated making people feel relaxed

37
Q

evaluation of treatments to OCD

A

-not a lasting cure may work in the short term but doesnt cure the underlying cure for OCD
-side effects for SSRis are nausea headache and insomnia TMB harsh side effects and possablity of addiction limit drugs usefullness
+Soomro reviewd 17 cases of drug vs placebo and drug was more effective in reducing symptoms TMB shown to be effective