PFPS/patellar dislocation - exam 3 Flashcards
PF GRF:
- walking
- near 30 deg flx
- stair climbing
- squatting
- peak at:
- 50% BW
- BW
- 3x BW
- > 7x BW
- 90 deg
what % of PFPS is recurrent and persistent?
70-90 %
risk factors for PFPS
military recruits
dynamic (NOT static) excessive pronation
females
– larger Q angle
– differing hip strength and coordination
patellar and femoral bone shape
is PFPS normally caused by gradual unknown onset or trauma?
gradual and unknown onset
what is the theory for the cause of PF malalignment and/or maltracking?
patella glides and tilts more laterally relative to femur
Someone with PFPS has ________ surface area contact between the patella and femur
decreased
less contact puts more force in a smaller area —-» wear down and causes P!
PF malalignment and/or maltracking is due to:
- patellar and femoral bone shape
- femoral IR and add
- quad weakness, incoordination and atrophy
- unclear contribution from excessive pronation, and tibial IR
pathomechanics of PFPS:
overload of patellar subchondral bone, esp lateral facet
tissue ischemia
loss of tissue homeostasis
neural ingrowth and increase in substance nerve fibers that transmit more P!
how does the infrapatellar fat pad become inflamed? where can it refer to?
excessive tibial IR and patellar hypermobility
groin
what makes up the bursae?
superficial infrapatellar - between skin and patellar tendon
deep infrapatellar - between patellar tendon and tibia
what are other structures involved in PFPS?
subchondral bone of patella
quad and patellar tendons
synovium
med & lat retincaculum - help hold patella and tendon in position
what are symptoms of PFPS?
- onset
- location of P!
- P! increased with:
- gradual
- anteromedial knee p!
- stairs, squatting, kneeling, or prolonged sitting (holding pressure on a small area for a period of time)
Observation of PFPS:
increased Q angle
open chain maltracking of patella
quad atrophy
observation of PFPS: impaired LE control
proprioceptive deficits
dynamic excessive pronation
–> possible contributions from impaired DF because if DF limited then more EV may occur
abnormal planar motions, esp in females
consider possible contributions from L4-S1 regional interdependence
observation of PFPS: impaired LE control in frontal and sagittal plane:
increased
e.g. hip add
- glue med and/or max incoordination
- hip ext and abd weakness
observation of PFPS: impaired LE control in transverse plane
hip ER weakness and incoordination
mixed conclusions with contributions from rotational impairments
Observation: PFPS
Trunk weakness including ___________
Consider possible contributions from __________ regional interdependence
excessive trunk lean
L4-S1
signs of PFPS:
- rom:
limited and painful, particularly at end ranges if symptomatic
- flx: greater PF compression
- ext: more fat pad irritation
signs of PFPS:
- resisted:
possible P! with ext MMT and weakness
likely inhibited quad activity, part. VM
potential anti-gravity trunk and hip weakness
signs of PFPS:
- stress tests:
- neuro tests
- accessory motion
- possible pain with PF compression
- limited Dural mobility of femoral nerve in 1/3 patients
- typically excessive lateral motion and limited medial motion
- all glides could be hypermobile